Late stage erythroid precursor production is impaired in mice with chronic inflammation - Wikidata - wikimedia - TriplyDB

Late stage erythroid precursor production is impaired in mice with chronic inflammation

Late stage erythroid precursor production is impaired in mice with chronic inflammation

http://www.wikidata.org/entity/Q36366570

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HRG1 is essential for heme transport from the phagolysosome of macrophages during erythrophagocytosis Distinct roles for hepcidin and interleukin-6 in the recovery from anemia in mice injected with heat-killed Brucella abortus Isocitrate ameliorates anemia by suppressing the erythroid iron restriction response.Hepcidin-dependent and hepcidin-independent regulation of erythropoiesis in a mouse model of anemia of chronic inflammation.RAP-011, an activin receptor ligand trap, increases hemoglobin concentration in hepcidin transgenic mice Intravenous Iron Carboxymaltose as a Potential Therapeutic in Anemia of Inflammation.Isocitrate treatment of acute anemia of inflammation in a mouse model.Effects of Testosterone on Erythropoiesis in a Female Mouse Model of Anemia of Inflammation Out of balance--systemic iron homeostasis in iron-related disorders Investigation of the role of interleukin-6 and hepcidin antimicrobial peptide in the development of anemia with age.Sequestration and scavenging of iron in infection.Interleukin-6 directly impairs the erythroid development of human TF-1 erythroleukemic cells The type I BMP receptor Alk3 is required for the induction of hepatic hepcidin gene expression by interleukin-6.Manipulation of the hepcidin pathway for therapeutic purposes.The pathophysiology and pharmacology of hepcidin.Oral administration of a bone morphogenetic protein type I receptor inhibitor prevents the development of anemia of inflammation.Pathogenesis and Treatment Options of Cancer Related Anemia: Perspective for a Targeted Mechanism-Based Approach Modulation of Iron Metabolism in Response to Infection: Twists for All Tastes

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P2860

Late stage erythroid precursor production is impaired in mice with chronic inflammation

http://www.wikidata.org/entity/Q36366570

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

@zh-hant

name

Late stage erythroid precursor production is impaired in mice with chronic inflammation

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Late stage erythroid precursor production is impaired in mice with chronic inflammation

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type

label

Late stage erythroid precursor production is impaired in mice with chronic inflammation

@ast

Late stage erythroid precursor production is impaired in mice with chronic inflammation

@en

prefLabel

Late stage erythroid precursor production is impaired in mice with chronic inflammation

@ast

Late stage erythroid precursor production is impaired in mice with chronic inflammation

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HAEMATOL.2011.053397

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Late stage erythroid precursor production is impaired in mice with chronic inflammation

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Alan E Berger

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Andrew J Layman

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Chris Cheadle

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Cindy N Roy

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Curt I Civin

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Francis J Chrest

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Howard H Mak

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Ian C Prince

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Jacqueline M Langdon

http://www.w3.org/2001/XMLSchema#string

Jeremy D Walston

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P2860

Hepcidin regulates cellular iron efflux by binding to ferroportin and inducing its internalization

Mutations in TMPRSS6 cause iron-refractory iron deficiency anemia (IRIDA)

IL-6 mediates hypoferremia of inflammation by inducing the synthesis of the iron regulatory hormone hepcidin

Hepcidin mediates transcriptional changes that modulate acute cytokine-induced inflammatory responses in mice

Hepcidin as a therapeutic tool to limit iron overload and improve anemia in β-thalassemic mice

Inappropriate expression of hepcidin is associated with iron refractory anemia: implications for the anemia of chronic disease

Hepcidin, a putative mediator of anemia of inflammation, is a type II acute-phase protein

The serine protease TMPRSS6 is required to sense iron deficiency

Acute-phase proteins and other systemic responses to inflammation

Loss of alpha-hemoglobin-stabilizing protein impairs erythropoiesis and exacerbates beta-thalassemia

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1648-1656

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