Identification of human erythrocyte blood group antigens on decay-accelerating factor (DAF) and an erythrocyte phenotype negative for DAF
about
Pathogenesis and mechanisms of antibody-mediated hemolysisRole of decay-accelerating factor in regulating complement activation on the erythrocyte surface as revealed by gene targetingInfectious diseases associated with complement deficiencies.Rapid receptor-clustering assay to detect uropathogenic and diarrheal Escherichia coli isolates bearing adhesins of the Dr familyDr(a-) polymorphism of decay accelerating factor. Biochemical, functional, and molecular characterization and production of allele-specific transfectants.Acquired and transient RBC CD55 deficiency (Inab phenotype) and anti-IFC.The complement system in human reproduction.Rapid cyclic changes in density and accessibility of endometrial ligands for Escherichia coli Dr fimbriaeDecay-accelerating factor ameliorates systemic autoimmune disease in MRL/lpr mice via both complement-dependent and -independent mechanismsComplement-mediated haemolysis and the role of blood transfusion in paroxysmal nocturnal haemoglobinuria.Blood groups systems.Protection of host cells by complement regulators.Epithelial cells secrete interleukin-8 in response to adhesion and invasion of diffusely adhering Escherichia coli lacking Afa/Dr genes.Human regulator of complement activation (RCA) gene family proteins and their relationship to microbial infection.Blood group antigens defined by the amino acid sequences of red cell surface proteins.Mechanisms by which the surface expression of the glycosyl-phosphatidylinositol-anchored complement regulatory proteins decay-accelerating factor (CD55) and CD59 is lost in human leukaemia cell lines.Studies on the defect which causes absence of decay accelerating factor (DAF) from the peripheral blood cells of an individual with the Inab phenotype.Short consensus repeat-3 domain of recombinant decay-accelerating factor is recognized by Escherichia coli recombinant Dr adhesin in a model of a cell-cell interaction.SERF: a new antigen in the Cromer blood group system.Complement regulation.Three new high-prevalence antigens in the Cromer blood group system.Interleukin-8 secretion by epithelial cells infected with diffusely adherent Escherichia coli possessing Afa adhesin-coding genes.
P2860
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P2860
Identification of human erythrocyte blood group antigens on decay-accelerating factor (DAF) and an erythrocyte phenotype negative for DAF
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年学术文章
@wuu
1988年学术文章
@zh-cn
1988年学术文章
@zh-hans
1988年学术文章
@zh-my
1988年学术文章
@zh-sg
1988年學術文章
@yue
1988年學術文章
@zh
1988年學術文章
@zh-hant
name
Identification of human erythr ...... yte phenotype negative for DAF
@ast
Identification of human erythr ...... yte phenotype negative for DAF
@en
type
label
Identification of human erythr ...... yte phenotype negative for DAF
@ast
Identification of human erythr ...... yte phenotype negative for DAF
@en
prefLabel
Identification of human erythr ...... yte phenotype negative for DAF
@ast
Identification of human erythr ...... yte phenotype negative for DAF
@en
P2093
P2860
P356
P1476
Identification of human erythr ...... yte phenotype negative for DAF
@en
P2093
P2860
P304
P356
10.1084/JEM.167.6.1993
P407
P577
1988-06-01T00:00:00Z