Transgenic mouse models that explore the multistep hypothesis of intestinal neoplasia.
about
Genetic evaluation of candidate genes for the Mom1 modifier of intestinal neoplasia in miceFoxn1 regulates lineage progression in cortical and medullary thymic epithelial cells but is dispensable for medullary sublineage divergenceIntestinal tumorigenesis is suppressed in mice lacking the metalloproteinase matrilysinRegulation of intracellular beta-catenin levels by the adenomatous polyposis coli (APC) tumor-suppressor proteinA gnotobiotic transgenic mouse model for studying interactions between small intestinal enterocytes and intraepithelial lymphocytes.Field cancerization, clonality, and epithelial stem cells: the spread of mutated clones in epithelial sheets.Intestinal dysplasia induced by simian virus 40 T antigen is independent of p53.The adenomatous polyposis coli protein.Transforming functions of Simian Virus 40.ERK and Akt signaling pathways function through parallel mechanisms to promote mTORC1 signalingMechanisms of uropathogenic Escherichia coli persistence and eradication from the urinary tractExpression of wild-type and mutant simian virus 40 large tumor antigens in villus-associated enterocytes of transgenic mice.Tracing cell fates in human colorectal tumors from somatic microsatellite mutations: evidence of adenomas with stem cell architecture.Biomaterial-induced sarcoma: A novel model to study preneoplastic change.Enterocyte proliferation and intestinal hyperplasia induced by simian virus 40 T antigen require a functional J domainEffects of forced expression of an NH2-terminal truncated beta-Catenin on mouse intestinal epithelial homeostasis.Bi-transgenic mice reveal that K-rasVal12 augments a p53-independent apoptosis when small intestinal villus enterocytes reenter the cell cycle.Intestinal hyperplasia induced by simian virus 40 large tumor antigen requires E2F2.In vivo analysis of cadherin function in the mouse intestinal epithelium: essential roles in adhesion, maintenance of differentiation, and regulation of programmed cell deathThe mouse ileal lipid-binding protein gene: a model for studying axial patterning during gut morphogenesisSynergy between Apc min and an activated ras mutation is sufficient to induce colon carcinomas.Effects of p53 mutations on apoptosis in mouse intestinal and human colonic adenomas.Estrogenic modulation of uropathogenic Escherichia coli infection pathogenesis in a murine menopause model.Expression of conditional cre recombinase in epithelial tissues of transgenic mice.Cell-type specific regulation of gene expression by simian virus 40 T antigens.Simian virus 40 T-antigen-mediated gene regulation in enterocytes is controlled primarily by the Rb-E2F pathwayForced expression of the tumor suppressor adenomatosis polyposis coli protein induces disordered cell migration in the intestinal epithelium.T antigen transgenic mouse modelsGenetic engineering of carbohydrate biosynthetic pathways in transgenic mice demonstrates cell cycle-associated regulation of glycoconjugate production in small intestinal epithelial cells.Mutant KRAS promotes hyperplasia and alters differentiation in the colon epithelium but does not expand the presumptive stem cell pool.Transgenic Mouse Models of SV40-Induced Cancer.Expression of thin aggregative fimbriae promotes interaction of Salmonella typhimurium SR-11 with mouse small intestinal epithelial cells.Altered phenotype of HT29 colonic adenocarcinoma cells following expression of the DCC gene.Enterocyte differentiation is compatible with SV40 large T expression and loss of p53 function in human colonic Caco-2 cells. Status of the pRb1 and pRb2 tumor suppressor gene products.Viral oncogene expression in the stem/progenitor cell compartment of the mouse intestine induces adenomatous polyps.Ablation of 4E-BP1/2 prevents hyperglycemia-mediated induction of VEGF expression in the rodent retina and in Muller cells in culture.The Ron receptor tyrosine kinase is not required for adenoma formation in Apc(Min/+) miceMutated K-ras(Asp12) promotes tumourigenesis in Apc(Min) mice more in the large than the small intestines, with synergistic effects between K-ras and Wnt pathways.An intronic enhancer essential for tissue-specific expression of the aldolase B transgenes.Clinicopathological characteristics and survival analysis of primary duodenal cancers: a 14-year experience in a tertiary centre in South China.
P2860
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P2860
Transgenic mouse models that explore the multistep hypothesis of intestinal neoplasia.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Transgenic mouse models that explore the multistep hypothesis of intestinal neoplasia.
@ast
Transgenic mouse models that explore the multistep hypothesis of intestinal neoplasia.
@en
type
label
Transgenic mouse models that explore the multistep hypothesis of intestinal neoplasia.
@ast
Transgenic mouse models that explore the multistep hypothesis of intestinal neoplasia.
@en
prefLabel
Transgenic mouse models that explore the multistep hypothesis of intestinal neoplasia.
@ast
Transgenic mouse models that explore the multistep hypothesis of intestinal neoplasia.
@en
P2093
P2860
P356
P1476
Transgenic mouse models that explore the multistep hypothesis of intestinal neoplasia.
@en
P2093
P2860
P304
P356
10.1083/JCB.123.4.877
P407
P577
1993-11-01T00:00:00Z