Virulent strains of Helicobacter pylori demonstrate delayed phagocytosis and stimulate homotypic phagosome fusion in macrophages - Wikidata - wikimedia - TriplyDB

Virulent strains of Helicobacter pylori demonstrate delayed phagocytosis and stimulate homotypic phagosome fusion in macrophages

Virulent strains of Helicobacter pylori demonstrate delayed phagocytosis and stimulate homotypic phagosome fusion in macrophages

http://www.wikidata.org/entity/Q36375886

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Helicobacter pylori persistence: biology and disease An Overview of Helicobacter pylori VacA Toxin Biology Role of Helicobacter pylori infection in pathogenesis of gastric carcinoma Morphological and Cellular Features of Innate Immune Reaction in Helicobacter pylori Gastritis: A Brief Review A role for altered phagosome maturation in the long-term persistence of Helicobacter pylori infection The Immunomodulator VacA Promotes Immune Tolerance and Persistent Helicobacter pylori Infection through Its Activities on T-Cells and Antigen-Presenting Cells Fluorescence microscopy methods for determining the viability of bacteria in association with mammalian cells.Rab14 is critical for maintenance of Mycobacterium tuberculosis phagosome maturation arrest Helicobacter pylori resists phagocytosis by macrophages: quantitative assessment by confocal microscopy and fluorescence-activated cell sorting.Interaction of Helicobacter pylori with professional phagocytes: role of the cag pathogenicity island and translocation, phosphorylation and processing of CagA.Helicobacter pylori impairs murine dendritic cell responses to infection Oxidative Stress Resulting From Helicobacter pylori Infection Contributes to Gastric Carcinogenesis.Immune responses to Helicobacter pylori infection.ppGpp conjures bacterial virulence VAMP3 null mice display normal constitutive, insulin- and exercise-regulated vesicle trafficking.Helicobacter pylori and gastric cancer: factors that modulate disease risk.Smooth and rough lipopolysaccharide phenotypes of Brucella induce different intracellular trafficking and cytokine/chemokine release in human monocytes.Immune evasion strategies used by Helicobacter pylori.Pathogenic trickery: deception of host cell processes.Mechanisms of microbial escape from phagocyte killing.Role of innate immunity in Helicobacter pylori-induced gastric malignancy.Helicobacter pylori colonization ameliorates glucose homeostasis in mice through a PPAR γ-dependent mechanism.Modeling the Regulatory Mechanisms by Which NLRX1 Modulates Innate Immune Responses to Helicobacter pylori Infection Helicobacter pylori phagosome maturation in primary human macrophages.Helicobacter pylori protein JHP0290 binds to multiple cell types and induces macrophage apoptosis via tumor necrosis factor (TNF)-dependent and independent pathways Helicobacter pylori persistence: an overview of interactions between H. pylori and host immune defenses Francisella tularensis LVS evades killing by human neutrophils via inhibition of the respiratory burst and phagosome escape Immunofluorescence and confocal microscopy of neutrophils.Suppression of cell division-associated genes by Helicobacter pylori attenuates proliferation of RAW264.7 monocytic macrophage cells.Differential infection of mononuclear phagocytes by Francisella tularensis: role of the macrophage mannose receptor.Role of urease in megasome formation and Helicobacter pylori survival in macrophages.Phosphoinositide3-kinase regulates actin polymerization during delayed phagocytosis of Helicobacter pylori.Helicobacter pylori is invasive and it may be a facultative intracellular organism.Persistent bacterial infections: the interface of the pathogen and the host immune system.Helicobacter pylori VacA, a paradigm for toxin multifunctionality.VacA-associated inhibition of T-cell function: reviewed and reconsidered.Phagocytosis and persistence of Helicobacter pylori.Multifactorial etiology of gastric cancer The impact of autophagic processes on the intracellular fate of Helicobacter pylori: more tricks from an enigmatic pathogen?Regulation of vacuolar pH and its modulation by some microbial species

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P2860

Virulent strains of Helicobacter pylori demonstrate delayed phagocytosis and stimulate homotypic phagosome fusion in macrophages

http://www.wikidata.org/entity/Q36375886

description

2000 nî lūn-bûn

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2000年の論文

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2000年学术文章

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Virulent strains of Helicobact ...... hagosome fusion in macrophages

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Journal of Experimental Medicine

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Virulent strains of Helicobact ...... hagosome fusion in macrophages

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P2093

B Kang

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L A Allen

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L S Schlesinger

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P2860

Phagocytosis of Mycobacterium tuberculosis is mediated by human monocyte complement receptors and complement component C3

Activation of phospholipase D is tightly coupled to the phagocytosis of Mycobacterium tuberculosis or opsonized zymosan by human macrophages

Lack of acidification in Mycobacterium phagosomes produced by exclusion of the vesicular proton-ATPase

Virulence and pathogenicity of Helicobacter pylori.

Characterization of an acidic-pH-inducible stress protein (hsp70), a putative sulfatide binding adhesin, from Helicobacter pylori.

Biochemistry of type IV secretion.

Heterogeneity in levels of vacuolating cytotoxin gene (vacA) transcription among Helicobacter pylori strains

Fusion of Chlamydia trachomatis-containing inclusions is inhibited at low temperatures and requires bacterial protein synthesis.

Parasitism by the "slow" bacterium Helicobacter pylori leads to altered gastric homeostasis and neoplasia.

Epithelial cytotoxicity, immune responses, and inflammatory components of Helicobacter pylori gastritis.

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115-128

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scholarly article

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10.1084/JEM.191.1.115

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1

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191

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12692894

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10620610

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Helicobacter pylori

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2195807

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