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Toll-like Receptor 4 Signaling Pathway in the Protective Effect of Pioglitazone on Experimental Immunoglobulin A Nephropathy.Development of immunoglobulin A nephropathy- like disease in beta-1,4-galactosyltransferase-I-deficient miceGlomerular immune deposits in experimental IgA nephropathy. A continuum of circulating and in situ formed immune complexes.Circulating and deposited immune complexes in patients with glomerular disease: immunopathologic correlationsExperimental cholestasis promotes the deposition of glomerular IgA immune complexesAn evaluation of experimental models of glomerulonephritisExperimental IgA nephropathy induced by oral immunizationClearance of circulating IgA immune complexes is mediated by a specific receptor on Kupffer cells in mice.Microbial IgA protease removes IgA immune complexes from mouse glomeruli in vivo: potential therapy for IgA nephropathy.Fcalpha receptor (CD89) mediates the development of immunoglobulin A (IgA) nephropathy (Berger's disease). Evidence for pathogenic soluble receptor-Iga complexes in patients and CD89 transgenic miceDysregulated LIGHT expression on T cells mediates intestinal inflammation and contributes to IgA nephropathyAcute lung injury in rat caused by immunoglobulin A immune complexes.Circulating immune complexes and immunoglobulin A rheumatoid factor in patients with mesangial immunoglobulin A nephropathies.Influence of antigen on immune complex behavior in mice.Selective deposition of immunoglobulin A1 in immunoglobulin A nephropathy, anaphylactoid purpura nephritis, and systemic lupus erythematosusChanges in size, subclass, and metabolic properties of serum immunoglobulin A in liver diseases and in other diseases with high serum immunoglobulin A.Increased intestinal permeability to (51 Cr) EDTA is correlated with IgA immune complex-plasma levels in children with IgA-associated nephropathies.IgA nephropathy--human disease and animal model.IgA nephropathy: characterization of the polymeric nature of mesangial deposits by in vitro binding of free secretory component.Deposition of IgA is associated with macrophage influx in the kidney of rats.Modulation of acute immune complex-mediated tissue injury by the presence of polyionic substances.[Formation of IgA deposits in Berger's disease: what we learned from animal models].Development of immune-complex glomerulonephritis in athymic mice: T cells are not required for the genesis of glomerular injury.Advanced IgA nephropathy with impaired renal function benefits from losartan treatment in rats.Kidney disease models: tools to identify mechanisms and potential therapeutic targets.Depletion of gut commensal bacteria attenuates intestinal ischemia/reperfusion injury.New insights into the pathogenesis of IgA nephropathy
P2860
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P2860
description
1979 nî lūn-bûn
@nan
1979年の論文
@ja
1979年学术文章
@wuu
1979年学术文章
@zh-cn
1979年学术文章
@zh-hans
1979年学术文章
@zh-my
1979年学术文章
@zh-sg
1979年學術文章
@yue
1979年學術文章
@zh
1979年學術文章
@zh-hant
name
Experimental IgA nephropathy
@ast
Experimental IgA nephropathy
@en
type
label
Experimental IgA nephropathy
@ast
Experimental IgA nephropathy
@en
prefLabel
Experimental IgA nephropathy
@ast
Experimental IgA nephropathy
@en
P2093
P2860
P356
P1476
Experimental IgA nephropathy
@en
P2093
P2860
P304
P356
10.1084/JEM.150.5.1161
P407
P577
1979-11-01T00:00:00Z