Evidence in support of a self-perpetuating HLA-DR-dependent delayed-type cell reaction in rheumatoid arthritis.
about
Interferon-gamma production in response to in vitro stimulation with collagen type II in rheumatoid arthritis is associated with HLA-DRB1(*)0401 and HLA-DQ8Current perspectives on synovitis.Lasker Clinical Medical Research Award. TNF defined as a therapeutic target for rheumatoid arthritis and other autoimmune diseasesLymphocyte-macrophage co-operation during induction of T-suppressor cell activity in rats with adjuvant arthritisExperimental autoimmune arthritis in mice. I. Homologous type II collagen is responsible for self-perpetuating chronic polyarthritis.T cell subsets and expression of immunological activation markers in the arterial walls of patients with giant cell arteritis.Analysis of lymphocyte subsets of bone marrow in patients with rheumatoid arthritis by two colour immunofluorescence and flow cytometry.Persistence of collagen type II-specific T-cell clones in the synovial membrane of a patient with rheumatoid arthritis.Identification of immunostimulatory dendritic cells in the synovial effusions of patients with rheumatoid arthritis.Influences of gamma interferon on synovial fibroblast-like cells. Ia induction and inhibition of collagen synthesis.Rheumatoid arthritis and tumor necrosis factor alpha.Immune function in severe, active rheumatoid arthritis. A relationship between peripheral blood mononuclear cell proliferation to soluble antigens and synovial tissue immunohistologic characteristics.Adoptive transfer of suppression of arthritis in the mouse model of collagen-induced arthritis. Evidence for a type II collagen-specific suppressor T cell.Plasminogen activators and their inhibitors in arthritic disease.Arthritis is associated with T-cell-induced upregulation of Toll-like receptor 3 on synovial fibroblastsDifferential expression of Ia antigens by rheumatoid synovial lining cells.Pristane-induced arthritis in rats: a new model for rheumatoid arthritis with a chronic disease course influenced by both major histocompatibility complex and non-major histocompatibility complex genesMultifunctional T cell reactivity with native and glycosylated type II collagen in rheumatoid arthritisModulating co-stimulation: a rational strategy in the treatment of rheumatoid arthritis?Induction of platelet-derived growth factor receptor expression in smooth muscle cells and fibroblasts upon tissue culturing.Spontaneous production of fibroblast-activating factor(s) by synovial inflammatory cells. A potential mechanism for enhanced tissue destruction.An arthritogenic lymphokine in the rat.Novel therapies for rheumatoid arthritis.Transforming growth factor beta 1 (TGF-beta 1) controls expression of major histocompatibility genes in the postnatal mouse: aberrant histocompatibility antigen expression in the pathogenesis of the TGF-beta 1 null mouse phenotype.Activation of macrophages in an experimental rat model of arthritis induced by Erysipelothrix rhusiopathiae infectionLymphocyte-derived cytokines in inflammatory arthritis.The inflammatory role of phagocyte apoptotic pathways in rheumatic diseases.Development of dendritic cell-based immunotherapy for autoimmunity.Mapping pathogenesis of arthritis through small animal models.The immunopathogenesis of seropositive rheumatoid arthritis: from triggering to targeting.Study of sokei-kakketu-to (shu-jing-huo-xue-tang) in adjuvant arthritis rats.Collagen arthritis in rats, arthritogenic lymphokines and other aspects.Occurrence of autoantibodies against intrinsic factor, H,K-ATPase, and pepsinogen in atrophic gastritis and rheumatoid arthritis.Lymphoid abnormalities in rats with adjuvant-induced arthritis. I. Mitogen responsiveness and lymphokine synthesis.Th17 in Animal Models of Rheumatoid Arthritis.Expression of vascular cell adhesion molecule-1 in fibroblastlike synoviocytes after stimulation with tumor necrosis factor.The caspase-8/RIPK3 signaling axis in antigen presenting cells controls the inflammatory arthritic response.Synovial biology and T cells in rheumatoid arthritis.HLA-DR expression in the vascular lesion and circulating T lymphocytes of patients with giant cell arteritis.A novel paradigm for dendritic cells as effectors of cartilage destruction.
