The activation of AMPK in cardiomyocytes at the very early stage of hypoxia relies on an adenine nucleotide-independent mechanism.
about
Exposure to 15% oxygen in vivo up-regulates cardioprotective SUR2A without affecting ERK1/2 and AKT: a crucial role for AMPK.Thyrotropin and obesity: increased adipose triglyceride content through glycerol-3-phosphate acyltransferase 3.Increased expression of estrogen-related receptor β during adaptation of adult cardiomyocytes to sustained hypoxia
P2860
The activation of AMPK in cardiomyocytes at the very early stage of hypoxia relies on an adenine nucleotide-independent mechanism.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
The activation of AMPK in card ...... leotide-independent mechanism.
@ast
The activation of AMPK in card ...... leotide-independent mechanism.
@en
type
label
The activation of AMPK in card ...... leotide-independent mechanism.
@ast
The activation of AMPK in card ...... leotide-independent mechanism.
@en
prefLabel
The activation of AMPK in card ...... leotide-independent mechanism.
@ast
The activation of AMPK in card ...... leotide-independent mechanism.
@en
P2093
P2860
P1476
The activation of AMPK in card ...... leotide-independent mechanism.
@en
P2093
Dongxia Zhang
Qiong Zhang
Yuesheng Huang
P2860
P304
P577
2012-10-01T00:00:00Z