Modifying L-type calcium current kinetics: consequences for cardiac excitation and arrhythmia dynamics. - Wikidata - wikimedia - TriplyDB

Modifying L-type calcium current kinetics: consequences for cardiac excitation and arrhythmia dynamics.

Modifying L-type calcium current kinetics: consequences for cardiac excitation and arrhythmia dynamics.

http://www.wikidata.org/entity/Q36303246

about

Calmodulin regulation (calmodulation) of voltage-gated calcium channels Towards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium Channels Perspective: a dynamics-based classification of ventricular arrhythmias Suppression of re-entrant and multifocal ventricular fibrillation by the late sodium current blocker ranolazine Intra- and interdomain effects due to mutation of calcium-binding sites in calmodulin Voltage and calcium dynamics both underlie cellular alternans in cardiac myocytes.Mechanisms and determinants of ultralong action potential duration and slow rate-dependence in cardiac myocytes.The role of Purkinje-myocardial coupling during ventricular arrhythmia: a modeling study Biomaterial based cardiac tissue engineering and its applications Role of substrate and triggers in the genesis of cardiac alternans, from the myocyte to the whole heart: implications for therapy.Impaired Inactivation of L-Type Ca2+ Current as a Potential Mechanism for Variable Arrhythmogenic Liability of HERG K+ Channel Blocking Drugs.Dynamics of early afterdepolarization-mediated triggered activity in cardiac monolayers.Arrhythmogenesis in Timothy Syndrome is associated with defects in Ca(2+)-dependent inactivation Mechanisms Underlying AF: Triggers, Rotors, Other?The link between abnormal calcium handling and electrical instability in acquired long QT syndrome--Does calcium precipitate arrhythmic storms?Action potential dynamics explain arrhythmic vulnerability in human heart failure: a clinical and modeling study implicating abnormal calcium handling T-wave alternans as an arrhythmic risk stratifier: state of the art Accelerated inactivation of cardiac L-type calcium channels triggered by anaesthetic-induced preconditioning.Sensitivity of rabbit ventricular action potential and Ca²⁺ dynamics to small variations in membrane currents and ion diffusion coefficients.Increased susceptibility of aged hearts to ventricular fibrillation during oxidative stress.Rad GTPase deletion increases L-type calcium channel current leading to increased cardiac contraction.A translational approach to probe the proarrhythmic potential of cardiac alternans: a reversible overture to arrhythmogenesis?Optical mapping of sarcoplasmic reticulum Ca2+ in the intact heart: ryanodine receptor refractoriness during alternans and fibrillation.Spontaneous high-frequency action potential.Redox control of cardiac excitability.Bifurcation theory and cardiac arrhythmias.A rendezvous with the queen of ion channels: Three decades of ion channel research by David T Yue and his Calcium Signals Laboratory.Studying dyadic structure-function relationships: a review of current modeling approaches and new insights into Ca2+ (mis)handling.Rabbit-specific computational modelling of ventricular cell electrophysiology: Using populations of models to explore variability in the response to ischemia.Dynamic switching of calmodulin interactions underlies Ca2+ regulation of CaV1.3 channels.The role of fine-scale anatomical structure in the dynamics of reentry in computational models of the rabbit ventricles.Conservation of Ca2+/calmodulin regulation across Na and Ca2+ channels.Non-linear dynamics of cardiac alternans: subcellular to tissue-level mechanisms of arrhythmia.Mesenchymal stem cells suppress cardiac alternans by activation of PI3K mediated nitroso-redox pathway.Calmodulin mutations associated with long QT syndrome prevent inactivation of cardiac L-type Ca(2+) currents and promote proarrhythmic behavior in ventricular myocytes.Revealing kinetics and state-dependent binding properties of IKur-targeting drugs that maximize atrial fibrillation selectivity.Mechanisms of ventricular rate adaptation as a predictor of arrhythmic risk.Shaping a new Ca²⁺ conductance to suppress early afterdepolarizations in cardiac myocytes.Calcium-Dependent Arrhythmogenic Foci Created by Weakly Coupled Myocytes in the Failing Heart.Theoretical study of L-type Ca(2+) current inactivation kinetics during action potential repolarization and early afterdepolarizations.