P2860
Q24792193-0B4E019E-A069-413D-BDD3-1C6EF86F1091Q24804893-FBB589BE-8EA7-4227-A88A-5E836D4EB000Q28206018-64B5F1F7-BAF6-4FFA-8307-329B56AFEAE5Q33554859-58693BF9-BF5B-4160-AE3C-26B81B4512D6Q33557750-0FBD709D-4423-40BD-A04C-1246C5E3B501Q33560535-C293073A-850B-4CC7-B7A9-A4C01D08A038Q33562445-6F40BA17-F6DD-4A54-88C9-8FD968E92422Q33832421-D9A2506F-6C65-413D-8649-BA3833F4E53BQ34549783-8395ADC5-5E99-4610-9F60-C6C42B61C683Q34550337-28E4383F-E80E-4BDA-813D-2090FA09BDCEQ34572250-0BF78A6C-0E56-4325-AB31-3A9BD7F68676Q34616313-108990E3-C353-49D9-8A01-3C9A113BAA88Q34630874-12B0BF22-BC18-4822-8A3D-E93C8B0846F9Q35224564-8E5EBEE0-4E3F-44E4-84B6-BF5C776879ABQ35557956-316453D5-19BA-45C6-A658-F95A736C0C17Q35577936-8A1D3E3E-C791-4113-8AE4-F049B4C33ADFQ35782480-BD10CC4F-DF44-45A2-BEA7-D59B50D4B295Q36069629-99C2FA07-E671-4D70-B75D-36A3C8189B46Q36099553-8C683D51-16BA-45BF-92F7-40691FB58E4BQ36219663-64952AD6-0906-4405-A638-B30FA865069FQ36349752-D200D2C4-44B9-44F8-9B0B-51928E44E660Q36351245-33620806-E5B7-4AC8-BC45-E10C8B607C12Q36623081-5FB19F96-15CB-44DE-99BD-C0358AEA618FQ36623685-317F4920-E7FD-4675-9DF6-513CD6049761Q37025151-35B42F6C-4FB8-40B4-92F1-A902FF104D04Q37118786-D2CA0A25-74E9-4795-ADC1-E2D1D6E97F7EQ37631369-70BFBF22-441D-4B3D-8E32-643393A78ADDQ37701864-7DC4CF85-3B24-4FB9-902E-B13B9F5AB276Q37994660-399100F7-F05A-4A68-BBCE-1AB20F760A65Q39029106-D3DEF54A-F895-44BC-BB25-1285B9C1A925Q39383831-4F4EAE0B-361B-4DA0-B5A2-E8A32C111688Q39572035-E366E78C-1BDF-4714-936B-B9186E0EDD50Q41131362-11A3D50D-42E2-4D35-9172-48ECC6381ACCQ41144864-71311EFB-96D6-4C30-ACCD-E2AD43C09210Q41161406-60582F41-6328-4ED6-AE75-A537E33617D1Q41857345-F5F8B322-36B1-423E-8F74-869D4BCBF042Q42278840-AE660258-059D-44DA-BF2B-C8FFD00BF1DBQ42446281-EDAE2A59-1D11-4EA2-A0D3-9DFE2F3293D4Q43819617-7AECF378-FDBE-4C51-B040-A43D54B3F60AQ46095815-80BE0A6B-4343-42B4-9BE4-1737D9336205
P2860
Evidence in support of a self-perpetuating HLA-DR-dependent delayed-type cell reaction in rheumatoid arthritis.
description
1982 nî lūn-bûn
@nan
1982年の論文
@ja
1982年学术文章
@wuu
1982年学术文章
@zh-cn
1982年学术文章
@zh-hans
1982年学术文章
@zh-my
1982年学术文章
@zh-sg
1982年學術文章
@yue
1982年學術文章
@zh
1982年學術文章
@zh-hant
name
Evidence in support of a self- ...... ction in rheumatoid arthritis.
@ast
Evidence in support of a self- ...... ction in rheumatoid arthritis.
@en
type
label
Evidence in support of a self- ...... ction in rheumatoid arthritis.
@ast
Evidence in support of a self- ...... ction in rheumatoid arthritis.
@en
prefLabel
Evidence in support of a self- ...... ction in rheumatoid arthritis.
@ast
Evidence in support of a self- ...... ction in rheumatoid arthritis.
@en
P2093
P2860
P356
P1476
Evidence in support of a self- ...... ction in rheumatoid arthritis.
@en
P2093
P2860
P304
P356
10.1073/PNAS.79.11.3632
P407
P577
1982-06-01T00:00:00Z