Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice. - Wikidata - wikimedia - TriplyDB

Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.

Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.

http://www.wikidata.org/entity/Q36300691

about

Biological Rhythms in the Skin Psychopathological Variables and Sleep Quality in Psoriatic Patients Imiquimod has strain-dependent effects in mice and does not uniquely model human psoriasis.Nutrients and the microenvironment to feed a T cell army.The circadian clock regulates inflammatory arthritis.TIMP3 is a CLOCK-dependent diurnal gene that inhibits the expression of UVB-induced inflammatory cytokines in human keratinocytes.Trichosanthes kirilowii extract enhances repair of UVB radiation‑induced DNA damage by regulating BMAL1 and miR‑142‑3p in human keratinocytes.Biological clocks: their relevance to immune-allergic diseases.The association between Korean employed workers' on-call work and health problems, injuries.From Early Immunomodulatory Triggers to Immunosuppressive Outcome: Therapeutic Implications of the Complex Interplay Between the Wavebands of Sunlight and the Skin

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P2860

Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.

http://www.wikidata.org/entity/Q36300691

description

2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

@zh-hans

2015年学术文章

@zh-my

2015年学术文章

@zh-sg

2015年學術文章

@yue

2015年學術文章

@zh

2015年學術文章

@zh-hant

name

Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.

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Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.

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type

label

Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.

@ast

Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.

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prefLabel

Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.

@ast

Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.

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Journal of Investigative Dermatology

P1476

Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.

@en

P2093

Atsuhito Nakao

http://www.w3.org/2001/XMLSchema#string

Hideoki Ogawa

http://www.w3.org/2001/XMLSchema#string

Kayoko Ishimaru

http://www.w3.org/2001/XMLSchema#string

Ko Okumura

http://www.w3.org/2001/XMLSchema#string

Noriko Ando

http://www.w3.org/2001/XMLSchema#string

Rui Aoki

http://www.w3.org/2001/XMLSchema#string

Shinji Shimada

http://www.w3.org/2001/XMLSchema#string

Yuki Nakamura

http://www.w3.org/2001/XMLSchema#string

P2860

Immunology of psoriasis

Molecular architecture of the mammalian circadian clock

Mutagenesis and mapping of a mouse gene, Clock, essential for circadian behavior

The IL-23/Th(17) axis: therapeutic targets for autoimmune inflammation

Central and peripheral circadian clocks in mammals

The mammalian circadian timing system: organization and coordination of central and peripheral clocks

A new player on the psoriasis block: IL-17A- and IL-22-producing innate lymphoid cells.

Pivotal role of dermal IL-17-producing γδ T cells in skin inflammation.

IL-23 stimulates epidermal hyperplasia via TNF and IL-20R2-dependent mechanisms with implications for psoriasis pathogenesis

Microbiome and skin diseases.

P2888

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3001-3008

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scholarly article

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10.1038/JID.2015.316

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12

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135

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Shigenobu Shibata

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2015-08-20T00:00:00Z

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281170689

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26291684

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circadian rhythm

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4653315

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