P2860

Q26827282-3C215EAA-169C-4D1E-9994-5652A699BC25 Q27002550-C65ED4B3-CAFA-40FE-AA85-C108D6FE98E8 Q28082968-94859128-99FC-4E41-ABDA-ADC4FDD801B2 Q30622882-F65BAB18-8A8C-46C8-A767-0A2325936826 Q33707049-E2333E7C-4C5F-4F1F-8971-C8C30BF47DE4 Q33736840-C0E9618C-D402-4A47-906A-F142E206A306 Q34405346-81202B35-CF4E-42BB-9814-FCD008505A77 Q35091501-87068C2D-EFBD-41E8-A7B1-E693302E497D Q35731251-4BD41983-70F2-4931-ACD6-93FD4C942A04 Q35762284-45499FEB-E1CD-4B81-B060-9EB76C539EB3 Q35941355-C436F80D-9AF8-40C0-A838-A2171789A8A3 Q36045415-FC47967E-B62E-4083-855D-02798F2F7D9C Q36540252-7188B285-4D2D-4276-8998-CC0AE2576355 Q36709755-62DEB2F1-52AD-4D91-991F-FF0EB8E23022 Q36730574-75B2AB66-06AE-405E-9FF4-C0BE31F3F825 Q37037251-E47A8AD7-06CA-4DF4-95F5-9642EE00E0B6 Q37148999-78F69407-D73E-4302-876F-384F61F36CD9 Q37230651-6E38658C-986A-4844-99CE-70F11D492923 Q37270323-0A1A0A74-AEE3-4582-887C-B7CD46D87517 Q37430956-B7F9BEC3-2118-4E67-9AE6-CC10CF274A43 Q37457760-FC40FCF9-CF73-4986-B4F9-E6B3537379DA Q37576148-6A49A29D-3ACA-45C8-BC2C-D584CEF44B99 Q37724865-12E4727E-A586-4088-80B6-5A8E68650A98 Q37867552-4A3CE532-6C28-4E59-BEA0-1CDFD79206A6 Q38035054-2845E926-20EA-401E-B2C6-979B0718F528 Q38086408-D27FB33C-A592-4267-B688-DC15BBEB4DBD Q38547497-64486E4A-BFFB-4677-B7B4-4014E398DED4 Q38687960-AE9EB47F-693F-440F-BA33-53BC27325477 Q38869987-B798C474-E4FD-491D-B1E0-6505462DDED0 Q41881272-62F64D92-5C27-41FE-8B51-2562DC7E1A89 Q42019623-5DDED325-32BA-4913-B1FB-113E488AD540 Q42035315-30474A8F-5B5E-4A42-B83E-DBD11C7C4CE9 Q42064986-CF20C492-F74E-47B1-9FEF-F3EC29DBCF6F Q42353140-34BC533F-BFF1-48EA-83B5-C54A8800A1FA Q42551776-852775B8-AB0C-4897-94B6-57B3AED9E1E6 Q42694191-C8C761F2-3DEC-4674-B56D-527C1CF16A64 Q45764137-9A3B8D48-DEAD-4A8C-A087-3751206A5C4F Q46036586-3604D82F-E54F-4C0B-83B4-C17346A21923 Q46141791-397A1AE2-E229-409F-8B98-7B9EF0F20493 Q46393948-8B75AFD6-F248-45D5-97A5-C40A1774507E

P2860

Modifying L-type calcium current kinetics: consequences for cardiac excitation and arrhythmia dynamics.

http://www.wikidata.org/entity/Q36303246

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年学术文章

@wuu

2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh

2008年學術文章

@zh-hant

name

Modifying L-type calcium curre ...... ation and arrhythmia dynamics.

@ast

Modifying L-type calcium curre ...... ation and arrhythmia dynamics.

@en

type

label

Modifying L-type calcium curre ...... ation and arrhythmia dynamics.

@ast

Modifying L-type calcium curre ...... ation and arrhythmia dynamics.

@en

prefLabel

Modifying L-type calcium curre ...... ation and arrhythmia dynamics.

@ast

Modifying L-type calcium curre ...... ation and arrhythmia dynamics.

