Can ErbB2 overexpression protect against doxorubicin cardiotoxicity? - Wikidata - wikimedia - TriplyDB

Can ErbB2 overexpression protect against doxorubicin cardiotoxicity?

Can ErbB2 overexpression protect against doxorubicin cardiotoxicity?

http://www.wikidata.org/entity/Q36339397

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Letter to the editor: "Doxorubicin and ErbB2 overexpression: another piece in the mitochondrial jigsaw".

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Can ErbB2 overexpression protect against doxorubicin cardiotoxicity?

http://www.wikidata.org/entity/Q36339397

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2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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name

Can ErbB2 overexpression protect against doxorubicin cardiotoxicity?

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Can ErbB2 overexpression protect against doxorubicin cardiotoxicity?

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type

label

Can ErbB2 overexpression protect against doxorubicin cardiotoxicity?

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Can ErbB2 overexpression protect against doxorubicin cardiotoxicity?

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prefLabel

Can ErbB2 overexpression protect against doxorubicin cardiotoxicity?

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Can ErbB2 overexpression protect against doxorubicin cardiotoxicity?

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AJPHEART.00647.2015

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American Journal of Physiology - Heart and Circulatory Physiology

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Can ErbB2 overexpression protect against doxorubicin cardiotoxicity?

@en

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Petra Rocic

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Use of chemotherapy plus a monoclonal antibody against HER2 for metastatic breast cancer that overexpresses HER2.

Trastuzumab plus adjuvant chemotherapy for human epidermal growth factor receptor 2-positive breast cancer: planned joint analysis of overall survival from NSABP B-31 and NCCTG N9831.

Coronary microvascular rarefaction and myocardial fibrosis in heart failure with preserved ejection fraction.

ErbB2 overexpression upregulates antioxidant enzymes, reduces basal levels of reactive oxygen species, and protects against doxorubicin cardiotoxicity.

Echocardiographic Detection of Cardiac Dysfunction in Childhood Cancer Survivors: How Long Is Screening Required?

Cardiac toxicity of trastuzumab-related regimens in HER2-overexpressing breast cancer.

Patients' satisfaction in early breast cancer treatment: Change in treatment over time and impact of HER2-targeted therapy.

Anthracyclines/trastuzumab: new aspects of cardiotoxicity and molecular mechanisms.

Resolution of mitochondrial oxidative stress rescues coronary collateral growth in Zucker obese fatty rats.

Uncoupling protein downregulation in doxorubicin-induced heart failure improves mitochondrial coupling but increases reactive oxygen species generation.

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10.1152/AJPHEART.00647.2015

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