2,3-Oxidosqualene cyclase protects liver cells from the injury of intermittent hypoxia by regulating lipid metabolism.

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Tauroursodeoxycholic acid attenuates endoplasmic reticulum stress and protects the liver from chronic intermittent hypoxia induced injury.Liver-heart crosstalk controls IL-22 activity in cardiac protection after myocardial infarction

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2,3-Oxidosqualene cyclase protects liver cells from the injury of intermittent hypoxia by regulating lipid metabolism.

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2015 nî lūn-bûn

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2,3-Oxidosqualene cyclase prot ...... y regulating lipid metabolism.

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2,3-Oxidosqualene cyclase prot ...... y regulating lipid metabolism.

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Ling Yu

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Qiu-Yan Song

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Rui-Hong Dong

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Xiao-Juan Rao

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Yan Wang

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Yan-Hong Sang

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Yu-Min Wu

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Yue-Qiao Zhen

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P2860

Suppression of 2,3-oxidosqualene cyclase by high fat diet contributes to liver X receptor-alpha-mediated improvement of hepatic lipid profile

Sleep disturbances, body fat distribution, food intake and/or energy expenditure: pathophysiological aspects

Potential role of nonstatin cholesterol lowering agents

Preferential cyclization of 2,3(S):22(S),23-dioxidosqualene by mammalian 2,3-oxidosqualene-lanosterol cyclase

The monotopic membrane protein human oxidosqualene cyclase is active as monomer

A novel inhibitor of oxidosqualene:lanosterol cyclase inhibits very low-density lipoprotein apolipoprotein B100 (apoB100) production and enhances low-density lipoprotein apoB100 catabolism through marked reduction in hepatic cholesterol content

The occurrence of sleep-disordered breathing among middle-aged adults

Prospective study of the association between sleep-disordered breathing and hypertension

Effect of intermittent hypoxia on atherosclerosis in apolipoprotein E-deficient mice.

Association of sleep-disordered breathing, sleep apnea, and hypertension in a large community-based study. Sleep Heart Health Study.

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2,3-Oxidosqualene cyclase protects liver cells from the injury of intermittent hypoxia by regulating lipid metabolism.

about

P2860

P2860

2,3-Oxidosqualene cyclase protects liver cells from the injury of intermittent hypoxia by regulating lipid metabolism.

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