Secondary heavy chain rearrangement: a mechanism for generating anti-double-stranded DNA B cells.
about
Secondary receptor editing in the generation of autoimmunityB cell receptor editing in tolerance and autoimmunityTwo distinct populations of H chain-edited B cells show differential surrogate L chain dependence.The role of rearrangement at the second Ig heavy chain locus in maintaining B cell tolerance to DNA.Receptor editing in lymphocyte development and central tolerance.Contribution of V(H) replacement products in mouse antibody repertoire.VH replacement in primary immunoglobulin repertoire diversification.The pathophysiology of chronic graft-versus-host disease: the unveiling of an enigmaTherapeutic potential of CD8+ cytotoxic T lymphocytes in SLEPositive selection of natural poly-reactive B cells in the periphery occurs independent of heavy chain allelic inclusionThe molecular basis and biological significance of VH replacement.B-cell tolerance defects in the B6.Aec1/2 mouse model of Sjögren's syndrome.The regulation and activation of lupus-associated B cells.Light chain editing generates polyreactive antibodies in chronic graft-versus-host reaction.Potential contribution of VH gene replacement in immunity and disease.Positive selection of the peripheral B cell repertoire in gut-associated lymphoid tissues.Editing and escape from editing in anti-DNA B cells.T cells, murine chronic graft-versus-host disease and autoimmunity.A new site-directed transgenic rheumatoid factor mouse model demonstrates extrafollicular class switch and plasmablast formationTrials and Tribulations with VH Replacement.Central tolerance regulates B cells reactive with Goodpasture antigen alpha3(IV)NC1 collagen.Btk regulates localization, in vivo activation, and class switching of anti-DNA B cells.Diversification of the Ig variable region gene repertoire of synovial B lymphocytes by nucleotide insertion and deletion.Antibodies in a heavy chain knock-in mouse exhibit characteristics of early heavy chain rearrangement.Interleukin-6 is responsible for aberrant B-cell receptor-mediated regulation of RAG expression in systemic lupus erythematosus.The inhibitory Fcγ receptor modulates autoimmunity by limiting the accumulation of immunoglobulin G+ anti-DNA plasma cells
P2860
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P2860
Secondary heavy chain rearrangement: a mechanism for generating anti-double-stranded DNA B cells.
description
2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年学术文章
@zh-my
2003年学术文章
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2003年學術文章
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2003年學術文章
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2003年學術文章
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name
Secondary heavy chain rearrang ...... i-double-stranded DNA B cells.
@ast
Secondary heavy chain rearrang ...... i-double-stranded DNA B cells.
@en
type
label
Secondary heavy chain rearrang ...... i-double-stranded DNA B cells.
@ast
Secondary heavy chain rearrang ...... i-double-stranded DNA B cells.
@en
prefLabel
Secondary heavy chain rearrang ...... i-double-stranded DNA B cells.
@ast
Secondary heavy chain rearrang ...... i-double-stranded DNA B cells.
@en
P2093
P2860
P356
P1476
Secondary heavy chain rearrang ...... i-double-stranded DNA B cells.
@en
P2093
Debora R Sekiguchi
Martin Weigert
Robert A Eisenberg
P2860
P356
10.1084/JEM.20020737
P407
P577
2003-01-01T00:00:00Z