Macrophage origin limits functional plasticity in helminth-bacterial co-infection
about
Mannose receptor high, M2 dermal macrophages mediate nonhealing Leishmania major infection in a Th1 immune environment.The Transcription Factor STAT6 Mediates Direct Repression of Inflammatory Enhancers and Limits Activation of Alternatively Polarized Macrophages.Growth of Mycobacterium tuberculosis in vivo segregates with host macrophage metabolism and ontogeny.
P2860
Macrophage origin limits functional plasticity in helminth-bacterial co-infection
description
2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年學術文章
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2017年學術文章
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2017年學術文章
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name
Macrophage origin limits functional plasticity in helminth-bacterial co-infection
@ast
Macrophage origin limits functional plasticity in helminth-bacterial co-infection
@en
type
label
Macrophage origin limits functional plasticity in helminth-bacterial co-infection
@ast
Macrophage origin limits functional plasticity in helminth-bacterial co-infection
@en
prefLabel
Macrophage origin limits functional plasticity in helminth-bacterial co-infection
@ast
Macrophage origin limits functional plasticity in helminth-bacterial co-infection
@en
P2093
P2860
P50
P1433
P1476
Macrophage origin limits functional plasticity in helminth-bacterial co-infection
@en
P2093
Rick M Maizels
Sharon M Campbell
Sheelagh Duncan
Tom A Barr
P2860
P304
P356
10.1371/JOURNAL.PPAT.1006233
P50
P577
2017-03-23T00:00:00Z