Loss of perivascular Kir4.1 potassium channels in the sclerotic hippocampus of patients with mesial temporal lobe epilepsy.
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Transplantation of stem cell-derived astrocytes for the treatment of amyotrophic lateral sclerosis and spinal cord injuryRole of astrocytes in epilepsyPhysiological roles of aquaporin-4 in brainKIR4.1: K+ Channel Illusion or Reality in the Autoimmune Pathogenesis of Multiple SclerosisHomeostasis or channelopathy? Acquired cell type-specific ion channel changes in temporal lobe epilepsy and their antiepileptic potentialRegulation of neurovascular coupling in autoimmunity to water and ion channelsPostnatal development of the molecular complex underlying astrocyte polarization.Novel KCNJ10 Gene Variations Compromise Function of Inwardly Rectifying Potassium Channel 4.1.Dystrophin Distribution and Expression in Human and Experimental Temporal Lobe Epilepsy.Regulation of astrocyte glutamine synthetase in epilepsyNovel astrocyte targets: new avenues for the therapeutic treatment of epilepsyEpilepsy surgery and the evolution of clinical and translational science.Transcriptome analysis suggests a role for the differential expression of cerebral aquaporins and the MAPK signalling pathway in human temporal lobe epilepsy.Astroglial Scarring and Seizures: A Cell Biological Perspective on Epilepsy.Potassium Channels in Epilepsy.Augmentation of Ca(2+) signaling in astrocytic endfeet in the latent phase of temporal lobe epilepsy.Human astrocytes in the diseased brain.Turning down the volume: Astrocyte volume change in the generation and termination of epileptic seizures.Expressional analysis of the astrocytic Kir4.1 channel in a pilocarpine-induced temporal lobe epilepsy model.A Nanoscale Interface Promoting Molecular and Functional Differentiation of Neural Cells.Reassessing the role of astrocytes in ammonia neurotoxicity.Inhibition of Inwardly Rectifying Potassium (Kir) 4.1 Channels Facilitates Brain-Derived Neurotrophic Factor (BDNF) Expression in Astrocytes.Astrogliosis and impaired aquaporin-4 and dystrophin systems in idiopathic normal pressure hydrocephalus.Hippocampal "gliosis only" on MR imaging represents a distinct entity in epilepsy patients.Pathophysiological Characteristics Associated With Epileptogenesis in Human Hippocampal Sclerosis.Astrocytic Kir4.1 channels and gap junctions account for spontaneous epileptic seizure.Differential loss of KIR4.1 immunoreactivity in multiple sclerosis lesionsAntiepileptic Drugs Elevate Astrocytic Kir4.1 Expression in the Rat Limbic Region
P2860
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P2860
Loss of perivascular Kir4.1 potassium channels in the sclerotic hippocampus of patients with mesial temporal lobe epilepsy.
description
2012 nî lūn-bûn
@nan
2012年の論文
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2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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2012年學術文章
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name
Loss of perivascular Kir4.1 po ...... mesial temporal lobe epilepsy.
@ast
Loss of perivascular Kir4.1 po ...... mesial temporal lobe epilepsy.
@en
type
label
Loss of perivascular Kir4.1 po ...... mesial temporal lobe epilepsy.
@ast
Loss of perivascular Kir4.1 po ...... mesial temporal lobe epilepsy.
@en
prefLabel
Loss of perivascular Kir4.1 po ...... mesial temporal lobe epilepsy.
@ast
Loss of perivascular Kir4.1 po ...... mesial temporal lobe epilepsy.
@en
P2093
P2860
P50
P1476
Loss of perivascular Kir4.1 po ...... mesial temporal lobe epilepsy.
@en
P2093
Anna E Thoren
Dennis D Spencer
Fredrik Lauritzen
Gry F Vindedal
Kjell Heuser
Nihal C de Lanerolle
P2860
P304
P356
10.1097/NEN.0B013E318267B5AF
P577
2012-09-01T00:00:00Z