Immune response: the key to bone resorption in periodontal disease. - Wikidata - wikimedia - TriplyDB

Immune response: the key to bone resorption in periodontal disease.

Immune response: the key to bone resorption in periodontal disease.

http://www.wikidata.org/entity/Q36309235

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Management of diabolical diabetes mellitus and periodontitis nexus: Are we doing enough?Periodontitis and bone metabolism Association between Periodontitis and Alzheimer's Disease Animal Models for Periodontal Disease Porphyromonas gingivalis Periodontal Infection and Its Putative Links with Alzheimer's Disease Melatonin: a novel indolamine in oral health and disease RNA-sequencing study of peripheral blood monocytes in chronic periodontitis DNA from Porphyromonas gingivalis and Tannerella forsythia induce cytokine production in human monocytic cell lines.Histopathologic and histomorphometric studies and determination of IL-8 in patients with periodontal disease Sex steroids, periodontal health, and tooth loss in older men A novel murine model for chronic inflammatory alveolar bone loss.Breaking the Gingival Epithelial Barrier: Role of the Aggregatibacter actinomycetemcomitans Cytolethal Distending Toxin in Oral Infectious Disease.Impact of the Oral Commensal Flora on Alveolar Bone Homeostasis.Antigen-presenting cell marker expression and phagocytotic activity in periodontal ligament cells.Platelet rich fibrin (Prf) and β-tricalcium phosphate with coronally advanced flap for the management of grade-II furcation defect.Cytokine gene expression profiles during initiation, progression and resolution of periodontitis.Porphyromonas gingivalis exacerbates ligature-induced, RANKL-dependent alveolar bone resorption via differential regulation of Toll-like receptor 2 (TLR2) and TLR4.Osteoclast activated FoxP3+ CD8+ T-cells suppress bone resorption in vitro.Antibody to receptor activator of NF-κB ligand ameliorates T cell-mediated periodontal bone resorption.Mobilization of endothelial progenitors by recurrent bacteremias with a periodontal pathogen.Proresolving nanomedicines activate bone regeneration in periodontitis.Role of periodontal pathogenic bacteria in RANKL-mediated bone destruction in periodontal disease.Anti-inflammatory effect of MAPK phosphatase-1 local gene transfer in inflammatory bone loss.A comparison of Aggregatibacter actinomycetemcomitans (Aa) virulence traits in a rat model for periodontal disease.Metabolic syndrome exacerbates inflammation and bone loss in periodontitis Selective serotonin reuptake inhibitors attenuate the antigen presentation from dendritic cells to effector T lymphocytes.Cytolethal distending toxin damages the oral epithelium of gingival explants The role of RANK/RANKL/OPG signalling pathways in osteoclastogenesis in odontogenic keratocysts, radicular cysts, and ameloblastomas.IFN-gamma stimulates osteoclast formation and bone loss in vivo via antigen-driven T cell activation.The role of polyunsaturated ω-3 fatty acid eicosapentaenoic acid-derived resolvin E1 (RvE1) in bone preservation.Aggregatibacter actinomycetemcomitans leukotoxin: a powerful tool with capacity to cause imbalance in the host inflammatory response Enhancement of periodontal tissue regeneration by transplantation of osteoprotegerin-engineered periodontal ligament stem cells IL-12 Inhibits Lipopolysaccharide Stimulated Osteoclastogenesis in Mice.Mechanisms of Bone Resorption in Periodontitis.Inflammation Biomarkers of Advanced Disease in Nongingival Tissues of Chronic Periodontitis Patients.Receptor activator of NF-kappaB ligand (RANKL) and CD 31 expressions in chronic periodontitis patients before and after surgery.Study of the association between the interleukin-1 β c.3954C>T polymorphism and periodontitis in a population sample from Bahia, Brazil MAPK usage in periodontal disease progression.An essential role for IL-17 in preventing pathogen-initiated bone destruction: recruitment of neutrophils to inflamed bone requires IL-17 receptor-dependent signals.An oligodeoxynucleotide that induces differentiation of bone marrow mesenchymal stem cells to osteoblasts in vitro and reduces alveolar bone loss in rats with periodontitis

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P2860

Immune response: the key to bone resorption in periodontal disease.

http://www.wikidata.org/entity/Q36309235

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2005 nî lūn-bûn

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2005年学术文章

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2005年學術文章

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2005年學術文章

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Immune response: the key to bone resorption in periodontal disease.

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Immune response: the key to bone resorption in periodontal disease.

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Immune response: the key to bone resorption in periodontal disease.

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Immune response: the key to bone resorption in periodontal disease.

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Immune response: the key to bone resorption in periodontal disease.

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JOP.2005.76.11-S.2033

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Journal of Periodontology

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Immune response: the key to bone resorption in periodontal disease.

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Martin A Taubman

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Paloma Valverde

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Toshihisa Kawai

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Xiaozhe Han

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P2860

Selective blockade of T lymphocyte K(+) channels ameliorates experimental autoimmune encephalomyelitis, a model for multiple sclerosis

Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL

RANK-L and RANK: T cells, bone loss, and mammalian evolution

Potassium channel-blockers as therapeutic agents to interfere with bone resorption of periodontal disease

TH1 and TH2 cells: different patterns of lymphokine secretion lead to different functional properties

Activated T cells regulate bone loss and joint destruction in adjuvant arthritis through osteoprotegerin ligand

Expression of major histocompatibility complex class II and CD80 by gingival epithelial cells induces activation of CD4+ T cells in response to bacterial challenge

Involvement of T-lymphocytes in periodontal disease and in direct and indirect induction of bone resorption.

Infection by Streptococcus pyogenes induces the receptor activator of NF-kappaB ligand expression in mouse osteoblastic cells.

Functional human T-cell immunity and osteoprotegerin ligand control alveolar bone destruction in periodontal infection.

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