Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

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Central Mechanisms Mediating Thrombospondin-4-induced Pain States.Painful Cervical Facet Joint Injury Is Accompanied by Changes in the Number of Excitatory and Inhibitory Synapses in the Superficial Dorsal Horn That Differentially Relate to Local Tissue Injury Severity.

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Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

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2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

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Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

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label

Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

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Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

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Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

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Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

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P1476

Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

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P2093

Ilse Sears-Kraxberger

http://www.w3.org/2001/XMLSchema#string

John Park

http://www.w3.org/2001/XMLSchema#string

Kang-Wu Li

http://www.w3.org/2001/XMLSchema#string

Oswald Steward

http://www.w3.org/2001/XMLSchema#string

Van Nancy Trinh

http://www.w3.org/2001/XMLSchema#string

Z David Luo

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P2860

Gabapentin receptor alpha2delta-1 is a neuronal thrombospondin receptor responsible for excitatory CNS synaptogenesis

Neuronal circuitry for pain processing in the dorsal horn

Organization of intralaminar and translaminar neuronal connectivity in the superficial spinal dorsal horn

Molecular mechanisms of nociception

GABA decreases in the spinal cord dorsal horn after peripheral neurectomy

Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states

Quantitative assessment of tactile allodynia in the rat paw

Transmission efficacy and plasticity in glutamatergic synapses formed by excitatory interneurons of the substantia gelatinosa in the rat spinal cord.

Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

Estimation of the number of synapses in the cerebral cortex: methodological considerations.

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309-322

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scholarly article

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10.1002/CNE.23844

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English

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524

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Van Nancy Trinh

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2015-07-01T00:00:00Z

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26132987

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4675675

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Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

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P2860

P2860

Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.

description

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type

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P1433

P1476

P2093

P2860

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