Effect of a recA mutation on cholera toxin gene amplification and deletion events.
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Crystal Structures of a CTX pIII Domain Unbound and in Complex with a Vibrio cholerae TolA Domain Reveal Novel Interaction InterfacesThe sixth and seventh cholera pandemics are due to independent clones separately derived from environmental, nontoxigenic, non-O1 Vibrio cholerae.Enteric bacterial toxins: mechanisms of action and linkage to intestinal secretion.Analysis of clinical and environmental strains of nontoxigenic Vibrio cholerae for susceptibility to CTXPhi: molecular basis for origination of new strains with epidemic potentialInduction of DNA amplification in the Bacillus subtilis chromosomeMycobacterium bovis BCG recA deletion mutant shows increased susceptibility to DNA-damaging agents but wild-type survival in a mouse infection model.Sunlight-induced propagation of the lysogenic phage encoding cholera toxin.Diminished diarrheal response to Vibrio cholerae strains carrying the replicative form of the CTX(Phi) genome instead of CTX(Phi) lysogens in adult rabbits.Lysogenic conversion by a filamentous phage encoding cholera toxin.Thermostable direct hemolysin gene of Vibrio parahaemolyticus: a virulence gene acquired by a marine bacterium.Heterologous antigen expression in Vibrio cholerae vector strainsMechanisms of inflammasome activation by Vibrio cholerae secreted toxins vary with strain biotype.A new type of conjugative transposon encodes resistance to sulfamethoxazole, trimethoprim, and streptomycin in Vibrio cholerae O139.Development and characterization of recA mutants of Campylobacter jejuni for inclusion in attenuated vaccines.Encoded errors: mutations and rearrangements mediated by misalignment at repetitive DNA sequences.Competing for Iron: Duplication and Amplification of the isd Locus in Staphylococcus lugdunensis HKU09-01 Provides a Competitive Advantage to Overcome Nutritional Limitation.Neutrophils are essential for containment of Vibrio cholerae to the intestine during the proinflammatory phase of infection.Cloning of the recA gene of Neisseria gonorrhoeae and construction of gonococcal recA mutantsExpression and regulation of a Vibrio alginolyticus sucrose utilization system cloned in Escherichia coliIsolation and characterization of the Vibrio cholerae recA gene.CTX genetic element encodes a site-specific recombination system and an intestinal colonization factor.Multiple pathways of duplication formation with and without recombination (RecA) in Salmonella enterica.Cloning of the Vibrio cholerae recA gene and construction of a Vibrio cholerae recA mutant.Gene amplification in Rhizobium: identification and in vivo cloning of discrete amplifiable DNA regions (amplicons) from Rhizobium leguminosarum biovar phaseoliAccessory cholera enterotoxin (Ace), the third toxin of a Vibrio cholerae virulence cassette.Homologous plasmid recombination is elevated in immortally transformed cells.Diminished immunogenicity of a recombination-deficient derivative of Vibrio cholerae vaccine strain CVD103.Genetics of Vibrio cholerae and its bacteriophages.Mycobacterium tuberculosis RecA is indispensable for inhibition of the mitogen-activated protein kinase-dependent bactericidal activity of THP-1-derived macrophages in vitro.The Vibrio cholerae O139 Calcutta bacteriophage CTXphi is infectious and encodes a novel repressor.Lysogenic conversion of environmental Vibrio mimicus strains by CTXPhiToxR-independent expression of cholera toxin from the replicative form of CTXphi.Induction of the lysogenic phage encoding cholera toxin in naturally occurring strains of toxigenic Vibrio cholerae O1 and O139.Vibrio cholerae hemagglutinin/protease inactivates CTXphi.Molecular analyses of a putative CTXphi precursor and evidence for independent acquisition of distinct CTX(phi)s by toxigenic Vibrio cholerae.Discrete amplifiable regions (amplicons) in the symbiotic plasmid of Rhizobium etli CFN42.Transposition of Tn1545-delta 3 in the pathogenic Neisseriae: a genetic tool for mutagenesis.Amplification and deletion of a nod-nif region in the symbiotic plasmid of Rhizobium phaseoli.Evidence for insertion sequence-mediated spread of the thermostable direct hemolysin gene among Vibrio species.Formation of SXT tandem arrays and SXT-R391 hybrids.
P2860
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P2860
Effect of a recA mutation on cholera toxin gene amplification and deletion events.
description
1986 nî lūn-bûn
@nan
1986年の論文
@ja
1986年学术文章
@wuu
1986年学术文章
@zh-cn
1986年学术文章
@zh-hans
1986年学术文章
@zh-my
1986年学术文章
@zh-sg
1986年學術文章
@yue
1986年學術文章
@zh
1986年學術文章
@zh-hant
name
Effect of a recA mutation on cholera toxin gene amplification and deletion events.
@ast
Effect of a recA mutation on cholera toxin gene amplification and deletion events.
@en
type
label
Effect of a recA mutation on cholera toxin gene amplification and deletion events.
@ast
Effect of a recA mutation on cholera toxin gene amplification and deletion events.
@en
prefLabel
Effect of a recA mutation on cholera toxin gene amplification and deletion events.
@ast
Effect of a recA mutation on cholera toxin gene amplification and deletion events.
@en
P2860
P1476
Effect of a recA mutation on cholera toxin gene amplification and deletion events.
@en
P2093
I Goldberg
J J Mekalanos
P2860
P304
P356
10.1128/JB.165.3.723-731.1986
P407
P577
1986-03-01T00:00:00Z