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Disruption of a Regulatory Network Consisting of Neutrophils and Platelets Fosters Persisting Inflammation in Rheumatic DiseasesThe role of high mobility group box 1 (HMGB1) in the pathogenesis of kidney diseasesImmunosenescence in renal transplantation: a changing balance of innate and adaptive immunityOxidant Mechanisms in Renal Injury and DiseaseCellular and Molecular Mechanisms of AKI.HMGB1 in health and disease.HMGB1 exacerbates renal tubulointerstitial fibrosis through facilitating M1 macrophage phenotype at the early stage of obstructive injury.Role of high-mobility group box-1 in myocardial ischemia/reperfusion injury and the effect of ethyl pyruvate.Continuous hemodiafiltration therapy reduces damage of multi-organs by ameliorating of HMGB1/TLR4/NFκB in a dog sepsis modelCharting the course of renal cryoinjury.Serum-stabilized naked caspase-3 siRNA protects autotransplant kidneys in a porcine model.High-mobility group box 1 inhibits HCO(3)(-) absorption in medullary thick ascending limb through a basolateral receptor for advanced glycation end products pathwayThe Effect of the Antioxidant Drug "U-74389G" on Creatinine Levels during Ischemia Reperfusion Injury in Rats.Toll-like receptor 4-induced IL-22 accelerates kidney regeneration.Redox modulation of HMGB1-related signaling.Processes of sterile inflammation.Inflammatory triggers of acute rejection of organ allografts.Role of high mobility group box 1 and its signaling pathways in renal diseases.Beyond tissue injury-damage-associated molecular patterns, toll-like receptors, and inflammasomes also drive regeneration and fibrosis.High-mobility group box-1 in sterile inflammation.The cell secretome, a mediator of cell-to-cell communication.Role of TLRs and DAMPs in allograft inflammation and transplant outcomes.Innate immune mechanisms in transplant allograft vasculopathy.The in vivo evidence for regulated necrosis.Inflammasomes in the urinary tract: a disease-based review.Ethyl pyruvate prevents methyglyoxal-induced retinal vascular injury in rats.High-mobility group box 1 inhibits HCO3- absorption in the medullary thick ascending limb through RAGE-Rho-ROCK-mediated inhibition of basolateral Na+/H+ exchange.High-mobility group box 1 is involved in the development of benign prostatic hyperplasia with chronic prostatic inflammation.HMGB1 redox during sepsisReduced HMGB 1-Mediated Pathway and Oxidative Stress in Resveratrol-Treated Diabetic Mice: A Possible Mechanism of Cardioprotection of Resveratrol in Diabetes MellitusHigh-mobility group box 1 is a novel deacetylation target of Sirtuin1.Necrostatin-1 inhibits Hmgb1-IL-23/IL-17 pathway and attenuates cardiac ischemia reperfusion injury.Serum levels of high mobility group box 1 correlate with disease severity in recessive dystrophic epidermolysis bullosa.Carbon monoxide inhibits the nuclear-cytoplasmic translocation of HMGB1 in an in vitro oxidative stress injury model of mouse renal tubular epithelial cells.Allopurinol preconditioning attenuates renal ischemia/reperfusion injury by inhibiting HMGB1 expression in a rat model.
P2860
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P2860
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
HMGB1 in renal ischemic injury.
@ast
HMGB1 in renal ischemic injury.
@en
type
label
HMGB1 in renal ischemic injury.
@ast
HMGB1 in renal ischemic injury.
@en
prefLabel
HMGB1 in renal ischemic injury.
@ast
HMGB1 in renal ischemic injury.
@en
P2093
P2860
P1476
HMGB1 in renal ischemic injury.
@en
P2093
Brian B Ratliff
May M Rabadi
Michael S Goligorksy
Tammer Ghaly
P2860
P304
P356
10.1152/AJPRENAL.00092.2012
P577
2012-07-03T00:00:00Z