Human cytomegalovirus stimulates monocyte-to-macrophage differentiation via the temporal regulation of caspase 3.
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Mutation in osteoactivin decreases bone formation in vivo and osteoblast differentiation in vitro.Host microRNA regulation of human cytomegalovirus immediate early protein translation promotes viral latency.Cytomegalovirus, Macrophages and Breast Cancer.Human cytomegalovirus modulates monocyte-mediated innate immune responses during short-term experimental latency in vitroBH3 Profiling Reveals Selectivity by Herpesviruses for Specific Bcl-2 Proteins To Mediate Survival of Latently Infected Cells.The human cytomegalovirus lytic cycle is induced by 1,25-dihydroxyvitamin D3 in peripheral blood monocytes and in the THP-1 monocytic cell lineHCMV Displays a Unique Transcriptome of Immunomodulatory Genes in Primary Monocyte-Derived Cell Types.Human Cytomegalovirus Promotes Survival of Infected Monocytes via a Distinct Temporal Regulation of Cellular Bcl-2 Family Proteins.Human cytomegalovirus interleukin-10 polarizes monocytes toward a deactivated M2c phenotype to repress host immune responsesViral binding-induced signaling drives a unique and extended intracellular trafficking pattern during infection of primary monocytes.HCMV reprogramming of infected monocyte survival and differentiation: a Goldilocks phenomenon.The HCMV US28 vGPCR induces potent Gαq/PLC-β signaling in monocytes leading to increased adhesion to endothelial cells.Human Cytomegalovirus Requires Epidermal Growth Factor Receptor Signaling To Enter and Initiate the Early Steps in the Establishment of Latency in CD34+ Human Progenitor Cells.Human Cytomegalovirus Stimulates the Synthesis of Select Akt-Dependent Antiapoptotic Proteins during Viral Entry To Promote Survival of Infected Monocytes.Human Cytomegalovirus Induces an Atypical Activation of Akt To Stimulate the Survival of Short-Lived Monocytes.Intratumoral infection by CMV may change the tumor environment by directly interacting with tumor-associated macrophages to promote cancer immunity.A little cooperation helps murine cytomegalovirus (MCMV) go a long way: MCMV co-infection rescues a chemokine salivary gland defect.HCMV utilizes a non-traditional STAT1 activation cascade via signaling through EGFR and integrins to efficiently promote the motility, differentiation, and polarization of infected monocytes.Role of Human Macrophage Polarization in Inflammation during Infectious Diseases.HCMV Infection and Apoptosis: How Do Monocytes Survive HCMV Infection?Molecular Determinants and the Regulation of Human Cytomegalovirus Latency and Reactivation
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P2860
Human cytomegalovirus stimulates monocyte-to-macrophage differentiation via the temporal regulation of caspase 3.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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name
Human cytomegalovirus stimulat ...... poral regulation of caspase 3.
@ast
Human cytomegalovirus stimulat ...... poral regulation of caspase 3.
@en
type
label
Human cytomegalovirus stimulat ...... poral regulation of caspase 3.
@ast
Human cytomegalovirus stimulat ...... poral regulation of caspase 3.
@en
prefLabel
Human cytomegalovirus stimulat ...... poral regulation of caspase 3.
@ast
Human cytomegalovirus stimulat ...... poral regulation of caspase 3.
@en
P2093
P2860
P356
P1433
P1476
Human cytomegalovirus stimulat ...... poral regulation of caspase 3.
@en
P2093
Andrew D Yurochko
Maciej T Nogalski
P2860
P304
10714-10723
P356
10.1128/JVI.07129-11
P407
P577
2012-07-25T00:00:00Z