KLF6-SV1 drives breast cancer metastasis and is associated with poor survival - Wikidata - wikimedia - TriplyDB

KLF6-SV1 drives breast cancer metastasis and is associated with poor survival

KLF6-SV1 drives breast cancer metastasis and is associated with poor survival

http://www.wikidata.org/entity/Q36324431

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A novel KLF6-Rho GTPase axis regulates hepatocellular carcinoma cell migration and dissemination KLF6 depletion promotes NF-κB signaling in glioblastoma.Prognostic cancer gene signatures share common regulatory motifs Identification of miRNAs that specifically target tumor suppressive KLF6-FL rather than oncogenic KLF6-SV1 isoform.Expression profiling of migrated and invaded breast cancer cells predicts early metastatic relapse and reveals Krüppel-like factor 9 as a potential suppressor of invasive growth in breast cancer.A molecular analysis provides novel insights into androgen receptor signalling in breast cancer.Alternative RNA splicing and cancer Landscape of expression profiles in esophageal carcinoma by The Cancer Genome Atlas data.Increased H3K9me3 drives dedifferentiated phenotype via KLF6 repression in liposarcoma.A summary of relationships between alternative splicing and breast cancer.Oncogenic alternative splicing switches: role in cancer progression and prospects for therapy.KLF4α stimulates breast cancer cell proliferation by acting as a KLF4 antagonist.Krüppel-like factors in cancer progression: three fingers on the steering wheel.Kruppel-like factor 6 suppresses growth and invasion of hepatocellular carcinoma cells in vitro and in vivo.Long-term soy consumption and tumor tissue MicroRNA and gene expression in triple-negative breast cancer.A splicing twist on metastasis.Alternative Splicing in Breast Cancer and the Potential Development of Therapeutic Tools.Targeted knockdown of DJ-1 induces multiple myeloma cell death via KLF6 upregulation.Autologous T cells expressing the oncogenic transcription factor KLF6-SV1 prevent apoptosis of chronic lymphocytic leukemia cells.Alternative-splicing defects in cancer: Splicing regulators and their downstream targets, guiding the way to novel cancer therapeutics.SRrp35 suppresses cell proliferation and malignancy in hepatocellular carcinoma.Overexpression of the Oncogenic Variant (KLF6-SV1) in Young NPC Patients and Correlation with Lack of E-Cadherin.KLF6-SV1 is a new prognostic biomarker in postoperative patients with non-small cell lung cancer

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P2860

KLF6-SV1 drives breast cancer metastasis and is associated with poor survival

http://www.wikidata.org/entity/Q36324431

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2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

@zh-sg

2013年學術文章

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2013年學術文章

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2013年學術文章

@zh-hant

name

KLF6-SV1 drives breast cancer metastasis and is associated with poor survival

@ast

KLF6-SV1 drives breast cancer metastasis and is associated with poor survival

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type

label

KLF6-SV1 drives breast cancer metastasis and is associated with poor survival

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KLF6-SV1 drives breast cancer metastasis and is associated with poor survival

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prefLabel

KLF6-SV1 drives breast cancer metastasis and is associated with poor survival

@ast

KLF6-SV1 drives breast cancer metastasis and is associated with poor survival

@en

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SCITRANSLMED.3004688

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Science Translational Medicine

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KLF6-SV1 drives breast cancer metastasis and is associated with poor survival

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Alexander Kirschenbaum

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Alice C Levine

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Analisa DiFeo

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Anieta M Sieuwerts

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David Burstein

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Doris Germain

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Goutham Narla

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Jessica Ohlssen

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John A Foekens

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Luena Papa

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P2860

Induction of EMT by twist proteins as a collateral effect of tumor-promoting inactivation of premature senescence

Direct regulation of TWIST by HIF-1alpha promotes metastasis

Contextual extracellular cues promote tumor cell EMT and metastasis by regulating miR-200 family expression

Bioluminescent human breast cancer cell lines that permit rapid and sensitive in vivo detection of mammary tumors and multiple metastases in immune deficient mice

Cancer invasion and the microenvironment: plasticity and reciprocity

Epithelial-mesenchymal transitions in tumour progression

Epithelial-mesenchymal transitions in development and disease

Gene expression patterns of breast carcinomas distinguish tumor subclasses with clinical implications

Gene expression profiling predicts clinical outcome of breast cancer

A gene-expression signature as a predictor of survival in breast cancer

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