Superantigen-reactive CD4+ T cells are required to stimulate B cells after infection with mouse mammary tumor virus.
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Mouse mammary tumor virus superantigen expression in B cells is regulated by a central enhancer within the pol geneEndogenous mouse mammary tumor viruses (mtv): new roles for an old virus in cancer, infection, and immunityVirus-encoded superantigensGenetics of mouse mammary tumor virus-induced mammary tumors: linkage of tumor induction to the gag gene.Systemic antibodies can inhibit mouse mammary tumor virus-driven superantigen response in mucosa-associated lymphoid tissues.Jaagsiekte retrovirus is widely distributed both in T and B lymphocytes and in mononuclear phagocytes of sheep with naturally and experimentally acquired pulmonary adenomatosis.Preferential binding of mouse mammary tumor virus to B lymphocytes.Interplays between mouse mammary tumor virus and the cellular and humoral immune response.Mtv-1 superantigen trafficks independently of major histocompatibility complex class II directly to the B-cell surface by the exocytic pathway.Immune response to mouse mammary tumor virus in mice lacking the alpha/beta interferon or the gamma interferon receptor.Superantigens: mechanisms by which they may induce, exacerbate and control autoimmune diseases.The mouse mammary tumor virus transcription enhancers for hematopoietic progenitor and mammary gland cells share functional elements.Expression of mouse mammary tumor virus superantigen mRNA in the thymus correlates with kinetics of self-reactive T-cell loss.Role of dendritic cells in the immune response induced by mouse mammary tumor virus superantigen.Mouse mammary tumor virus carrying a bacterial supF gene has wild-type pathogenicity and enables rapid isolation of proviral integration sitesIdentification of key amino acids of the mouse mammary tumor virus superantigen involved in the specific interaction with T-cell receptor V(beta) domains.Expression of mouse mammary tumor virus superantigen accelerates tumorigenicity of myeloma cells.Stimulation of mouse mammary tumor virus superantigen expression by an intragenic enhancerThe role of neutralizing antibodies for mouse mammary tumor virus transmission and mammary cancer development.Negative-acting factor and superantigen are separable activities of the mouse mammary tumor virus long terminal repeatTolerance to staphylococcal enterotoxin B initiated Th1 cell differentiation in mice infected with Candida albicansSequences within the gag gene of mouse mammary tumor virus needed for mammary gland cell transformation.Passive immunization with neutralizing antibodies interrupts the mouse mammary tumor virus life cycleRevenge of the microbes. Superantigens of the T and B cell lineageEvidence that the murine AIDS defective virus does not encode a superantigen.Major histocompatibility complex class II I-E-independent transmission of C3H mouse mammary tumor virus.The neonatal Fc receptor is not required for mucosal infection by mouse mammary tumor virus.Shared promoter elements between a viral superantigen and the major histocompatibility complex class II-associated invariant chain.Generation of a tumorigenic milk-borne mouse mammary tumor virus by recombination between endogenous and exogenous viruses.Both T and B cells shed infectious mouse mammary tumor virus.A highly sensitive in vitro infection assay to explore early stages of mouse mammary tumor virus infection.Retrovirus-induced target cell activation in the early phases of infection: the mouse mammary tumor virus modelPeripheral T cell activation and deletion induced by transfer of lymphocyte subsets expressing endogenous or exogenous mouse mammary tumor virus.B cells are essential for murine mammary tumor virus transmission, but not for presentation of endogenous superantigens.Neonatal deletion and selective expansion of mouse T cells by exposure to rabies virus nucleocapsid superantigen.Reverse transcriptase-dependent and -independent phases of infection with mouse mammary tumor virus: implications for superantigen functionCD40 ligation induces Apo-1/Fas expression on human B lymphocytes and facilitates apoptosis through the Apo-1/Fas pathway.Unique resistance of I/LnJ mice to a retrovirus is due to sustained interferon gamma-dependent production of virus-neutralizing antibodies.Viral superantigen drives extrafollicular and follicular B cell differentiation leading to virus-specific antibody production.The nasal-associated lymphoid tissue of adult mice acts as an entry site for the mouse mammary tumor retrovirus.
P2860
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P2860
Superantigen-reactive CD4+ T cells are required to stimulate B cells after infection with mouse mammary tumor virus.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Superantigen-reactive CD4+ T c ...... ith mouse mammary tumor virus.
@ast
Superantigen-reactive CD4+ T c ...... ith mouse mammary tumor virus.
@en
type
label
Superantigen-reactive CD4+ T c ...... ith mouse mammary tumor virus.
@ast
Superantigen-reactive CD4+ T c ...... ith mouse mammary tumor virus.
@en
prefLabel
Superantigen-reactive CD4+ T c ...... ith mouse mammary tumor virus.
@ast
Superantigen-reactive CD4+ T c ...... ith mouse mammary tumor virus.
@en
P2093
P2860
P356
P1476
Superantigen-reactive CD4+ T c ...... ith mouse mammary tumor virus.
@en
P2093
Acha-Orbea H
MacDonald HR
Scarpellino L
Shakhov AN
Waanders GA
P2860
P304
P356
10.1084/JEM.177.2.359
P407
P577
1993-02-01T00:00:00Z