Processing of Mycobacterium tuberculosis antigen 85B involves intraphagosomal formation of peptide-major histocompatibility complex II complexes and is inhibited by live bacilli that decrease phagosome maturation.
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Mycobacterium tuberculosis: Manipulator of Protective ImmunityThe role of IL-10 in Mycobacterium avium subsp. paratuberculosis infectionCurrent efforts and future prospects in the development of live mycobacteria as vaccinesA genetic screen for Mycobacterium tuberculosis mutants defective for phagosome maturation arrest identifies components of the ESX-1 secretion systemThe Delta fbpA mutant derived from Mycobacterium tuberculosis H37Rv has an enhanced susceptibility to intracellular antimicrobial oxidative mechanisms, undergoes limited phagosome maturation and activates macrophages and dendritic cells.The Mycobacterium tuberculosis phagosome is a HLA-I processing competent organelleMycobacterium tuberculosis EsxH inhibits ESCRT-dependent CD4+ T-cell activationTo achieve an earlier IFN-γ response is not sufficient to control Mycobacterium tuberculosis infection in mice.BCG vaccine mediated reduction in the MHC-II expression of macrophages and dendritic cells is reversed by activation of Toll-like receptors 7 and 9.Regulation of antigen presentation by Mycobacterium tuberculosis: a role for Toll-like receptorsThe fbpA/sapM double knock out strain of Mycobacterium tuberculosis is highly attenuated and immunogenic in macrophagesRole of phagosomes and major histocompatibility complex class II (MHC-II) compartment in MHC-II antigen processing of Mycobacterium tuberculosis in human macrophages.Mycobacterium and the coat of many lipids.Relief from Zmp1-mediated arrest of phagosome maturation is associated with facilitated presentation and enhanced immunogenicity of mycobacterial antigens.Eicosanoid pathways regulate adaptive immunity to Mycobacterium tuberculosis.Bacterial Membrane Vesicles Mediate the Release of Mycobacterium tuberculosis Lipoglycans and Lipoproteins from Infected Macrophages.Class II MHC antigen presentation defect in neonatal monocytes is not correlated with decreased MHC-II expressionPresentation of phagocytosed antigens by MHC class I and II.MHC molecules and microbial antigen processing in phagosomes.Delayed protection by ESAT-6-specific effector CD4+ T cells after airborne M. tuberculosis infection.Evasion and subversion of antigen presentation by Mycobacterium tuberculosis.Effect of isoniazid therapy for latent TB infection on QuantiFERON-TB gold in-tube responses in adults with positive tuberculin skin test results in a high TB incidence area: a controlled study.A mutant of Mycobacterium tuberculosis H37Rv that lacks expression of antigen 85A is attenuated in mice but retains vaccinogenic potential.The ins and outs of MHC class II-mediated antigen processing and presentation.Giving CD4+ T cells the slip: viral interference with MHC class II-restricted antigen processing and presentation.Human phagosome processing of Mycobacterium tuberculosis antigens is modulated by interferon-γ and interleukin-10.Cutting edge: Nicastrin and related components of γ-secretase generate a peptide epitope facilitating immune recognition of intracellular mycobacteria, through MHC class II-dependent priming of T cells.Suboptimal Antigen Presentation Contributes to Virulence of Mycobacterium tuberculosis In Vivo.Mycobacterium tuberculosis Membrane Vesicles Inhibit T Cell Activation.Phagosomal processing of Mycobacterium tuberculosis antigen 85B is modulated independently of mycobacterial viability and phagosome maturation.Insilico analysis and molecular docking of resuscitation promoting factor B (RpfB) protein of Mycobacterium tuberculosis.Identification of the Mycobacterium tuberculosis protein PE-PGRS62 as a novel effector that functions to block phagosome maturation and inhibit iNOS expression.Mycobacterium tuberculosis synergizes with ATP to induce release of microvesicles and exosomes containing major histocompatibility complex class II molecules capable of antigen presentation.Autophagy enhances the efficacy of BCG vaccine by increasing peptide presentation in mouse dendritic cells.Mycobacterium tuberculosis-induced miR-155 subverts autophagy by targeting ATG3 in human dendritic cells.Mycobacterium tuberculosis Exploits a Molecular Off Switch of the Immune System for Intracellular Survival.MHC class II antigen internalizationRapid loss of early antigen-presenting activity of lymph node dendritic cells against Ag85A protein following Mycobacterium bovis BCG infection.
P2860
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P2860
Processing of Mycobacterium tuberculosis antigen 85B involves intraphagosomal formation of peptide-major histocompatibility complex II complexes and is inhibited by live bacilli that decrease phagosome maturation.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Processing of Mycobacterium tu ...... decrease phagosome maturation.
@ast
Processing of Mycobacterium tu ...... decrease phagosome maturation.
@en
type
label
Processing of Mycobacterium tu ...... decrease phagosome maturation.
@ast
Processing of Mycobacterium tu ...... decrease phagosome maturation.
@en
prefLabel
Processing of Mycobacterium tu ...... decrease phagosome maturation.
@ast
Processing of Mycobacterium tu ...... decrease phagosome maturation.
@en
P2093
P2860
P356
P1476
Processing of Mycobacterium tu ...... decrease phagosome maturation.
@en
P2093
P2860
P304
P356
10.1084/JEM.194.10.1421
P407
P577
2001-11-01T00:00:00Z