Recognition of the class Ib molecule Qa-1(b) by putative activating receptors CD94/NKG2C and CD94/NKG2E on mouse natural killer cells - Wikidata - wikimedia - TriplyDB

Recognition of the class Ib molecule Qa-1(b) by putative activating receptors CD94/NKG2C and CD94/NKG2E on mouse natural killer cells

Recognition of the class Ib molecule Qa-1(b) by putative activating receptors CD94/NKG2C and CD94/NKG2E on mouse natural killer cells

http://www.wikidata.org/entity/Q36375639

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Natural killer cell responses during viral infections: flexibility and conditioning of innate immunity by experience Crystal structure of the murine NK cell-activating receptor NKG2D at 1.95 A Bone marrow allograft rejection mediated by a novel murine NK receptor, NKG2I An MHC class Ib-restricted CD8 T cell response confers antiviral immunity The nonpolymorphic MHC Qa-1b mediates CD8+ T cell surveillance of antigen-processing defects Natural killer cell differentiation driven by Tyro3 receptor tyrosine kinases Regulation of NK cell repertoire and function in the liver.NK gene complex dynamics and selection for NK cell receptors.The fester locus in Botryllus schlosseri experiences selection.Analysis of Qa-1(b) peptide binding specificity and the capacity of CD94/NKG2A to discriminate between Qa-1-peptide complexes Upregulation of CD94 on CD8+T cells in anterior chamber-associated immune deviation Uterine Natural Killer Cells: Functional Distinctions and Influence on Pregnancy in Humans and Mice.NKG2A inhibits invariant NKT cell activation in hepatic injury.Development and function of CD94-deficient natural killer cells Implications of CD94 deficiency and monoallelic NKG2A expression for natural killer cell development and repertoire formation gammadeltaTCR ligands and lineage commitment.Primate-specific regulation of natural killer cells.On guard--activating NK cell receptors.Decrease of peripheral and intestinal NKG2A-positive T cells in patients with ulcerative colitis.Mammalian non-classical major histocompatibility complex I and its receptors: Important contexts of gene, evolution, and immunity.Diverse roles of non-diverse molecules: MHC class Ib molecules in host defense and control of autoimmunity The innate immune response to tumors and its role in the induction of T-cell immunity.The natural killer cell dysfunction of aged mice is due to the bone marrow stroma and is not restored by IL-15/IL-15Rα treatment.A subset of natural killer cells achieves self-tolerance without expressing inhibitory receptors specific for self-MHC molecules.IFN-gamma-mediated negative feedback regulation of NKT-cell function by CD94/NKG2.The role of NK cells and NK cell receptors in autoimmune disease.Macrophages help NK cells to attack tumor cells by stimulatory NKG2D ligand but protect themselves from NK killing by inhibitory ligand Qa-1.Non-classical MHC class I molecules on intestinal epithelial cells: mediators of mucosal crosstalk.Nonstochastic coexpression of activation receptors on murine natural killer cells Elimination in vivo of developing T cells by natural killer cells.Expression of natural killer receptor alleles at different Ly49 loci occurs independently and is regulated by major histocompatibility complex class I molecules.The Inhibitory Receptor NKG2A Sustains Virus-Specific CD8⁺ T Cells in Response to a Lethal Poxvirus Infection KLRE/I1 and KLRE/I2: a novel pair of heterodimeric receptors that inversely regulate NK cell cytotoxicity Natural killer cells and their receptors in multiple sclerosis.Antiviral NK cell responses in HIV infection: I. NK cell receptor genes as determinants of HIV resistance and progression to AIDS.Gammadelta T lymphocyte homeostasis is negatively regulated by beta2-microglobulin.Activation of natural killer cells inhibits liver fibrosis: a novel strategy to treat liver fibrosis.DAP10- and DAP12-associated receptors in innate immunity.Class I HLA haplotypes form two schools that educate NK cells in different ways.Contribution of the Ly49E natural killer receptor in the immune response to Plasmodium berghei infection and control of hepatic parasite development

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Recognition of the class Ib molecule Qa-1(b) by putative activating receptors CD94/NKG2C and CD94/NKG2E on mouse natural killer cells

http://www.wikidata.org/entity/Q36375639

description

1999 nî lūn-bûn

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name

Recognition of the class Ib mo ...... on mouse natural killer cells

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Recognition of the class Ib mo ...... on mouse natural killer cells

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Recognition of the class Ib mo ...... on mouse natural killer cells

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Recognition of the class Ib mo ...... on mouse natural killer cells

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Recognition of the class Ib mo ...... on mouse natural killer cells

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A M Jamieson

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D H Raulet

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R E Vance

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P2860

HLA-E-bound peptides influence recognition by inhibitory and triggering CD94/NKG2 receptors: preferential response to an HLA-G-derived nonamer

Activation of NK cells and T cells by NKG2D, a receptor for stress-inducible MICA

An activating immunoreceptor complex formed by NKG2D and DAP10

CLUSTAL W: improving the sensitivity of progressive multiple sequence alignment through sequence weighting, position-specific gap penalties and weight matrix choice

Immunoreceptor DAP12 bearing a tyrosine-based activation motif is involved in activating NK cells

Mouse CD94/NKG2A is a natural killer cell receptor for the nonclassical major histocompatibility complex (MHC) class I molecule Qa-1(b)

Simple methods for estimating the numbers of synonymous and nonsynonymous nucleotide substitutions

The human major histocompatibility complex class Ib molecule HLA-E binds signal sequence-derived peptides with primary anchor residues at positions 2 and 9

In search of the 'missing self': MHC molecules and NK cell recognition

Cloning of NKG2-F, a new member of the NKG2 family of human natural killer cell receptor genes

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