Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction. - Wikidata - wikimedia - TriplyDB

Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.

Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.

http://www.wikidata.org/entity/Q36309320

about

Functional O-GlcNAc modifications: implications in molecular regulation and pathophysiology O-GlcNAcase: promiscuous hexosaminidase or key regulator of O-GlcNAc signaling?Post-translational modifications of the cardiac proteome in diabetes and heart failure Activation of AKT by O-linked N-acetylglucosamine induces vascular calcification in diabetes mellitus Proteasomal degradation of O-GlcNAc transferase elevates hypoxia-induced vascular endothelial inflammatory response†.Protein O-GlcNAcylation and cardiovascular (patho)physiology.O-GlcNAcylation and Inflammation: A Vast Territory to Explore Alterations in left ventricular function during intermittent hypoxia: Possible involvement of O-GlcNAc protein and MAPK signaling.Pharmacological Inhibition of O-GlcNAcase Does Not Increase Sensitivity of Glucocorticoid Receptor-Mediated Transrepression.O-GlcNAcase overexpression reverses coronary endothelial cell dysfunction in type 1 diabetic mice.Chitosan oligosaccharides block LPS-induced O-GlcNAcylation of NF-κB and endothelial inflammatory response.O-GlcNAc and the cardiovascular system.Retinol-binding protein 7 is an endothelium-specific PPARγ cofactor mediating an antioxidant response through adiponectin.New insights: A role for O-GlcNAcylation in diabetic complications.O-GlcNAc protein modification in C2C12 myoblasts exposed to oxidative stress indicates parallels with endogenous antioxidant defense.O-GlcNAcylation and cardiovascular disease.High-fat diet increases O-GlcNAc levels in cerebral arteries: a link to vascular dysfunction associated with hyperlipidaemia/obesity?O-GlcNAcylation as a novel ammonia-induced posttranslational protein modification in cultured rat astrocytes.Glycosylation with O-Linked β-N-acetylglucosamine (O-GlcNAc) induces vascular dysfunction via production of superoxide anion / reactive oxygen species.Antrodia cinnamomea Oligosaccharides Suppress Lipopolysaccharide-Induced Inflammation through Promoting O-GlcNAcylation and Repressing p38/Akt Phosphorylation.Thiamet G mediates neuroprotection in experimental stroke by modulating microglia/macrophage polarization and inhibiting NF-κB p65 signaling.The Role of Stress-Induced O-GlcNAc Protein Modification in the Regulation of Membrane Transport.MicroRNA-200a/200b Modulate High Glucose-Induced Endothelial Inflammation by Targeting O-linked N-Acetylglucosamine Transferase Expression.Protein O-GlcNAc Modification Increases in White Blood Cells After a Single Bout of Physical Exercise.

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Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.

http://www.wikidata.org/entity/Q36309320

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

@wuu

2012年学术文章

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2012年学术文章

@zh-hans

2012年学术文章

@zh-my

2012年学术文章

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2012年學術文章

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2012年學術文章

@zh

2012年學術文章

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name

Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.

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Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.

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type

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Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.

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Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.

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prefLabel

Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.

@ast

Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.

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AJPHEART.01175.2011

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American Journal of Physiology - Heart and Circulatory Physiology

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Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.

@en

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Dongqi Xing

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John C Chatham

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Kaizheng Gong

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Rob H P Hilgers

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Suzanne Oparil

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Yiu-Fai Chen

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A potent mechanism-inspired O-GlcNAcase inhibitor that blocks phosphorylation of tau in vivo

Coordinated regulation of glutamine:fructose-6-phosphate amidotransferase activity by insulin, glucose, and glutamine. Role of hexosamine biosynthesis in enzyme regulation

Cycling of O-linked beta-N-acetylglucosamine on nucleocytoplasmic proteins

Increased O-GlcNAc levels during reperfusion lead to improved functional recovery and reduced calpain proteolysis

O-linked β-N-acetylglucosamine supports p38 MAPK activation by high glucose in glomerular mesangial cells

Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associated O-GlcNAc

Impaired vasodilator activity in deoxycorticosterone acetate-salt hypertension is associated with increased protein O-GlcNAcylation

Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signaling

Peroxynitrite-mediated tyrosine nitration catalyzed by superoxide dismutase.

NG-methyl-L-arginine inhibits tumor necrosis factor-induced hypotension: implications for the involvement of nitric oxide.

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10.1152/AJPHEART.01175.2011

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22777418

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