@en

P1433

Q36303246-146E022A-9225-4257-864B-39EBFFCA090A

P1476

Q36303246-6EB64C5B-5773-42ED-B0BF-723697A39B43

P2093

Q36303246-034A6258-A7B9-4C66-9206-5A7D4DB808B5

Q36303246-21487F5B-4E68-459D-B6AF-630EC24C52E9

Q36303246-249921DF-E99B-4469-B2B5-D00C4C86DFAC

Q36303246-28B534B1-7FED-404E-9728-18C4745F4EDF

Q36303246-42963834-0635-4697-B1C4-B4AE6C7F67B1

Q36303246-9A03607D-D6A1-4FA6-BB50-35F6CAD172A3

Q36303246-D78855BE-7779-4182-9699-4C3FBF1944EE

Q36303246-D9562201-34FC-40A6-9BC7-49CB9ACDB3DE

Q36303246-F993778D-BAD8-4A84-B441-B8EEDE590592

Q36303246-FEF9AB61-C062-48AB-8005-27E193072E51

P2860

Q36303246-0102E0AF-0E3B-4AB6-8122-CE811F191B02

Q36303246-056A0EE4-F99B-4F1C-9DFB-F63DA1593B21

Q36303246-19352B32-FAF3-475E-A77A-C7AE75445BFE

Q36303246-1B72D4FE-9FD7-4787-8914-71B63BAC4472

Q36303246-26F2777F-8630-45C6-9251-1DCDBA420062

Q36303246-4424312C-424D-463C-978A-3EC0C0BF4C2C

Q36303246-4B21BF85-DD3F-455C-9253-B70FBDE03871

Q36303246-4BF66D84-BD52-4C92-B5CE-C55B8C5FFCD9

Q36303246-5EBFE79C-B1D9-4D21-B3B8-D30FE7DA23FD

Q36303246-7D1AAF86-78AD-4CF5-8B1C-1FFCA846F1FB

P304

Q36303246-6F68C1CE-DA8C-498E-BAF7-D06782F03281

P31

Q36303246-57BC6146-1274-4FEF-80D6-BF5C92A8C8FF

P356

Q36303246-61DB8F77-5CEF-4F8C-8380-E43CF0A33E0C

P407

Q36303246-5E0ED62A-7887-4839-9A75-6543A66E0DDF

P433

Q36303246-AD4A15C0-C1D4-4A34-88A6-17238FF25832

P478

Q36303246-37939885-E371-441D-A786-E77C4F03D145

P577

Q36303246-AD0CFB32-1E6E-4ABE-92B0-0A51E5DC1C0B

P5875

Q36303246-65481F3D-A2BF-4234-B629-4D4A3EA444A8

P698

Q36303246-7B2BD87C-DBB3-4DEB-8630-79ADD0A7420F

P932

Q36303246-2CEECDB1-771B-4445-A684-8C5EC4ADF56C

P356

BIOPHYSJ.106.98590

P1433

Biophysical Journal

P1476

Modifying L-type calcium curre ...... ation and arrhythmia dynamics.

@en

P2093

Alan Garfinkel

http://www.w3.org/2001/XMLSchema#string

Ali Baher

http://www.w3.org/2001/XMLSchema#string

Aman Mahajan

http://www.w3.org/2001/XMLSchema#string

Daisuke Sato

http://www.w3.org/2001/XMLSchema#string

James N Weiss

http://www.w3.org/2001/XMLSchema#string

Lai-Hua Xie

http://www.w3.org/2001/XMLSchema#string

Riccardo Olcese

http://www.w3.org/2001/XMLSchema#string

Robert Peralta

http://www.w3.org/2001/XMLSchema#string

Yohannes Shiferaw

http://www.w3.org/2001/XMLSchema#string

Zhilin Qu

http://www.w3.org/2001/XMLSchema#string

P2860

Severe arrhythmia disorder caused by cardiac L-type calcium channel mutations

Fractional SR Ca release is regulated by trigger Ca and SR Ca content in cardiac myocytes

Model of intracellular calcium cycling in ventricular myocytes

A mathematical treatment of integrated Ca dynamics within the ventricular myocyte.

Engineered calmodulins reveal the unexpected eminence of Ca2+ channel inactivation in controlling heart excitation.

Voltage- and calcium-dependent inactivation in high voltage-gated Ca(2+) channels.

Ca2+ channel moving tail: link between Ca2+-induced inactivation and Ca2+ signal transduction.

A rabbit ventricular action potential model replicating cardiac dynamics at rapid heart rates

From pulsus to pulseless: the saga of cardiac alternans.

Critical mass hypothesis revisited: role of dynamical wave stability in spontaneous termination of cardiac fibrillation.

P304

411-423

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1529/BIOPHYSJ.106.98590

http://www.w3.org/2001/XMLSchema#string

P407

P433

2

http://www.w3.org/2001/XMLSchema#string

P478

94

http://www.w3.org/2001/XMLSchema#string

P577

2008-01-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

5688134

http://www.w3.org/2001/XMLSchema#string

P698

18160661

http://www.w3.org/2001/XMLSchema#string

P932

2157257

http://www.w3.org/2001/XMLSchema#string