Host-virus interactions during malaria infection in hepatitis B virus transgenic mice. - Wikidata - wikimedia - TriplyDB

Host-virus interactions during malaria infection in hepatitis B virus transgenic mice.

Host-virus interactions during malaria infection in hepatitis B virus transgenic mice.

http://www.wikidata.org/entity/Q36368728

about

Stealth and cunning: hepatitis B and hepatitis C viruses.Current progress in the development of therapeutic vaccines for chronic hepatitis B virus infection Immunology of hepatitis B virus and hepatitis C virus infection Hepatitis C virus infection may lead to slower emergence of P. falciparum in blood Hepatitis B infection is associated with asymptomatic malaria in the Brazilian Amazon.Pathogenesis of hepatitis B virus infection Temporal association of acute hepatitis A and Plasmodium falciparum malaria in children.Hydrodynamic injection of viral DNA: a mouse model of acute hepatitis B virus infection Mycobacterium-induced potentiation of type 1 immune responses and protection against malaria are host specific.Interleukin-18 inhibits hepatitis B virus replication in the livers of transgenic mice.The dynamics, causes and possible prevention of Hepatitis E outbreaks Hepatitis B virus infection does not significantly influence Plasmodium parasite density in asymptomatic infections in Ghanaian transfusion recipients.T cell-, interleukin-12-, and gamma interferon-driven viral clearance in measles virus-infected brain tissue The chimpanzee model for hepatitis B virus infection.Activation and function of hepatic NK cells in hepatitis B infection: an underinvestigated innate immune response.Jaundice in malaria.The expanding realm of heterologous immunity: friend or foe?Immunopathogenesis of hepatitis B virus infection.The impact of successive infections on the lung microenvironment.A novel IL-17-dependent mechanism of cross protection: respiratory infection with mycoplasma protects against a secondary listeria infection.Experimental models and therapeutic approaches for HBV.New concepts in the immunopathogenesis of chronic hepatitis B: the importance of the innate immune response.HBV Core Protein Enhances Cytokine Production.Co-infection of malaria and gamma-herpesvirus: exacerbated lung inflammation or cross-protection depends on the stage of viral infection.[Seroprevalence of hepatitis B and malaria infection in Lábrea, Brazilian western Amazon: estimates of coinfection rates].[Coinfection between hepatitis B virus and malaria: clinical, serologic and immunologic aspects].Pathogenetic and antiviral immune responses against hepatitis B virus

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Host-virus interactions during malaria infection in hepatitis B virus transgenic mice.

http://www.wikidata.org/entity/Q36368728

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2000 nî lūn-bûn

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2000年の論文

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2000年学术文章

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2000年学术文章

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2000年学术文章

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2000年学术文章

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2000年学术文章

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2000年學術文章

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2000年學術文章

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2000年學術文章

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Host-virus interactions during malaria infection in hepatitis B virus transgenic mice.

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Host-virus interactions during malaria infection in hepatitis B virus transgenic mice.

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Host-virus interactions during malaria infection in hepatitis B virus transgenic mice.

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Host-virus interactions during malaria infection in hepatitis B virus transgenic mice.

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Host-virus interactions during malaria infection in hepatitis B virus transgenic mice.

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Host-virus interactions during malaria infection in hepatitis B virus transgenic mice.

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Journal of Experimental Medicine

P1476

Host-virus interactions during malaria infection in hepatitis B virus transgenic mice.

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P2093

F V Chisari

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K Kakimi

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L G Guidotti

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V Pasquetto

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P2860

Functional role of type I and type II interferons in antiviral defense

Multiple defects of immune cell function in mice with disrupted interferon-gamma genes

Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection

Inhibition of hepatitis B virus replication during adenovirus and cytomegalovirus infections in transgenic mice

Relative sensitivity of hepatitis B virus and other hepatotropic viruses to the antiviral effects of cytokines.

Gamma interferon production is critical for protective immunity to infection with blood-stage Plasmodium berghei XAT but neither NO production nor NK cell activation is critical.

IFN-gamma inhibits development of Plasmodium berghei exoerythrocytic stages in hepatocytes by an L-arginine-dependent effector mechanism.

Immunopathology of malaria: role of cytokine production and adhesion molecules.

Cellular mechanisms in the immune response to malaria in Plasmodium vinckei-infected mice.

A Th1-associated increase in tumor necrosis factor alpha expression in the spleen correlates with resistance to blood-stage malaria in mice.

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529-536

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10.1084/JEM.192.4.529

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192

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10952722

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2193244

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