Sapap3 deletion causes mGluR5-dependent silencing of AMPAR synapses
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Altered Striatal Synaptic Function and Abnormal Behaviour in Shank3 Exon4-9 Deletion Mouse Model of AutismSelective Disruption of Metabotropic Glutamate Receptor 5-Homer Interactions Mimics Phenotypes of Fragile X Syndrome in Mice.Optogenetic stimulation of lateral orbitofronto-striatal pathway suppresses compulsive behaviorsDysregulation of group-I metabotropic glutamate (mGlu) receptor mediated signalling in disorders associated with Intellectual Disability and AutismGenetic inhibition of CaMKII in dorsal striatal medium spiny neurons reduces functional excitatory synapses and enhances intrinsic excitability.Changes in sensitivity of reward and motor behavior to dopaminergic, glutamatergic, and cholinergic drugs in a mouse model of fragile X syndromeNeuroligin 1 modulates striatal glutamatergic neurotransmission in a pathway and NMDAR subunit-specific manner.Rare Synaptogenesis-Impairing Mutations in SLITRK5 Are Associated with Obsessive Compulsive Disorder.Learning From Animal Models of Obsessive-Compulsive DisorderMouse models of neurodevelopmental disease of the basal ganglia and associated circuits.Double deletion of melanocortin 4 receptors and SAPAP3 corrects compulsive behavior and obesity in miceAn integrated molecular landscape implicates the regulation of dendritic spine formation through insulin-related signalling in obsessive-compulsive disorder.Pharmacological, experimental therapeutic, and transcranial magnetic stimulation treatments for compulsivity and impulsivity.AMPA-silent synapses in brain development and pathology.Synaptic basis of social dysfunction: a focus on postsynaptic proteins linking group-I mGluRs with AMPARs and NMDARs.Gene expression profiling predicts pathways and genes associated with Parkinson's disease.Parvalbumin Interneurons of the Mouse Nucleus Accumbens are Required for Amphetamine-Induced Locomotor Sensitization and Conditioned Place Preference.A review of animal models of obsessive-compulsive disorder: a focus on developmental, immune, endocrine and behavioral models.Cross-generational THC exposure alters the developmental sensitivity of ventral and dorsal striatal gene expression in male and female offspring.Rodent models of obsessive compulsive disorder: Evaluating validity to interpret emerging neurobiology.The DLGAP family: neuronal expression, function and role in brain disorders.Increased Metabotropic Glutamate Receptor 5 Signaling Underlies Obsessive-Compulsive Disorder-like Behavioral and Striatal Circuit Abnormalities in MiceMouse model of rare TOR1A variant found in sporadic focal dystonia impairs domains affected in DYT1 dystonia patients and animal modelsCircuit-selective striatal synaptic dysfunction in the Sapap3 knockout mouse model of obsessive-compulsive disorder.And yet it moves: Recovery of volitional control after spinal cord injury.Glutamate Genetics in Obsessive-Compulsive Disorder: A Review.Novel Shank3 mutant exhibits behaviors with face validity for autism and altered striatal and hippocampal function.GRK5 Regulates Social Behavior Via Suppression of mTORC1 Signaling in Medial Prefrontal Cortex.Tweaking the social network.Defective Synapse Maturation and Enhanced Synaptic Plasticity in Shank2 Δex7–/– Mice.Glutamate delta-1 receptor regulates cocaine-induced plasticity in the nucleus accumbens
P2860
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P2860
Sapap3 deletion causes mGluR5-dependent silencing of AMPAR synapses
description
2011 nî lūn-bûn
@nan
2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
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2011年學術文章
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2011年學術文章
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name
Sapap3 deletion causes mGluR5-dependent silencing of AMPAR synapses
@ast
Sapap3 deletion causes mGluR5-dependent silencing of AMPAR synapses
@en
type
label
Sapap3 deletion causes mGluR5-dependent silencing of AMPAR synapses
@ast
Sapap3 deletion causes mGluR5-dependent silencing of AMPAR synapses
@en
prefLabel
Sapap3 deletion causes mGluR5-dependent silencing of AMPAR synapses
@ast
Sapap3 deletion causes mGluR5-dependent silencing of AMPAR synapses
@en
P2093
P2860
P1476
Sapap3 deletion causes mGluR5-dependent silencing of AMPAR synapses
@en
P2093
Guoping Feng
Nicole Calakos
Yehong Wan
P2860
P304
16685-16691
P356
10.1523/JNEUROSCI.2533-11.2011
P407
P577
2011-11-01T00:00:00Z