Cholera toxin suppresses interleukin (IL)-12 production and IL-12 receptor beta1 and beta2 chain expression.
about
Vibrio cholerae evades neutrophil extracellular traps by the activity of two extracellular nucleasesHistamine regulates cytokine production in maturing dendritic cells, resulting in altered T cell polarizationActivation of c-Kit in dendritic cells regulates T helper cell differentiation and allergic asthmaIntranasal delivery of cholera toxin induces th17-dominated T-cell response to bystander antigens.Immunomodulatory effects of Yersinia pestis lipopolysaccharides on human macrophages.IL-12 as a therapeutic target for pharmacological modulation in immune-mediated and inflammatory diseases: regulation of T helper 1/T helper 2 responsesRegulation of the IL-12/IL-12R axis: a critical step in T-helper cell differentiation and effector function.New paradigms in type 2 immunity.Mucosal adjuvant activity of cholera toxin requires Th17 cells and protects against inhalation anthrax.Indigenous enteric eosinophils control DCs to initiate a primary Th2 immune response in vivoMutants of Escherichia coli heat-labile toxin act as effective mucosal adjuvants for nasal delivery of an acellular pertussis vaccine: differential effects of the nontoxic AB complex and enzyme activity on Th1 and Th2 cells.Tetanus toxin fragment C expressed in live Salmonella vaccines enhances antibody responses to its fusion partner Schistosoma haematobium glutathione S-transferase.Immunomodulation using bacterial enterotoxins.Intrarectal immunization with rotavirus 2/6 virus-like particles induces an antirotavirus immune response localized in the intestinal mucosa and protects against rotavirus infection in mice.Strategies for designing and optimizing new generation vaccines.Colonization of C57BL/6J and BALB/c wild-type and knockout mice with Helicobacter pylori: effect of vaccination and implications for innate and acquired immunityMutant Escherichia coli heat-labile toxin B subunit that separates toxoid-mediated signaling and immunomodulatory action from trafficking and delivery functionsCholera toxin impairs the differentiation of monocytes into dendritic cells, inducing professional antigen-presenting myeloid cells.Nasal delivery of antigen with the B subunit of Escherichia coli heat-labile enterotoxin augments antigen-specific T-cell clonal expansion and differentiation.Plasmid vectors encoding cholera toxin or the heat-labile enterotoxin from Escherichia coli are strong adjuvants for DNA vaccines.Effects of cholera toxin on innate and adaptive immunity and its application as an immunomodulatory agent.Endotoxic shock-expanded murine CD11c low CD45RB + regulatory dendritic cells modulate inflammatory T cell responses through multiple mechanismsInhibiting cytokines of the interleukin-12 family: recent advances and novel challenges.Modulating vaccine responses with dendritic cells and Toll-like receptors.Frontal and stealth attack strategies in microbial pathogenesis.Immunomodulation with enterotoxins for the generation of secretory immunity or tolerance: applications for oral infections.CD47 ligation selectively downregulates human interleukin 12 production.Signaling of c-kit in dendritic cells influences adaptive immunity.Cocoa Diet Prevents Antibody Synthesis and Modifies Lymph Node Composition and Functionality in a Rat Oral Sensitization ModelNod2-mediated recognition of the microbiota is critical for mucosal adjuvant activity of cholera toxinPhosphodiesterase 4 inhibition in the treatment of psoriasis, psoriatic arthritis and other chronic inflammatory diseases.Cholera toxin B subunit acts as a potent systemic adjuvant for HIV-1 DNA vaccination intramuscularly in mice.Cyclic AMP: master regulator of innate immune cell function.Transcutaneous immunization induces mucosal CTLs and protective immunity by migration of primed skin dendritic cellsCholera toxin inhibits IL-12 production and CD8alpha+ dendritic cell differentiation by cAMP-mediated inhibition of IRF8 function.Differential requirements for protection against mucosal challenge with Francisella tularensis in the presence versus absence of cholera toxin B and inactivated F. tularensisCholera toxin directly enhances IL-17A production from human CD4+ T cells.The B subunit of Escherichia coli heat-labile enterotoxin induces both caspase-dependent and -independent cell death pathways in CD8+ T cellsMechanisms of Cholera Toxin in the Modulation of TH17 Responses.Interleukin-12 as an adjuvant for induction of protective antibody responses.
P2860
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P2860
Cholera toxin suppresses interleukin (IL)-12 production and IL-12 receptor beta1 and beta2 chain expression.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Cholera toxin suppresses inter ...... a1 and beta2 chain expression.
@ast
Cholera toxin suppresses inter ...... a1 and beta2 chain expression.
@en
type
label
Cholera toxin suppresses inter ...... a1 and beta2 chain expression.
@ast
Cholera toxin suppresses inter ...... a1 and beta2 chain expression.
@en
prefLabel
Cholera toxin suppresses inter ...... a1 and beta2 chain expression.
@ast
Cholera toxin suppresses inter ...... a1 and beta2 chain expression.
@en
P2093
P2860
P356
P1476
Cholera toxin suppresses inter ...... a1 and beta2 chain expression.
@en
P2093
P2860
P304
P356
10.1084/JEM.189.3.541
P407
P577
1999-02-01T00:00:00Z