RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function.
about
Cre-mediated stress affects sirtuin expression levels, peroxisome biogenesis and metabolism, antioxidant and proinflammatory signaling pathwaysCNC-bZIP protein Nrf1-dependent regulation of glucose-stimulated insulin secretionpdx-1 function is specifically required in embryonic beta cells to generate appropriate numbers of endocrine cell types and maintain glucose homeostasisMembers of the miRNA-200 family regulate olfactory neurogenesisDistinct functions for IFT140 and IFT20 in opsin transportMetabolic and Behavioural Phenotypes in Nestin-Cre Mice Are Caused by Hypothalamic Expression of Human Growth HormonePancreatic β-cell Raf-1 is required for glucose tolerance, insulin secretion, and insulin 2 transcriptionAblation of AMP-activated protein kinase alpha1 and alpha2 from mouse pancreatic beta cells and RIP2.Cre neurons suppresses insulin release in vivo.Highly efficient cell-type-specific gene inactivation reveals a key function for the Drosophila FUS homolog cabeza in neuronsCharacterization of parameters required for effective use of tamoxifen-regulated recombination.Nestin-Cre mice are affected by hypopituitarism, which is not due to significant activity of the transgene in the pituitary gland.Gamma-protocadherins regulate the functional integrity of hypothalamic feeding circuitry in mice.Comparative analysis of the efficiency and specificity of myeloid-Cre deleting strains using ROSA-EYFP reporter mice.Sox9-haploinsufficiency causes glucose intolerance in miceTissue-specific insulin signaling in the regulation of metabolism and aging.rtTA toxicity limits the usefulness of the SP-C-rtTA transgenic mouseβ-Cells with relative low HIMP1 overexpression levels in a transgenic mouse line enhance basal insulin production and hypoxia/hypoglycemia tolerance.Coordinate lentiviral expression of Cre recombinase and RFP/EGFP mediated by FMDV 2A and analysis of Cre activity.The use of animal models in diabetes research.Conditional ablation of Gsk-3β in islet beta cells results in expanded mass and resistance to fat feeding-induced diabetes in miceDisruption of hepatic leptin signaling protects mice from age- and diet-related glucose intolerance.Insulin secretion stimulated by L-arginine and its metabolite L-ornithine depends on Gα(i2)Targeted inactivation of kinesin-1 in pancreatic β-cells in vivo leads to insulin secretory deficiency.MMTV-Cre transgenes can adversely affect lactation: considerations for conditional gene deletion in mammary tissue.Snapin mediates incretin action and augments glucose-dependent insulin secretionImpaired Ca(2+) signaling in β-cells lacking leptin receptors by Cre-loxP recombination.Ins1(Cre) knock-in mice for beta cell-specific gene recombination.Germ-line recombination activity of the widely used hGFAP-Cre and nestin-Cre transgenes.A practical guide to genetic engineering of pancreatic β-cells in vivo: getting a grip on RIP and MIP.Hemizygous Le-Cre transgenic mice have severe eye abnormalities on some genetic backgrounds in the absence of LoxP sitesNF-kappa B prevents beta cell death and autoimmune diabetes in NOD miceIncreased Specific Labeling of INS-1 Pancreatic Beta-Cell by Using RIP-Driven Cre Mutants with Reduced ActivityAcetyl-CoA carboxylase 2-/- mutant mice are protected against fatty liver under high-fat, high-carbohydrate dietary and de novo lipogenic conditions.Bladder function in mice with inducible smooth muscle-specific deletion of the manganese superoxide dismutase gene.Brain fatty acid synthase activates PPARalpha to maintain energy homeostasis.Evidence for baseline retinal pigment epithelium pathology in the Trp1-Cre mouse.Transcriptome of pancreas-specific Bmpr1a-deleted islets links to TPH1-5-HT axisβ-catenin is selectively required for the expansion and regeneration of mature pancreatic acinar cells in mice.Differential Insulin Secretion of High-Fat Diet-Fed C57BL/6NN and C57BL/6NJ Mice: Implications of Mixed Genetic Background in Metabolic Studies.Probing cell type-specific functions of Gi in vivo identifies GPCR regulators of insulin secretion.
P2860
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P2860
RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function.
@ast
RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function.
@en
type
label
RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function.
@ast
RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function.
@en
prefLabel
RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function.
@ast
RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function.
@en
P50
P356
P1476
RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function.
@en
P2093
Ji-Yeon Lee
Xueying Lin
P304
P356
10.1074/JBC.M512373200
P407
P577
2005-12-01T00:00:00Z