Erythrocyte stages of Plasmodium falciparum exhibit a high nitric oxide synthase (NOS) activity and release an NOS-inducing soluble factor
about
Role of oxidants in microbial pathophysiologyNitric oxide synthase is induced in sporulation of Physarum polycephalumChickens treated with a nitric oxide inhibitor became more resistant to Plasmodium gallinaceum infection due to reduced anemia, thrombocytopenia and inflammation.Blood coagulation, inflammation, and malaria.Extreme mutation bias and high AT content in Plasmodium falciparum.Plasmodium falciparum-infected erythrocytes induce NF-kappaB regulated inflammatory pathways in human cerebral endotheliumRegulation of extracellular ATP in human erythrocytes infected with Plasmodium falciparumTumor necrosis factor-alpha expression in the brain during fatal murine cerebral malaria: evidence for production by microglia and astrocytes.Correlation between enhanced vascular permeability, up-regulation of cellular adhesion molecules and monocyte adhesion to the endothelium in the retina during the development of fatal murine cerebral malaria.Cell-mediated effector molecules and complicated malaria.Nitric oxide in Tanzanian children with malaria: inverse relationship between malaria severity and nitric oxide production/nitric oxide synthase type 2 expression.Circulating NO pool in humans.Plasmodium falciparum: food vacuole localization of nitric oxide-derived species in intraerythrocytic stages of the malaria parasiteDisruption of a Plasmodium falciparum cyclic nucleotide phosphodiesterase gene causes aberrant gametogenesisSoluble factors from Plasmodium falciparum-infected erythrocytes induce apoptosis in human brain vascular endothelial and neuroglia cellsInducible nitric oxide synthase expression is increased in the brain in fatal cerebral malaria.Chloroquine stimulates nitric oxide synthesis in murine, porcine, and human endothelial cells.Sequential Serum Cytokine Levels of TNF-Alpha, IL-4 and IL-12 are Associated with Prognosis in Plasmodium falciparum Malaria.Sequestration and metabolism of host cell arginine by the intraerythrocytic malaria parasite Plasmodium falciparum.Regulation of host cell function by glycosylphosphatidylinositols of the parasitic protozoa.Compromised blood-nerve barrier, astrogliosis, and myelin disruption in optic nerves during fatal murine cerebral malaria.Malaria parasite-specific Th1-like T cells simultaneously reduce parasitemia and promote disease.Nitric oxide and reactive nitrogen intermediates during lethal and nonlethal strains of murine malaria.Nitric oxide induction as a novel immunoepidemiological target in malaria-infected patients from endemic areas of the Islamic Republic of Iran.Early activation of microglia in the pathogenesis of fatal murine cerebral malaria.Neutrophil extracellular traps contain mitochondrial as well as nuclear DNA and exhibit inflammatory potential.
P2860
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P2860
Erythrocyte stages of Plasmodium falciparum exhibit a high nitric oxide synthase (NOS) activity and release an NOS-inducing soluble factor
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh
1995年學術文章
@zh-hant
name
Erythrocyte stages of Plasmodi ...... an NOS-inducing soluble factor
@ast
Erythrocyte stages of Plasmodi ...... an NOS-inducing soluble factor
@en
type
label
Erythrocyte stages of Plasmodi ...... an NOS-inducing soluble factor
@ast
Erythrocyte stages of Plasmodi ...... an NOS-inducing soluble factor
@en
prefLabel
Erythrocyte stages of Plasmodi ...... an NOS-inducing soluble factor
@ast
Erythrocyte stages of Plasmodi ...... an NOS-inducing soluble factor
@en
P2093
P2860
P356
P1476
Erythrocyte stages of Plasmodi ...... an NOS-inducing soluble factor
@en
P2093
Bussolino F
Costamagna C
Gabrielli G
Ginsburg H
Giribaldi G
Pescarmona G
P2860
P304
P356
10.1084/JEM.182.3.677
P407
P577
1995-09-01T00:00:00Z