Hyaluronic acid, HAS1, and HAS2 are significantly upregulated during muscle hypertrophy.
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p38 MAPK signaling in postnatal tendon growth and remodelingDiscoidin domain receptor 1 contributes to tumorigenesis through modulation of TGFBI expression.T lymphocytes are not required for the development of fatty degeneration after rotator cuff tear.Synergist ablation induces rapid tendon growth through the synthesis of a neotendon matrixHighlights of glycosylation and adhesion related genes involved in myogenesis.Extracellular component hyaluronic acid and its receptor Hmmr are required for epicardial EMT during heart regeneration.Hyaluronan synthesis and myogenesis: a requirement for hyaluronan synthesis during myogenic differentiation independent of pericellular matrix formationThe role of hyaluronan in the pathobiology and treatment of respiratory disease.Targeted inhibition of TGF-β results in an initial improvement but long-term deficit in force production after contraction-induced skeletal muscle injury.MMP inhibition as a potential method to augment the healing of skeletal muscle and tendon extracellular matrix.Periostin induces intracellular cross-talk between kinases and hyaluronan in atrioventricular valvulogenesis.Platelet-Rich Plasma Activates Proinflammatory Signaling Pathways and Induces Oxidative Stress in Tendon Fibroblasts.Biomimetic Material-Assisted Delivery of Human Embryonic Stem Cell Derivatives for Enhanced In Vivo Survival and Engraftment.Matrix metalloproteinase-2 plays a critical role in overload induced skeletal muscle hypertrophy.Matrix metalloproteinase-2 plays a critical role in overload induced skeletal muscle hypertrophy.Apparent Diffusion Coefficient (ADC) predicts therapy response in pancreatic ductal adenocarcinoma.Glucocorticoids Improve Myogenic Differentiation In Vitro by Suppressing the Synthesis of Versican, a Transitional Matrix Protein Overexpressed in Dystrophic Skeletal Muscles.Pirfenidone reduces subchondral bone loss and fibrosis after murine knee cartilage injury.Changes in muscle fiber contractility and extracellular matrix production during skeletal muscle hypertrophy.Inhibition of platelet-derived growth factor signaling prevents muscle fiber growth during skeletal muscle hypertrophy.Notes for genera: Ascomycota
P2860
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P2860
Hyaluronic acid, HAS1, and HAS2 are significantly upregulated during muscle hypertrophy.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Hyaluronic acid, HAS1, and HAS2 are significantly upregulated during muscle hypertrophy.
@ast
Hyaluronic acid, HAS1, and HAS2 are significantly upregulated during muscle hypertrophy.
@en
type
label
Hyaluronic acid, HAS1, and HAS2 are significantly upregulated during muscle hypertrophy.
@ast
Hyaluronic acid, HAS1, and HAS2 are significantly upregulated during muscle hypertrophy.
@en
prefLabel
Hyaluronic acid, HAS1, and HAS2 are significantly upregulated during muscle hypertrophy.
@ast
Hyaluronic acid, HAS1, and HAS2 are significantly upregulated during muscle hypertrophy.
@en
P2093
P2860
P1476
Hyaluronic acid, HAS1, and HAS2 are significantly upregulated during muscle hypertrophy.
@en
P2093
Jahdonna Isaac
Jonathan P Gumucio
Sarah Calve
P2860
P304
P356
10.1152/AJPCELL.00057.2012
P577
2012-07-11T00:00:00Z