Modulation of host innate immune response in the bladder by uropathogenic Escherichia coli.
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Subversion of Host Innate Immunity by Uropathogenic Escherichia coliControversies and challenges in research on urogenital schistosomiasis-associated bladder cancerA novel mouse model of Schistosoma haematobium egg-induced immunopathologyReciprocal regulation between proinflammatory cytokine-induced inducible NO synthase (iNOS) and connexin43 in bladder smooth muscle cellsEscherichia coli colonizing the neurogenic bladder are similar to widespread clones causing disease in patients with normal bladder function.Waging war against uropathogenic Escherichia coli: winning back the urinary tractG-CSF induction early in uropathogenic Escherichia coli infection of the urinary tract modulates host immunity.Attenuation of human neutrophil migration and function by uropathogenic bacteria.Comparison of host response mechanisms evoked by extended spectrum beta lactamase (ESBL)--and non-ESBL-producing uropathogenic E. coli.Human bladder uroepithelial cells synergize with monocytes to promote IL-10 synthesis and other cytokine responses to uropathogenic Escherichia coli.Expression and antimicrobial function of beta-defensin 1 in the lower urinary tractThe UPEC pore-forming toxin α-hemolysin triggers proteolysis of host proteins to disrupt cell adhesion, inflammatory, and survival pathways.Role of Uropathogenic Escherichia coli Virulence Factors in Development of Urinary Tract Infection and Kidney Damage.ATG16L1 deficiency in macrophages drives clearance of uropathogenic E. coli in an IL-1β-dependent mannerInduction of indoleamine 2,3-dioxygenase by uropathogenic bacteria attenuates innate responses to epithelial infection.Genome-wide mapping of cystitis due to Streptococcus agalactiae and Escherichia coli in mice identifies a unique bladder transcriptome that signifies pathogen-specific antimicrobial defense against urinary tract infection.A Murine Model for Escherichia coli Urinary Tract Infection.YbcL of uropathogenic Escherichia coli suppresses transepithelial neutrophil migration.Cytotoxic necrotizing factor 1 and hemolysin from uropathogenic Escherichia coli elicit different host responses in the murine bladderInactivation of host Akt/protein kinase B signaling by bacterial pore-forming toxins.Association of O-Antigen Serotype with the Magnitude of Initial Systemic Cytokine Responses and Persistence in the Urinary Tract.Molecular basis of uropathogenic Escherichia coli evasion of the innate immune response in the bladder.Host-pathogen checkpoints and population bottlenecks in persistent and intracellular uropathogenic Escherichia coli bladder infection.Drug and Vaccine Development for the Treatment and Prevention of Urinary Tract Infections.Molecular epidemiology and phylogenetic distribution of the Escherichia coli pks genomic island.Uropathogenic Escherichia coli-Associated ExotoxinsAbnormal immunological profile and vaginal microbiota in women prone to urinary tract infections.TLR-mediated immune responses in the urinary tract.Urothelial cultures support intracellular bacterial community formation by uropathogenic Escherichia coliUropathogenic Escherichia coli-induced inflammation alters mouse urinary bladder contraction via an interleukin-6-activated inducible nitric oxide synthase-related pathway.Interleukin-8 is essential for normal urothelial cell survival.Lipopolysaccharide Domains Modulate Urovirulence.Quantitative assessment of human neutrophil migration across a cultured bladder epitheliumThe Adherent/Invasive Escherichia coli Strain LF82 Invades and Persists in Human Prostate Cell Line RWPE-1, Activating a Strong Inflammatory Response.IL-8 and global gene expression analysis define a key role of ATP in renal epithelial cell responses induced by uropathogenic bacteria.Immunity to uropathogens: the emerging roles of inflammasomes.Protein-based profiling of the immune response to uropathogenic Escherichia coli in adult patients immediately following hospital admission for acute cystitis.Granulocyte-macrophage colony-stimulating factor (GM-CSF) is released by female mouse bladder urothelial cells and expressed by the urothelium as an early response to lipopolysaccharides (LPS).Uropathogenic Escherichia coli Engages CD14-Dependent Signaling to Enable Bladder-Macrophage-Dependent Control of Acute Urinary Tract Infection.A novel ex vivo set-up for dynamic long-term characterization of processes on mucosal interfaces by confocal imaging and simultaneous cytokine measurements.
P2860
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P2860
Modulation of host innate immune response in the bladder by uropathogenic Escherichia coli.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Modulation of host innate immu ...... ropathogenic Escherichia coli.
@ast
Modulation of host innate immu ...... ropathogenic Escherichia coli.
@en
type
label
Modulation of host innate immu ...... ropathogenic Escherichia coli.
@ast
Modulation of host innate immu ...... ropathogenic Escherichia coli.
@en
prefLabel
Modulation of host innate immu ...... ropathogenic Escherichia coli.
@ast
Modulation of host innate immu ...... ropathogenic Escherichia coli.
@en
P2093
P2860
P356
P1476
Modulation of host innate immu ...... ropathogenic Escherichia coli.
@en
P2093
Anthony J Schaeffer
Benjamin K Billips
David J Klumpp
Matthew T Rycyk
Sarah G Forrestal
P2860
P304
P356
10.1128/IAI.00922-07
P407
P577
2007-08-27T00:00:00Z