Interleukin-13 protects from atherosclerosis and modulates plaque composition by skewing the macrophage phenotype. - Wikidata - wikimedia - TriplyDB

Interleukin-13 protects from atherosclerosis and modulates plaque composition by skewing the macrophage phenotype.

Interleukin-13 protects from atherosclerosis and modulates plaque composition by skewing the macrophage phenotype.

http://www.wikidata.org/entity/Q36379539

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Stabilization of high-risk plaques Macrophage Heterogeneity and Plasticity: Impact of Macrophage Biomarkers on Atherosclerosis I-L-C-2 it: type 2 immunity and group 2 innate lymphoid cells in homeostasis Pharmacological Treatment with Annexin A1 Reduces Atherosclerotic Plaque Burden in LDLR-/- Mice on Western Type Diet HDL induces the expression of the M2 macrophage markers arginase 1 and Fizz-1 in a STAT6-dependent process.Concise Review: Fat and Furious: Harnessing the Full Potential of Adipose-Derived Stromal Vascular Fraction Type-2 innate lymphoid cells control the development of atherosclerosis in mice Inflammation and its resolution as determinants of acute coronary syndromes.Cell-intrinsic lysosomal lipolysis is essential for alternative activation of macrophages Diabetes alters activation and repression of pro- and anti-inflammatory signaling pathways in the vasculature.Macrophages in atherosclerosis: a dynamic balance.Classical and Alternative Activation and Metalloproteinase Expression Occurs in Foam Cell Macrophages in Male and Female ApoE Null Mice in the Absence of T and B Lymphocytes.Programmed cell death-1 deficiency exacerbates T cell activation and atherogenesis despite expansion of regulatory T cells in atherosclerosis-prone mice.Sex differences in inflammation during atherosclerosis.Lymphocyte migration into atherosclerotic plaque.Endothelial cells suppress monocyte activation through secretion of extracellular vesicles containing antiinflammatory microRNAs.Cytokines in atherosclerosis: Key players in all stages of disease and promising therapeutic targets.Valsartan Attenuates Atherosclerosis via Upregulating the Th2 Immune Response in Prolonged Angiotensin II-Treated ApoE(-/-) Mice Plaque Size Is Decreased but M1 Macrophage Polarization and Rupture Related Metalloproteinase Expression Are Maintained after Deleting T-Bet in ApoE Null Mice.Cytokines: roles in atherosclerosis disease progression and potential therapeutic targets Macrophage deficiency of Akt2 reduces atherosclerosis in Ldlr null mice Targeting CD64 mediates elimination of M1 but not M2 macrophages in vitro and in cutaneous inflammation in mice and patient biopsies.MicroRNA-33-dependent regulation of macrophage metabolism directs immune cell polarization in atherosclerosis Adaptive immunity in atherogenesis: new insights and therapeutic approaches.Macrophage Phenotype and Function in Different Stages of Atherosclerosis.Liver-specific cholesteryl ester hydrolase deficiency attenuates sterol elimination in the feces and increases atherosclerosis in ldlr-/- mice Liver-specific transgenic expression of cholesteryl ester hydrolase reduces atherosclerosis in Ldlr-/- mice.Inflammation and immune system interactions in atherosclerosis.Molecular biology of atherosclerosis.High-density lipoprotein and atherosclerosis regression: evidence from preclinical and clinical studies Dynamic Aspects of Macrophage Polarization during Atherosclerosis Progression and Regression.Targeting the adaptive immune system: new strategies in the treatment of atherosclerosis.The potential role of IL-33/ST2 signaling in fibrotic diseases.Regression of Atherosclerosis: The Journey From the Liver to the Plaque and Back Regulatory T cells in atherosclerosis: critical immune regulatory function and therapeutic potential.Integrated regulation of the cellular metabolism and function of immune cells in adipose tissue.ATVB Distinguished Scientist Award: How Costimulatory and Coinhibitory Pathways Shape Atherosclerosis.Inflammatory processes in cardiovascular disease: a route to targeted therapies.MicroRNA regulation of macrophages in human pathologies.Malondialdehyde Epitopes as Targets of Immunity and the Implications for Atherosclerosis.

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Interleukin-13 protects from atherosclerosis and modulates plaque composition by skewing the macrophage phenotype.

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EMBO Molecular Medicine

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Larissa Cardilo-Reis

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Maik Drechsler

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Nikolina Papac-Milicevic

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Oliver Soehnlein

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Oswald Wagner

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Sabine M Schreier

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Sabrina Gruber

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P2860

Molecular and structural basis of cytokine receptor pleiotropy in the interleukin-4/13 system

Antagonism of miR-33 in mice promotes reverse cholesterol transport and regression of atherosclerosis

Cellular and molecular mechanisms of fibrosis

Insights into the initiation of type 2 immune responses

Interleukin-13 induces PSGL-1/P-selectin-dependent adhesion of eosinophils, but not neutrophils, to human umbilical vein endothelial cells under flow

IL-13 signaling through the IL-13alpha2 receptor is involved in induction of TGF-beta1 production and fibrosis

A major role for VCAM-1, but not ICAM-1, in early atherosclerosis

Interleukin-4-dependent production of PPAR-gamma ligands in macrophages by 12/15-lipoxygenase

Regression of atherosclerosis is characterized by broad changes in the plaque macrophage transcriptome

Inflammation, atherosclerosis, and coronary artery disease

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10.1002/EMMM.201201374

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10

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Christian Weber

Christoph J Binder

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231742251

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atherosclerosis

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3491837

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