Selective development of T helper (Th)2 cells induced by continuous administration of low dose soluble proteins to normal and beta(2)-microglobulin-deficient BALB/c mice
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Alacrity of cells engaged in the immune responseDevelopment of allergic airway disease in mice following antibiotic therapy and fungal microbiota increase: role of host genetics, antigen, and interleukin-13Leishmania major infection of inbred mice: unmasking genetic determinants of infectious diseases.Diminished monocyte function in microfilaremic patients with lymphatic filariasis and its relationship to altered lymphoproliferative responsesAltering the cellular location of an antigen expressed by a DNA-based vaccine modulates the immune responseCoadministration of interleukin 12 expression vector with antigen 2 cDNA enhances induction of protective immunity against Coccidioides immitisInterleukin-4 receptor alpha-deficient BALB/c mice show an unimpaired T helper 2 polarization in response to Leishmania major infectionAntigen depot is not required for alum adjuvanticity.Lethal host-versus-graft disease and hypereosinophilia in the absence of MHC I-T-cell interactions.Requirement for natural killer T (NKT) cells in the induction of allograft toleranceOcular allergy modulation to hi-dose antigen sensitization is a Treg-dependent process.Mouse V alpha 14i natural killer T cells are resistant to cytokine polarization in vivoAnti-interleukin-4 modulation of the Th2 polarized response to the parasitic nematode Brugia pahangi.Distinct roles of VEGFR-1 and VEGFR-2 in the aberrant hematopoiesis associated with elevated levels of VEGF.Dendritic cells but not B cells present antigenic complexes to class II-restricted T cells after administration of protein in adjuvant.Beta 2-microglobulin-dependent T cells are dispensable for allergen-induced T helper 2 responses.Beta 2-microglobulin-dependent NK1.1+ T cells are not essential for T helper cell 2 immune responsesInterleukin 4-producing CD4 T cells arise from different precursors depending on the conditions of antigen exposure in vivo.Low dose Leishmania major promotes a transient T helper cell type 2 response that is down-regulated by interferon gamma-producing CD8+ T cellsGenetic regulation of commitment to interleukin 4 production by a CD4(+) T cell-intrinsic mechanismInterferon gamma enhances both in vitro and in vivo priming of CD4+ T cells for IL-4 production.Antigen kinetics determines immune reactivity.An interleukin 4 (IL-4)-independent pathway for CD4+ T cell IL-4 production is revealed in IL-4 receptor-deficient mice.New routes for allergen immunotherapy.Real-time imaging of the cellular interactions underlying tolerance, priming, and responses to infection.The role of hepatic invariant NKT cells in systemic/local inflammation and mortality during polymicrobial septic shockComplement receptor 3 deficiency influences lesion progression during Leishmania major infection in BALB/c miceEnterotoxin-based mucosal adjuvants alter antigen trafficking and induce inflammatory responses in the nasal tract.Surface-modified P(HEMA-co-MAA) nanogel carriers for oral vaccine delivery: design, characterization, and in vitro targeting evaluation.Transcriptional repressor DREAM regulates T-lymphocyte proliferation and cytokine gene expression.Activation of Valpha14(+) natural killer T cells by alpha-galactosylceramide results in development of Th1 response and local host resistance in mice infected with Cryptococcus neoformans.CD4(+) Valpha14 natural killer T cells are essential for acceptance of rat islet xenografts in miceTh1 cells induce and Th2 inhibit antigen-dependent IL-12 secretion by dendritic cells.Manipulation of the Th1/Th2 cell balance: an approach to treat human autoimmune diseases?Inhibition of T helper cell type 2 cell differentiation and immunoglobulin E response by ligand-activated Valpha14 natural killer T cells.Nanoparticles prepared from porcine cells support the healing of cutaneous inflammation in mice and wound re-epithelialization in human skin.Negative feedback control of the autoimmune response through antigen-induced differentiation of IL-10-secreting Th1 cells.Antigen-based therapy for the treatment of type 1 diabetes.Th2 polarization enhanced by oral administration of higher doses of antigen.HgCl2-induced interleukin-4 gene expression in T cells involves a protein kinase C-dependent calcium influx through L-type calcium channels.
P2860
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P2860
Selective development of T helper (Th)2 cells induced by continuous administration of low dose soluble proteins to normal and beta(2)-microglobulin-deficient BALB/c mice
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Selective development of T hel ...... globulin-deficient BALB/c mice
@ast
Selective development of T hel ...... globulin-deficient BALB/c mice
@en
type
label
Selective development of T hel ...... globulin-deficient BALB/c mice
@ast
Selective development of T hel ...... globulin-deficient BALB/c mice
@en
prefLabel
Selective development of T hel ...... globulin-deficient BALB/c mice
@ast
Selective development of T hel ...... globulin-deficient BALB/c mice
@en
P2093
P2860
P356
P1476
Selective development of T hel ...... globulin-deficient BALB/c mice
@en
P2093
P2860
P304
P356
10.1084/JEM.183.2.485
P407
P577
1996-02-01T00:00:00Z