Rapid cytotoxic T lymphocyte activation occurs in the draining lymph nodes after cutaneous herpes simplex virus infection as a result of early antigen presentation and not the presence of virus
about
Inhibition of MHC class I is a virulence factor in herpes simplex virus infection of mice.The dendritic and T cell responses to herpes simplex virus-1 are modulated by dietary vitamin EImmunosuppressive networks in the tumour environment and their therapeutic relevanceDendritic cell migration limits the duration of CD8+ T-cell priming to peripheral viral antigen.Killing of targets by CD8 T cells in the mouse spleen follows the law of mass action.Herpes simplex virus-specific memory CD8+ T cells are selectively activated and retained in latently infected sensory gangliaCross-recognition of N-formylmethionine peptides is a general characteristic of H2-M3-restricted CD8+ T cells.Kinetics of virus-specific CD8+ -T-cell expansion and trafficking following central nervous system infection.The early expression of glycoprotein B from herpes simplex virus can be detected by antigen-specific CD8+ T cells.In vivo ablation of CD11c-positive dendritic cells increases susceptibility to herpes simplex virus type 1 infection and diminishes NK and T-cell responses.Cornea-infiltrating and lymph node dendritic cells contribute to CD4+ T cell expansion after herpes simplex virus-1 ocular infectionHome sweet home: How do virus specific T cells navigate to the skin?The role of dendritic cell subsets in selection between tolerance and immunity.Us3 kinase encoded by herpes simplex virus 1 mediates downregulation of cell surface major histocompatibility complex class I and evasion of CD8+ T cellsIntracellular regulation of cross-presentation during dendritic cell maturation.CD8+ T cells from a novel T cell receptor transgenic mouse induce liver-stage immunity that can be boosted by blood-stage infection in rodent malaria.Innate and adaptive immune responses to herpes simplex virusVirus-induced tumor inflammation facilitates effective DC cancer immunotherapy in a Treg-dependent manner in miceRapid quantification of naive alloreactive T cells by TNF-alpha production and correlation with allograft rejection in mice.Tumor-associated antigen expressing Listeria monocytogenes induces effective primary and memory T-cell responses against hepatic colorectal cancer metastasesEffector and memory CD8+ T cells as seen in immunity to malaria.Viral targeting of fibroblastic reticular cells contributes to immunosuppression and persistence during chronic infection.Dendritic cell activation and memory cell development are impaired among mice administered medroxyprogesterone acetate prior to mucosal herpes simplex virus type 1 infectionDancing with the enemy: the interplay of herpes simplex virus with dendritic cells.Vaginal submucosal dendritic cells, but not Langerhans cells, induce protective Th1 responses to herpes simplex virus-2Signal 3 determines tolerance versus full activation of naive CD8 T cells: dissociating proliferation and development of effector function.The CD8alpha(+) dendritic cell is responsible for inducing peripheral self-tolerance to tissue-associated antigens.Peyer's patch dendritic cells process viral antigen from apoptotic epithelial cells in the intestine of reovirus-infected mice.IL-4 receptor expression on CD8+ T cells is required for the development of protective memory responses against liver stages of malaria parasites.Dendritic cells loaded with apoptotic antibody-coated tumor cells provide protective immunity against B-cell lymphoma in vivoHerpes simplex virus type 2 induces rapid cell death and functional impairment of murine dendritic cells in vitro.The limits of protection by "memory" T cells in Ig-/- mice persistently infected with a gamma-herpesvirusStress-induced glucocorticoids at the earliest stages of herpes simplex virus-1 infection suppress subsequent antiviral immunity, implicating impaired dendritic cell functionA marked reduction in priming of cytotoxic CD8+ T cells mediated by stress-induced glucocorticoids involves multiple deficiencies in cross-presentation by dendritic cellsAntigen presentation by monocytes and monocyte-derived cells.Interplay between alpha/beta and gamma interferons with B, T, and natural killer cells in the defense against herpes simplex virus type 1.Differential roles of migratory and resident DCs in T cell priming after mucosal or skin HSV-1 infectionDifferential CMV-specific CD8+ effector T cell responses in the lung allograft predominate over the blood during human primary infectionTransience of MHC Class I-restricted antigen presentation after influenza A virus infection.Herpes simplex keratitis and dendritic cells at the crossroads: lessons from the past and a view into the future.
P2860
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P2860
Rapid cytotoxic T lymphocyte activation occurs in the draining lymph nodes after cutaneous herpes simplex virus infection as a result of early antigen presentation and not the presence of virus
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Rapid cytotoxic T lymphocyte a ...... and not the presence of virus
@ast
Rapid cytotoxic T lymphocyte a ...... and not the presence of virus
@en
type
label
Rapid cytotoxic T lymphocyte a ...... and not the presence of virus
@ast
Rapid cytotoxic T lymphocyte a ...... and not the presence of virus
@en
prefLabel
Rapid cytotoxic T lymphocyte a ...... and not the presence of virus
@ast
Rapid cytotoxic T lymphocyte a ...... and not the presence of virus
@en
P2093
P2860
P921
P356
P1476
Rapid cytotoxic T lymphocyte a ...... and not the presence of virus
@en
P2093
Chris M Smith
Claerwen M Jones
Francis R Carbone
Scott N Mueller
William R Heath
P2860
P304
P356
10.1084/JEM.20012023
P407
P577
2002-03-01T00:00:00Z