The Trypanosoma cruzi trans-sialidase, through its COOH-terminal tandem repeat, upregulates interleukin 6 secretion in normal human intestinal microvascular endothelial cells and peripheral blood mononuclear cells
about
Diversity of microbial sialic acid metabolismBioactive lipids in Trypanosoma cruzi infectionCurrent understanding of immunity to Trypanosoma cruzi infection and pathogenesis of Chagas diseaseMaternal Trypanosoma cruzi infection upregulates capacity of uninfected neonate cells To produce pro- and anti-inflammatory cytokines.Trypanosoma cruzi trans-sialidase: a potent and specific survival factor for human Schwann cells by means of phosphatidylinositol 3-kinase/Akt signaling.Heterologous expression of Trypanosoma cruzi trans-sialidase in Leishmania major enhances virulenceCell-specific activation of nuclear factor-kappaB by the parasite Trypanosoma cruzi promotes resistance to intracellular infection.A trypanosomal protein synergizes with the cytokines ciliary neurotrophic factor and leukemia inhibitory factor to prevent apoptosis of neuronal cells.Role of the Toll/interleukin-1 receptor signaling pathway in host resistance and pathogenesis during infection with protozoan parasites.Innate and acquired immunity in the pathogenesis of Chagas disease.The surface-anchored NanA protein promotes pneumococcal brain endothelial cell invasion.Trypanosoma cruzi-Derived Neurotrophic Factor: Role in Neural Repair and Neuroprotection.The chemistry and biology of trypanosomal trans-sialidases: virulence factors in Chagas disease and sleeping sickness.Trans-sialidase-like sequences from Trypanosoma congolense conserve most of the critical active site residues found in other trans-sialidases.Auto-antibodies to receptor tyrosine kinases TrkA, TrkB and TrkC in patients with chronic Chagas' disease.Endothelial cell signalling induced by trans-sialidase from Trypanosoma cruzi.Immediate/early response to Trypanosoma cruzi infection involves minimal modulation of host cell transcription.The Trypanosoma cruzi trans-sialidase is a T cell-independent B cell mitogen and an inducer of non-specific Ig secretion.Two trans-sialidase forms with different sialic acid transfer and sialidase activities from Trypanosoma congolense.Expression of trypomastigote trans-sialidase in metacyclic forms of Trypanosoma cruzi increases parasite escape from its parasitophorous vacuole.Proteomic analysis reveals different composition of extracellular vesicles released by two Trypanosoma cruzi strains associated with their distinct interaction with host cells.
P2860
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P2860
The Trypanosoma cruzi trans-sialidase, through its COOH-terminal tandem repeat, upregulates interleukin 6 secretion in normal human intestinal microvascular endothelial cells and peripheral blood mononuclear cells
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
The Trypanosoma cruzi trans-si ...... pheral blood mononuclear cells
@ast
The Trypanosoma cruzi trans-si ...... pheral blood mononuclear cells
@en
type
label
The Trypanosoma cruzi trans-si ...... pheral blood mononuclear cells
@ast
The Trypanosoma cruzi trans-si ...... pheral blood mononuclear cells
@en
prefLabel
The Trypanosoma cruzi trans-si ...... pheral blood mononuclear cells
@ast
The Trypanosoma cruzi trans-si ...... pheral blood mononuclear cells
@en
P2093
P2860
P921
P356
P1476
The Trypanosoma cruzi trans-si ...... pheral blood mononuclear cells
@en
P2093
P2860
P304
P356
10.1084/JEM.190.12.1825
P407
P577
1999-12-01T00:00:00Z