MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism.
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The sweet trap in tumors: aerobic glycolysis and potential targets for therapyMUC1 facilitates metabolomic reprogramming in triple-negative breast cancer.Validation of Metabolic Alterations in Microscale Cell Culture Lysates Using Hydrophilic Interaction Liquid Chromatography (HILIC)-Tandem Mass Spectrometry-Based MetabolomicsSilibinin-mediated metabolic reprogramming attenuates pancreatic cancer-induced cachexia and tumor growthMUC16 contributes to the metastasis of pancreatic ductal adenocarcinoma through focal adhesion mediated signaling mechanism.C-terminus of MUC16 activates Wnt signaling pathway through its interaction with β-catenin to promote tumorigenesis and metastasis.Natural compounds targeting major cell signaling pathways: a novel paradigm for osteosarcoma therapy.MUC1-mediated metabolic alterations regulate response to radiotherapy in pancreatic cancer.Expression of Rab5a correlates with tumor progression in pancreatic carcinoma.Apigenin alleviates STZ-induced diabetic cardiomyopathy.DUSP28 links regulation of Mucin 5B and Mucin 16 to migration and survival of AsPC-1 human pancreatic cancer cells.A Panel of CA19-9, Ca125, and Ca15-3 as the Enhanced Test for the Differential Diagnosis of the Pancreatic Lesion.Mucins in pancreatic cancer: biological role, implications in carcinogenesis and applications in diagnosis and therapy.Selective analysis of cancer-cell intrinsic transcriptional traits defines novel clinically relevant subtypes of colorectal cancer.The Glycolytic Switch in Tumors: How Many Players Are Involved?Identification of unique neoantigen qualities in long-term survivors of pancreatic cancer.Rab11a sustains GSK3β/Wnt/β-catenin signaling to enhance cancer progression in pancreatic cancer.De Novo Lipid Synthesis Facilitates Gemcitabine Resistance through Endoplasmic Reticulum Stress in Pancreatic Cancer.Phosphoinositide 3-Kinase Signaling Pathway in Pancreatic Ductal Adenocarcinoma Progression, Pathogenesis, and Therapeutics.
P2860
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P2860
MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年学术文章
@wuu
2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
@zh
2015年學術文章
@zh-hant
name
MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism.
@ast
MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism.
@en
type
label
MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism.
@ast
MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism.
@en
prefLabel
MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism.
@ast
MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism.
@en
P2093
P2860
P356
P1433
P1476
MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism
@en
P2093
Aaron R Sasson
Anusha Mishra
Audrey J Lazenby
Dhanya Haridas
Jaime Abrego
Nina V Chaika
Pankaj K Singh
Surendra K Shukla
Surinder K Batra
P2860
P304
19118-19131
P356
10.18632/ONCOTARGET.4078
P407
P577
2015-08-01T00:00:00Z