Studies on the mechanism of bacterial resistance to complement-mediated killing. I. Terminal complement components are deposited and released from Salmonella minnesota S218 without causing bacterial death.
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The neglected role of antibody in protection against bacteremia caused by nontyphoidal strains of Salmonella in African childrenIdentification of a domain in Rck, a product of the Salmonella typhimurium virulence plasmid, required for both serum resistance and cell invasionInfections of People with Complement Deficiencies and Patients Who Have Undergone SplenectomyThe ail gene of Yersinia enterocolitica has a role in the ability of the organism to survive serum killingBactericidal and bacteriolytic activity of serum against gram-negative bacteria.The dual role of lipopolysaccharide as effector and target molecule.Acquired resistance of Escherichia coli to complement lysis by binding of glycophosphoinositol-anchored protectin (CD59).Conflicting roles for a cell surface modification in SalmonellaThe polysaccharide portion of lipopolysaccharide regulates antigen-specific T-cell activation via effects on macrophage-mediated antigen processing.Functional recruitment of human complement inhibitor C4B-binding protein to outer membrane protein Rck of Salmonella.Mechanism of action of blocking immunoglobulin G for Neisseria gonorrhoeaeO-antigen structural variation: mechanisms and possible roles in animal/plant-microbe interactions.Complement activation and C3 binding by serum-sensitive and serum-resistant strains of Pseudomonas aeruginosa.Design of glycoconjugate vaccines against invasive African Salmonella enterica serovar TyphimuriumRelationship between antibody susceptibility and lipopolysaccharide O-antigen characteristics of invasive and gastrointestinal nontyphoidal Salmonellae isolates from Kenya.Burkholderia pseudomallei activates complement and is ingested but not killed by polymorphonuclear leukocytes.Pathogenesis of Campylobacter fetus infections. Failure of encapsulated Campylobacter fetus to bind C3b explains serum and phagocytosis resistance.Human natural anti-Gal IgG regulates alternative complement pathway activation on bacterial surfacesMechanism of resistance to complement-mediated killing of bacteria encoded by the Salmonella typhimurium virulence plasmid gene rck.Effects of the combination of lipopolysaccharide-specific monoclonal antibodies and sparfloxacin against Pseudomonas aeruginosa pneumonia in neutropenic miceBactericidal Immunity to Salmonella in Africans and Mechanisms Causing Its Failure in HIV Infection.The Salmonella typhimurium virulence plasmid complement resistance gene rck is homologous to a family of virulence-related outer membrane protein genes, including pagC and ailThe opsonizing ligand on Salmonella typhimurium influences incorporation of specific, but not azurophil, granule constituents into neutrophil phagosomesLipopolysaccharide size and distribution determine serum resistance in Salmonella montevideo.Characterization and biological role of the O-polysaccharide gene cluster of Yersinia enterocolitica serotype O:9Studies on the mechanism of bacterial resistance to complement-mediated killing. II. C8 and C9 release C5b67 from the surface of Salmonella minnesota S218 because the terminal complex does not insert into the bacterial outer membrane.Activation of complement by serum-resistant Neisseria gonorrhoeae. Assembly of the membrane attack complex without subsequent cell death.gp72, the 72 kilodalton glycoprotein, is the membrane acceptor site for C3 on Trypanosoma cruzi epimastigotes.Complement binding by two developmental stages of Leishmania major promastigotes varying in expression of a surface lipophosphoglycanThe PmrAB system-inducing conditions control both lipid A remodeling and O-antigen length distribution, influencing the Salmonella Typhimurium-host interactionsPhagocytosis of virulent Porphyromonas gingivalis by human polymorphonuclear leukocytes requires specific immunoglobulin G.Lipopolysaccharide phase variation determines the complement-mediated serum susceptibility of Coxiella burnetii.Role of immunoglobulin G in killing of Borrelia burgdorferi by the classical complement pathway.Characterization of the susceptibility of Pseudomonas aeruginosa to complement-mediated killing: role of antibodies to the rough lipopolysaccharide on serum-sensitive strains.Protective role of magnesium in the neutralization by antibodies of Chlamydia trachomatis infectivity.Role of antibodies in the opsonization of Yersinia spp.Plasmid-associated resistance of Salmonella typhimurium to complement activated by the classical pathwayHaemophilus influenzae type b lipooligosaccharide: stability of expression and association with virulenceInhibition of serum bactericidal reaction by lipopolysaccharide.Role of lipopolysaccharide and complement in susceptibility of Haemophilus ducreyi to human serum
P2860
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P2860
Studies on the mechanism of bacterial resistance to complement-mediated killing. I. Terminal complement components are deposited and released from Salmonella minnesota S218 without causing bacterial death.
description
1982 nî lūn-bûn
@nan
1982年の論文
@ja
1982年学术文章
@wuu
1982年学术文章
@zh-cn
1982年学术文章
@zh-hans
1982年学术文章
@zh-my
1982年学术文章
@zh-sg
1982年學術文章
@yue
1982年學術文章
@zh
1982年學術文章
@zh-hant
name
Studies on the mechanism of ba ...... thout causing bacterial death.
@ast
Studies on the mechanism of ba ...... thout causing bacterial death.
@en
type
label
Studies on the mechanism of ba ...... thout causing bacterial death.
@ast
Studies on the mechanism of ba ...... thout causing bacterial death.
@en
prefLabel
Studies on the mechanism of ba ...... thout causing bacterial death.
@ast
Studies on the mechanism of ba ...... thout causing bacterial death.
@en
P2093
P2860
P356
P1476
Studies on the mechanism of ba ...... thout causing bacterial death.
@en
P2093
P2860
P304
P356
10.1084/JEM.155.3.797
P407
P577
1982-03-01T00:00:00Z