Human mitochondrial oxidative capacity is acutely impaired after burn trauma.
about
The impact of severe burns on skeletal muscle mitochondrial functionSerendipity and the discovery of novel compounds that restore mitochondrial plasticityTime-Dependent and Organ-Specific Changes in Mitochondrial Function, Mitochondrial DNA Integrity, Oxidative Stress and Mononuclear Cell Infiltration in a Mouse Model of Burn InjuryEffect of Human Burn Wound Exudate on Pseudomonas aeruginosa VirulenceClinical review: Glucose control in severely burned patients - current best practice.Method for controlled mitochondrial perturbation during phosphorus MRS in children.Uncoupled skeletal muscle mitochondria contribute to hypermetabolism in severely burned adults.Predictors of insulin resistance in pediatric burn injury survivors 24 to 36 months postburn.The Sick and the Weak: Neuropathies/Myopathies in the Critically Ill.Delayed skeletal muscle mitochondrial ADP recovery in youth with type 1 diabetes relates to muscle insulin resistance.Low-level laser therapy modulates musculoskeletal loss in a skin burn model in rats.Peripheral insulin resistance in obese girls with hyperandrogenism is related to oxidative phosphorylation and elevated serum free fatty acids.Novel mitochondria-targeted antioxidant peptide ameliorates burn-induced apoptosis and endoplasmic reticulum stress in the skeletal muscle of miceDifferential acute and chronic effects of burn trauma on murine skeletal muscle bioenergeticsAssociation of postburn fatty acids and triglycerides with clinical outcome in severely burned childrenWhole body and skeletal muscle protein turnover in recovery from burns.N-3 Polyunsaturated Fatty Acids Improve Liver Lipid Oxidation-Related Enzyme Levels and Increased the Peroxisome Proliferator-Activated Receptor α Expression Level in Mice Subjected to Hemorrhagic Shock/Resuscitation.The hepatic response to thermal injury: is the liver important for postburn outcomes?Intensive insulin therapy improves insulin sensitivity and mitochondrial function in severely burned children.Impaired glucose tolerance in pediatric burn patients at discharge from the acute hospital stay.The role of hyperglycemia in burned patients: evidence-based studies.Skeletal muscle mitochondria exhibit decreased pyruvate oxidation capacity and increased ROS emission during surgery-induced acute insulin resistance.Insulin resistance in type 2 diabetes youth relates to serum free fatty acids and muscle mitochondrial dysfunction.Taming the Flames: Targeting White Adipose Tissue Browning in Hypermetabolic Conditions.
P2860
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P2860
Human mitochondrial oxidative capacity is acutely impaired after burn trauma.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
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2008年學術文章
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2008年學術文章
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name
Human mitochondrial oxidative capacity is acutely impaired after burn trauma.
@ast
Human mitochondrial oxidative capacity is acutely impaired after burn trauma.
@en
type
label
Human mitochondrial oxidative capacity is acutely impaired after burn trauma.
@ast
Human mitochondrial oxidative capacity is acutely impaired after burn trauma.
@en
prefLabel
Human mitochondrial oxidative capacity is acutely impaired after burn trauma.
@ast
Human mitochondrial oxidative capacity is acutely impaired after burn trauma.
@en
P2093
P2860
P1476
Human mitochondrial oxidative capacity is acutely impaired after burn trauma.
@en
P2093
Asle Aarsland
Carlos Angel
David N Herndon
Justin M Green
Melanie G Cree
Ricki Y Fram
Robert R Wolfe
Ronald Mlcak
P2860
P304
P356
10.1016/J.AMJSURG.2007.09.048
P407
P577
2008-08-01T00:00:00Z