Helicobacter pylori DNA decreases pro-inflammatory cytokine production by dendritic cells and attenuates dextran sodium sulphate-induced colitis - Wikidata - wikimedia - TriplyDB

Helicobacter pylori DNA decreases pro-inflammatory cytokine production by dendritic cells and attenuates dextran sodium sulphate-induced colitis

Helicobacter pylori DNA decreases pro-inflammatory cytokine production by dendritic cells and attenuates dextran sodium sulphate-induced colitis

http://www.wikidata.org/entity/Q36302922

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Enterohepatic Helicobacter Species as a Potential Causative Factor in Inflammatory Bowel Disease: A Meta-Analysis Differential inflammatory response to Helicobacter pylori infection: etiology and clinical outcomes Interplay of the Gastric Pathogen Helicobacter pylori with Toll-Like Receptors Polymorphism in the Helicobacter pylori CagA and VacA toxins and disease Metabolic Interaction of Helicobacter pylori Infection and Gut Microbiota Helicobacter pylori infection and inflammatory bowel disease in Asians: a meta-analysis IBD-what role do Proteobacteria play?Role of childhood infection in the sequelae of H. pylori disease Pathogenesis of Crohn's disease: Bug or no bug.The adjuvant effects of high-molecule-weight polysaccharides purified from Antrodia cinnamomea on dendritic cell function and DNA vaccines.Inverse association of esophageal eosinophilia with Helicobacter pylori based on analysis of a US pathology database Epidemiology, Diagnosis, and Management of Esophageal Adenocarcinoma Systems-wide analyses of mucosal immune responses to Helicobacter pylori at the interface between pathogenicity and symbiosis Helicobacter pylori DNA's anti-inflammatory effect on experimental colitis The immunomodulatory properties of Helicobacter pylori confer protection against allergic and chronic inflammatory disorders Helicobacter pylori γ-glutamyl transpeptidase and vacuolating cytotoxin promote gastric persistence and immune tolerance.Helicobacter pylori activates the TLR2/NLRP3/caspase-1/IL-18 axis to induce regulatory T-cells, establish persistent infection and promote tolerance to allergens Life in the human stomach: persistence strategies of the bacterial pathogen Helicobacter pylori.DC-LAMP+ dendritic cells are recruited to gastric lymphoid follicles in Helicobacter pylori-infected individuals Helicobacter pylori infection does not promote hepatocellular cancer in a transgenic mouse model of hepatitis C virus pathogenesis.Association between Helicobacter pylori and Barrett's esophagus, erosive esophagitis, and gastroesophageal reflux symptoms.Inflammation, immunity, and vaccine development for Helicobacter pylori.Helicobacter pylori infection and inflammatory bowel disease: is there a link?Inflammasome Activation by Helicobacter pylori and Its Implications for Persistence and Immunity.The Human Stomach in Health and Disease: Infection Strategies by Helicobacter pylori.DNA Transfer and Toll-like Receptor Modulation by Helicobacter pylori The association between Helicobacter pylori infection and inflammatory bowel disease based on meta-analysis.The influence of Helicobacter pylori on the ethnic distribution of Barrett's metaplasia.TLR9 activation suppresses inflammation in response to Helicobacter pylori infection.Helicobacter pylori-specific protection against inflammatory bowel disease requires the NLRP3 inflammasome and IL-18.Vacuolating cytotoxin and variants in Atg16L1 that disrupt autophagy promote Helicobacter pylori infection in humans.Helicobacter pylori-Mediated Protection against Extra-Gastric Immune and Inflammatory Disorders: The Evidence and Controversies.H. pylori attenuates TNBS-induced colitis via increasing mucosal Th2 cells in mice.Do Acid-Suppressing Medications in Inflammatory Bowel Disease Increase Risk for Flare?Helicobacter pylori and Extragastric Diseases.Helicobacter pylori: Does Gastritis Prevent Colitis?Helicobacter pylori infection and inflammatory bowel disease: a crosstalk between upper and lower digestive tract

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Helicobacter pylori DNA decreases pro-inflammatory cytokine production by dendritic cells and attenuates dextran sodium sulphate-induced colitis

http://www.wikidata.org/entity/Q36302922

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Helicobacter pylori DNA decrea ...... odium sulphate-induced colitis

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Helicobacter pylori DNA decrea ...... odium sulphate-induced colitis

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Helicobacter pylori DNA decrea ...... odium sulphate-induced colitis

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Helicobacter pylori DNA decrea ...... odium sulphate-induced colitis

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Helicobacter pylori DNA decrea ...... odium sulphate-induced colitis

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Helicobacter pylori DNA decrea ...... odium sulphate-induced colitis

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Helicobacter pylori DNA decrea ...... odium sulphate-induced colitis

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Anna V Pierzchala

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Chun-Chia Chen

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Jay Luther

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Joel H Rubenstein

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John Erb-Downward

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John Y Kao

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Jose A Paul

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Maochang Liu

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Min Zhang

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Stephanie Y Owyang

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P2860

Type I interferons for induction of remission in ulcerative colitis

An African origin for the intimate association between humans and Helicobacter pylori

Traces of human migrations in Helicobacter pylori populations

Helicobacter pylori's unconventional role in health and disease.

Mucosal FOXP3-expressing CD4+ CD25high regulatory T cells in Helicobacter pylori-infected patients

Toll-like receptor 9-induced type I IFN protects mice from experimental colitis.

Helicobacter pylori immune escape is mediated by dendritic cell-induced Treg skewing and Th17 suppression in mice.

Malignant tumors of the stomach. Gastric mucosa-associated lymphoid tissue lymphoma and Helicobacter pylori.

Identification of CpG oligonucleotide sequences with high induction of IFN-alpha/beta in plasmacytoid dendritic cells.

Development of ulcerative colitis in a patient with multiple sclerosis following treatment with interferon beta 1a.

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21471567

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Helicobacter pylori

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3466055

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