Polypathology and dementia after brain trauma: Does brain injury trigger distinct neurodegenerative diseases, or should they be classified together as traumatic encephalopathy?
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Emerging Roles for the Immune System in Traumatic Brain InjuryAnalysis of the Role of CX3CL1 (Fractalkine) and Its Receptor CX3CR1 in Traumatic Brain and Spinal Cord Injury: Insight into Recent Advances in Actions of Neurochemokine Agents.Risk of hospitalization with neurodegenerative disease after moderate-to-severe traumatic brain injury in the working-age population: A retrospective cohort study using the Finnish national health registriesDendritic Spine Loss and Chronic White Matter Inflammation in a Mouse Model of Highly Repetitive Head Trauma.Evidence to Consider Angiotensin II Receptor Blockers for the Treatment of Early Alzheimer's Disease.What is the Relationship of Traumatic Brain Injury to Dementia?Disordered APP metabolism and neurovasculature in trauma and aging: Combined risks for chronic neurodegenerative disorders.Salsalate treatment following traumatic brain injury reduces inflammation and promotes a neuroprotective and neurogenic transcriptional response with concomitant functional recovery.Association of Traumatic Brain Injury With Late-Life Neurodegenerative Conditions and Neuropathologic Findings.The Effect of the APOE4 Gene on Accumulation of Aβ40 After Brain Injury Cannot Be Reversed by Increasing apoE4 Protein.ApoE4-associated phospholipid dysregulation contributes to development of Tau hyper-phosphorylation after traumatic brain injury.Dementia After Moderate-Severe Traumatic Brain Injury: Coexistence of Multiple Proteinopathies.Inhibition of Endocannabinoid Degradation Improves Outcomes from Mild Traumatic Brain Injury: A Mechanistic Role for Synaptic Hyperexcitability.Traumatic brain injury may not increase the risk of Alzheimer disease.Targeting Neuroinflammation to Treat Alzheimer's Disease.Injury cascades in TBI-related neurodegeneration.Traumatic Brain Injury Results in Cellular, Structural and Functional Changes Resembling Motor Neuron Disease.Single severe traumatic brain injury produces progressive pathology with ongoing contralateral white matter damage one year after injury.Traumatic Brain Injury and Alzheimer's Disease: The Cerebrovascular Link.S100A9-Driven Amyloid-Neuroinflammatory Cascade in Traumatic Brain Injury as a Precursor State for Alzheimer's DiseaseIs traumatic brain injury a risk factor for neurodegeneration? A meta-analysis of population-based studies
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Polypathology and dementia after brain trauma: Does brain injury trigger distinct neurodegenerative diseases, or should they be classified together as traumatic encephalopathy?
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2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年學術文章
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name
Polypathology and dementia aft ...... r as traumatic encephalopathy?
@ast
Polypathology and dementia aft ...... r as traumatic encephalopathy?
@en
type
label
Polypathology and dementia aft ...... r as traumatic encephalopathy?
@ast
Polypathology and dementia aft ...... r as traumatic encephalopathy?
@en
prefLabel
Polypathology and dementia aft ...... r as traumatic encephalopathy?
@ast
Polypathology and dementia aft ...... r as traumatic encephalopathy?
@en
P2860
P921
P1476
Polypathology and dementia aft ...... r as traumatic encephalopathy?
@en
P2093
Patricia M Washington
P2860
P304
P356
10.1016/J.EXPNEUROL.2015.06.015
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P478
P577
2015-06-16T00:00:00Z