Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective. - Wikidata - wikimedia - TriplyDB

Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective.

Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective.

http://www.wikidata.org/entity/Q36314908

about

Podometrics as a Potential Clinical Tool for Glomerular Disease Management Altered lipid metabolism in the aging kidney identified by three layered omic analysis Digital pathology imaging as a novel platform for standardization and globalization of quantitative nephropathology.One Year of Enzyme Replacement Therapy Reduces Globotriaosylceramide Inclusions in Podocytes in Male Adult Patients with Fabry Disease.Podocyte Depletion in Thin GBM and Alport Syndrome.Urinary Podocyte Loss Is Increased in Patients with Fabry Disease and Correlates with Clinical Severity of Fabry Nephropathy Alteration of podocyte phenotype in the urine of women with preeclampsia Drug discovery in focal and segmental glomerulosclerosis Quantitative podocyte parameters predict human native kidney and allograft half-lives.A previously unrecognized role of C3a in proteinuric progressive nephropathy.MAGI-1 Interacts with Nephrin to Maintain Slit Diaphragm Structure through Enhanced Rap1 Activation in Podocytes.Compound effects of aging and experimental FSGS on glomerular epithelial cells Podocyte number and density changes during early human life.Digital pathology in nephrology clinical trials, research, and pathology practice.Accelerated podocyte detachment and progressive podocyte loss from glomeruli with age in Alport Syndrome.Links between coagulation, inflammation, regeneration, and fibrosis in kidney pathology.Are podocytes motile?Reduction in podocyte SIRT1 accelerates kidney injury in aging mice.Targeting mTOR Signaling Can Prevent the Progression of FSGS.Can podocytes be regenerated in adults?Parietal cells-new perspectives in glomerular disease.Quantifying podocyte depletion: theoretical and practical considerations.Human podocyte depletion in association with older age and hypertension.Single-Nephron Glomerular Filtration Rate in Healthy Adults.An endoplasmic reticulum stress-regulated lncRNA hosting a microRNA megacluster induces early features of diabetic nephropathy.Association of Increasing GFR with Change in Albuminuria in the General Population.Relationships among injury, fibrosis, and time in human kidney transplants.Validation of a Three-Dimensional Method for Counting and Sizing Podocytes in Whole Glomeruli.The diabetes pandemic suggests unmet needs for 'CKD with diabetes' in addition to 'diabetic nephropathy'-implications for pre-clinical research and drug testing.Reducing mTOR augments parietal epithelial cell density in a model of acute podocyte depletion and in aged kidneys.WT1 Is Necessary for the Proliferation and Migration of Cells of Renin Lineage Following Kidney Podocyte Depletion.Glomerular Effects of Age and APOL1.An inducible mouse model of podocin-mutation-related nephrotic syndrome.Structural and Functional Changes in Human Kidneys with Healthy Aging.The Renal Pathology of Obesity.Shiga toxin triggers endothelial and podocyte injury: the role of complement activation.Differentiating Primary, Genetic, and Secondary FSGS in Adults: A Clinicopathologic Approach.Cytochrome P450-2E1 is involved in aging-related kidney damage in mice through increased nitroxidative stress.FSGS as an Adaptive Response to Growth-Induced Podocyte Stress.A dual agonist of farnesoid X receptor (FXR) and the G protein-coupled receptor TGR5, INT-767, reverses age-related kidney disease in mice.

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Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective.

http://www.wikidata.org/entity/Q36314908

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2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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2015年學術文章

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2015年學術文章

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Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective.

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Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective.

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Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective.

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Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective.

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Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective.

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P1433

Journal of the American Society of Nephrology

P1476

Glomerular Aging and Focal Global Glomerulosclerosis: A Podometric Perspective.

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P2093

Christopher O'Connor

http://www.w3.org/2001/XMLSchema#string

Chrysta Meadowbrooke

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Farsad Afshinnia

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Jeffrey B Hodgin

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Jocelyn E Wiggins

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Larysa Wickman

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Mahboob Chowdhury

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Markus Bitzer

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Masao Kikuchi

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Su Q Wang

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P2860

Economy, speed and size matter: evolutionary forces driving nuclear genome miniaturization and expansion

NFkappaB promotes inflammation, coagulation, and fibrosis in the aging glomerulus.

The association between age and nephrosclerosis on renal biopsy among healthy adults

Vascular contributions to cognitive impairment and dementia: a statement for healthcare professionals from the american heart association/american stroke association.

Podocyte is the major culprit accounting for the progression of chronic renal disease.

Age changes in glomerular filtration rate, effective renal plasma flow, and tubular excretory capacity in adult males

The two kidney to one kidney transition and transplant glomerulopathy: a podocyte perspective

Age-related incidence of sclerotic glomeruli in human kidneys

Urinary excretion of viable podocytes in health and renal disease.

Pathways to nephron loss starting from glomerular diseases-insights from animal models.

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3162-3178

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scholarly article

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10.1681/ASN.2014080752

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12

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26

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Roger C Wiggins

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2015-06-02T00:00:00Z

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26038526

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4657829

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