Ly-49 mediates EL4 lymphoma adhesion to isolated class I major histocompatibility complex molecules.
about
An autosomal dominant locus, Nka, mapping to the Ly-49 region of a rat natural killer (NK) gene complex, controls NK cell lysis of allogeneic lymphocytesNatural killer cell receptors specific for major histocompatibility complex class I molecules.Regulatory role of T cells producing both interferon gamma and interleukin 10 in persistent infectionAltered phenotype and function of natural killer cells expressing the major histocompatibility complex receptor Ly-49 in mice transgenic for its ligandMajor histocompatibility complex class I antigens and the control of viral infections by natural killer cells.A natural killer cell receptor specific for a major histocompatibility complex class I molecule.Expression of different members of the Ly-49 gene family defines distinct natural killer cell subsets and cell adhesion properties.The Bw4 public epitope of HLA-B molecules confers reactivity with natural killer cell clones that express NKB1, a putative HLA receptor.NKR-P1A is a target-specific receptor that activates natural killer cell cytotoxicityLy-49-independent natural killer (NK) cell specificity revealed by NK cell clones derived from p53-deficient mice.Hybrid resistance and the Ly-49 family of natural killer cell receptors.Cloning and functional characteristics of murine large granular lymphocyte-1: a member of the Ly-49 gene family (Ly-49G2)Heterogeneous phenotypes of expression of the NKB1 natural killer cell class I receptor among individuals of different human histocompatibility leukocyte antigens types appear genetically regulated, but not linked to major histocompatibililty compleRecognition of class I major histocompatibility complex molecules by Ly-49: specificities and domain interactionsTransgenic expression of the Ly49A natural killer cell receptor confers class I major histocompatibility complex (MHC)-specific inhibition and prevents bone marrow allograft rejection.The murine nonclassical class I major histocompatibility complex-like CD1.1 molecule protects target cells from lymphokine-activated killer cell cytolysis.Mouse Ly-49D recognizes H-2Dd and activates natural killer cell cytotoxicityH-2D ligand expression by Ly49A+ natural killer (NK) cells precludes ligand uptake from environmental cells: implications for NK cell function.Inhibitory receptors alter natural killer cell interactions with target cells yet allow simultaneous killing of susceptible targets.Mouse Ly-49A interrupts early signaling events in natural killer cell cytotoxicity and functionally associates with the SHP-1 tyrosine phosphatase.Ly49A transgenic mice provide evidence for a major histocompatibility complex-dependent education process in natural killer cell development.The Ly-49D receptor activates murine natural killer cells.Contrasting effects of anti-Ly49A due to MHC class I cis binding on NK cell-mediated allogeneic bone marrow cell resistance.Direct binding of a soluble natural killer cell inhibitory receptor to a soluble human leukocyte antigen-Cw4 class I major histocompatibility complex moleculeH-2 allele specificity of the NK cell C-type lectin-like MHC class I receptor Ly49A visualized by soluble Ly49A tetramer.The activating Ly49W and inhibitory Ly49G NK cell receptors display similar affinities for identical MHC class I ligands.Lymphocyte-mediated cytotoxicity in vitro and in vivo: mechanisms and significance.Cis association of Ly49A with MHC class I restricts natural killer cell inhibition.Recognition of major histocompatibility complex class I antigens by natural killer cells.Impaired MHC class I (H-2Dd)-mediated protection against Ly-49A+ NK cells after amino acid substitutions in the antigen binding cleft.Inhibition of natural killer cell cytotoxicity by cell growth-related molecules.Host MHC class I gene control of NK-cell specificity in the mouse.Allorecognition by murine natural killer cells: lysis of T-lymphoblasts and rejection of bone-marrow grafts.The Ly-49 family: genes, proteins and recognition of class I MHC.Divergent functions of lectin-like receptors on NK cells.Arousal and inhibition of human NK cells.Natural killer cell education in mice with single or multiple major histocompatibility complex class I molecules.Glu227-->Lys substitution in the acidic loop of major histocompatibility complex class I alpha 3 domain distinguishes low avidity CD8 coreceptor and avidity-enhanced CD8 accessory functions.Differential ability of isolated H-2 Kb subsets to serve as TCR ligands for allo-specific CTL clones: potential role for N-linked glycosylation.A new mechanism of NK cell cytotoxicity activation: the CD40-CD40 ligand interaction.
P2860
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P2860
Ly-49 mediates EL4 lymphoma adhesion to isolated class I major histocompatibility complex molecules.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
1994年學術文章
@zh
1994年學術文章
@zh-hant
name
Ly-49 mediates EL4 lymphoma ad ...... mpatibility complex molecules.
@ast
Ly-49 mediates EL4 lymphoma ad ...... mpatibility complex molecules.
@en
type
label
Ly-49 mediates EL4 lymphoma ad ...... mpatibility complex molecules.
@ast
Ly-49 mediates EL4 lymphoma ad ...... mpatibility complex molecules.
@en
prefLabel
Ly-49 mediates EL4 lymphoma ad ...... mpatibility complex molecules.
@ast
Ly-49 mediates EL4 lymphoma ad ...... mpatibility complex molecules.
@en
P2860
P356
P1476
Ly-49 mediates EL4 lymphoma ad ...... mpatibility complex molecules.
@en
P2093
P2860
P304
P356
10.1084/JEM.179.3.1011
P407
P577
1994-03-01T00:00:00Z