Programming of the endocrine pancreas by the early nutritional environment.
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Environmental Contaminants and Pancreatic Beta-CellsEffect of in utero and early-life conditions on adult health and diseaseEarly metabolic defects in dexamethasone-exposed and undernourished intrauterine growth restricted ratsIncreased adrenergic signaling is responsible for decreased glucose-stimulated insulin secretion in the chronically hyperinsulinemic ovine fetusEarly postnatal low-protein nutrition, metabolic programming and the autonomic nervous system in adult life.Diabetic and metabolic programming: mechanisms altering the intrauterine milieuWhat is an epigenetic transgenerational phenotype? F3 or F2.In Utero Nutritional Manipulation Provokes Dysregulated Adipocytokines Production in F1 Offspring in Rats.The placenta and intrauterine programming.The timing of nutrient restriction during rat pregnancy/lactation alters metabolic syndrome phenotype.Maternal obesity accelerates fetal pancreatic beta-cell but not alpha-cell development in sheep: prenatal consequences.Intrauterine diabetic milieu instigates dysregulated adipocytokines production in F1 offspring.Developmental origins of health and disease: experimental and human evidence of fetal programming for metabolic syndrome.The regulation of pre- and post-maturational plasticity of mammalian islet cell mass.Early-life nutritional programming of longevity.Food contaminants and programming of type 2 diabetes: recent findings from animal studies.The timing of "catch-up growth" affects metabolism and appetite regulation in male rats born with intrauterine growth restriction.Feeding mink (Neovison vison) a protein-restricted diet during pregnancy induces higher birth weight and altered hepatic gene expression in the F(2) offspring.Comparison of two models of intrauterine growth restriction for early catch-up growth and later development of glucose intolerance and obesity in rats.Impaired β-cell function in the adult offspring of rats fed a protein-restricted diet during lactation is associated with changes in muscarinic acetylcholine receptor subtypes.The effects of maternal health and body condition on the endocrine responses of neonatal foals.Pre- and postnatal methyl deficiency in the rat differentially alters glucose homeostasis.Decreased expression of GLUT4 in male CG-IUGR rats may play a vital role in their increased susceptibility to diabetes mellitus in adulthood.Metabolic imprinting by maternal protein malnourishment impairs vagal activity in adult rats.Foetal life protein restriction in male mink (Neovison vison) kits lowers post-weaning protein oxidation and the relative abundance of hepatic fructose-1,6-bisphosphatase mRNA.
P2860
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P2860
Programming of the endocrine pancreas by the early nutritional environment.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Programming of the endocrine pancreas by the early nutritional environment.
@ast
Programming of the endocrine pancreas by the early nutritional environment.
@en
type
label
Programming of the endocrine pancreas by the early nutritional environment.
@ast
Programming of the endocrine pancreas by the early nutritional environment.
@en
prefLabel
Programming of the endocrine pancreas by the early nutritional environment.
@ast
Programming of the endocrine pancreas by the early nutritional environment.
@en
P1476
Programming of the endocrine pancreas by the early nutritional environment.
@en
P2093
Brigitte Reusens
Claude Remacle
P304
P356
10.1016/J.BIOCEL.2005.10.012
P577
2005-11-10T00:00:00Z