Bone marrow NK1.1(-) and NK1.1(+) T cells reciprocally regulate acute graft versus host disease. - Wikidata - wikimedia - TriplyDB

Bone marrow NK1.1(-) and NK1.1(+) T cells reciprocally regulate acute graft versus host disease.

Bone marrow NK1.1(-) and NK1.1(+) T cells reciprocally regulate acute graft versus host disease.

http://www.wikidata.org/entity/Q36368026

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Regulatory T-cells: diverse phenotypes integral to immune homeostasis and suppression Immune privilege: keeping an eye on natural killer T cells.Non-classical natural killer T cells modulate plasmid DNA vaccine antigen expression and vaccine-elicited immune responses by MCP-1 secretion after interaction with a beta2-microglobulin-independent CD1d.Natural Killer Cells in Graft-versus-Host-Disease after Allogeneic Hematopoietic Cell Transplantation.Current and future approaches for control of graft-versus-host disease.Regulatory T Cell and Forkhead Box Protein 3 as Modulators of Immune Homeostasis Path to clinical transplantation tolerance and prevention of graft-versus-host disease.Administration of anti-CD20 mAb is highly effective in preventing but ineffective in treating chronic graft-versus-host disease while preserving strong graft-versus-leukemia effects Natural killer T cells reactive to a single glycolipid exhibit a highly diverse T cell receptor beta repertoire and small clone size.Immunobiology of allogeneic peripheral blood mononuclear cells mobilized with granulocyte-colony stimulating factor.The role of interleukin-12 and interferon-gamma in GVHD and GVL.NKT cell-dependent leukemia eradication following stem cell mobilization with potent G-CSF analogs.Nuclear factor-kappaB inducing kinase is required for graft-versus-host disease.Influenza infection in suckling mice expands an NKT cell subset that protects against airway hyperreactivity.Lethal host-versus-graft disease and hypereosinophilia in the absence of MHC I-T-cell interactions.Mechanisms of action of antithymocyte globulin: T-cell depletion and beyond.B7H1/CD80 interaction augments PD-1-dependent T cell apoptosis and ameliorates graft-versus-host disease HDAC inhibition and graft versus host disease Invariant NKT cells are required for antitumor responses induced by host-versus-graft responses Regulatory T cells in the control of transplantation tolerance and autoimmunity.Innate self recognition by an invariant, rearranged T-cell receptor and its immune consequences.Mouse V alpha 14i natural killer T cells are resistant to cytokine polarization in vivo Bone marrow and the control of immunity.Physical Exercise Training versus Relaxation in Allogeneic stem cell transplantation (PETRA Study) - Rationale and design of a randomized trial to evaluate a yearlong exercise intervention on overall survival and side-effects after allogeneic stem c Interactions between NKT cells and Tregs are required for tolerance to combined bone marrow and organ transplants.Critical role of host gammadelta T cells in experimental acute graft-versus-host disease.Low doses of natural killer T cells provide protection from acute graft-versus-host disease via an IL-4-dependent mechanism Bystander activation of iNKT cells occurs during conventional T-cell alloresponses Going both ways: immune regulation via CD1d-dependent NKT cells.Pathophysiology of acute graft-versus-host disease: recent advances.A privileged view of NKT cells and peripheral tolerance through the eye.Activation of natural killer T cells in NZB/W mice induces Th1-type immune responses exacerbating lupus Housing Temperature-Induced Stress Is Suppressing Murine Graft-versus-Host Disease through β2-Adrenergic Receptor Signaling.Clonable progenitors committed to the T lymphocyte lineage in the mouse bone marrow; use of an extrathymic pathway.Tracking the response of natural killer T cells to a glycolipid antigen using CD1d tetramers A natural killer T (NKT) cell developmental pathway iInvolving a thymus-dependent NK1.1(-)CD4(+) CD1d-dependent precursor stage.Donor-type CD4(+)CD25(+) regulatory T cells suppress lethal acute graft-versus-host disease after allogeneic bone marrow transplantation.A critical role for natural killer T cells in immunosurveillance of methylcholanthrene-induced sarcomas.CD1-reactive natural killer T cells are required for development of systemic tolerance through an immune-privileged site.Regulatory T cells in lupus.

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Bone marrow NK1.1(-) and NK1.1(+) T cells reciprocally regulate acute graft versus host disease.

http://www.wikidata.org/entity/Q36368026

description

1999 nî lūn-bûn

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1999年学术文章

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Bone marrow NK1.1(-) and NK1.1 ...... ute graft versus host disease.

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Bone marrow NK1.1(-) and NK1.1 ...... ute graft versus host disease.

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Bone marrow NK1.1(-) and NK1.1 ...... ute graft versus host disease.

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Bone marrow NK1.1(-) and NK1.1 ...... ute graft versus host disease.

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Bone marrow NK1.1(-) and NK1.1 ...... ute graft versus host disease.

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Bone marrow NK1.1(-) and NK1.1 ...... ute graft versus host disease.

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Bone marrow NK1.1(-) and NK1.1 ...... ute graft versus host disease.

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D Lewis

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D Zeng

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F Lan

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P2860

Unusual T cell populations in adult murine bone marrow. Prevalence of CD3+CD4-CD8- and alpha beta TCR+NK1.1+ cells.

In vitro and in vivo analysis of bone marrow-derived CD3+, CD4-, CD8-, NK1.1+ cell lines.

Human bone marrow lymphocytes. I. Distribution of lymphocyte subpopulations in the bone marrow of normal individuals

Thymus-dependent lymphocytes in human bone marrow

Lethal graft-versus-host disease after bone marrow transplantation across minor histocompatibility barriers in mice. Prevention by removing mature T cells from marrow.

CD4pos, NK1.1pos T cells promptly produce interleukin 4 in response to in vivo challenge with anti-CD3.

An invariant T cell receptor alpha chain is used by a unique subset of major histocompatibility complex class I-specific CD4+ and CD4-8- T cells in mice and humans.

Beta 2-microglobulin-dependent T cells are dispensable for allergen-induced T helper 2 responses.

Requirements for CD1d recognition by human invariant Valpha24+ CD4-CD8- T cells

alpha/beta-T cell receptor (TCR)+CD4-CD8- (NKT) thymocytes prevent insulin-dependent diabetes mellitus in nonobese diabetic (NOD)/Lt mice by the influence of interleukin (IL)-4 and/or IL-10.

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