STAT-3 contributes to pulmonary fibrosis through epithelial injury and fibroblast-myofibroblast differentiation. - Wikidata - wikimedia - TriplyDB

STAT-3 contributes to pulmonary fibrosis through epithelial injury and fibroblast-myofibroblast differentiation.

STAT-3 contributes to pulmonary fibrosis through epithelial injury and fibroblast-myofibroblast differentiation.

http://www.wikidata.org/entity/Q36385387

about

Effects of lipopolysaccharide, multiwalled carbon nantoubes, and the combination on lung alveolar epithelial cells Combined inhibition of TGFβ and PDGF signaling attenuates radiation-induced pulmonary fibrosis Epigenetic Regulation of Interleukin 6 by Histone Acetylation in Macrophages and Its Role in Paraquat-Induced Pulmonary Fibrosis.SIRT3 is attenuated in systemic sclerosis skin and lungs, and its pharmacologic activation mitigates organ fibrosis.Involvement of NF-κBIZ and related cytokines in age-associated renal fibrosis.SIRT3 attenuates AngII-induced cardiac fibrosis by inhibiting myofibroblasts transdifferentiation via STAT3-NFATc2 pathway.HMGB1 induces lung fibroblast to myofibroblast differentiation through NF‑κB‑mediated TGF‑β1 release.Increased aerobic glycolysis is important for the motility of activated VSMC and inhibited by indirubin-3'-monoxime.Mechanisms of carbon nanotube-induced pulmonary fibrosis: a physicochemical characteristic perspective.Blockade of extracellular heat shock protein 90α by 1G6-D7 attenuates pulmonary fibrosis through inhibiting ERK signaling.STAT3 controls COL1A2 enhancer activation cooperatively with JunB, regulates type I collagen synthesis posttranscriptionally, and is essential for lung myofibroblast differentiation.Therapeutic effects of telomerase in mice with pulmonary fibrosis induced by damage to the lungs and short telomeres.Role of STAT3 in skin fibrosis and transforming growth factor beta signalling.The JAK2 pathway is activated in idiopathic pulmonary fibrosis.Novel role of NPY in neuroimmune interaction and lung growth after intrauterine growth restriction.MEKK1, JNK, and SMAD3 mediate CXCL12-stimulated connective tissue growth factor expression in human lung fibroblasts.RNAi screening identifies a mechanosensitive ROCK-JAK2-STAT3 network central to myofibroblast activation.MicroRNA-708-3p as a potential therapeutic target via the ADAM17-GATA/STAT3 axis in idiopathic pulmonary fibrosis.Identification of periplakin as a major regulator of lung injury and repair in mice.Wilms' tumor 1 drives fibroproliferation and myofibroblast transformation in severe fibrotic lung disease Contribution of STAT3 to Inflammatory and Fibrotic Diseases and Prospects for its Targeting for Treatment

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STAT-3 contributes to pulmonary fibrosis through epithelial injury and fibroblast-myofibroblast differentiation.

http://www.wikidata.org/entity/Q36385387

description

2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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2015年學術文章

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2015年學術文章

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name

STAT-3 contributes to pulmonar ...... myofibroblast differentiation.

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STAT-3 contributes to pulmonar ...... myofibroblast differentiation.

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type

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STAT-3 contributes to pulmonar ...... myofibroblast differentiation.

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STAT-3 contributes to pulmonar ...... myofibroblast differentiation.

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STAT-3 contributes to pulmonar ...... myofibroblast differentiation.

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STAT-3 contributes to pulmonar ...... myofibroblast differentiation.

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STAT-3 contributes to pulmonar ...... myofibroblast differentiation.

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Anuh T George

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David J Tweardy

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Katherine Lewis

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Mesias Pedroza

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Sandeep K Agarwal

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Sarah To

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Thuy T Le

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P2860

Principles of interleukin (IL)-6-type cytokine signalling and its regulation

Interleukin-6-type cytokine signalling through the gp130/Jak/STAT pathway

Interleukin-6 contributes to inflammation and remodeling in a model of adenosine mediated lung injury

Interleukin-6 family of cytokines and gp130

Chemical probes that competitively and selectively inhibit Stat3 activation

Suppression of expression of heat shock protein 70 by gefitinib and its contribution to pulmonary fibrosis

Alterations in adenosine metabolism and signaling in patients with chronic obstructive pulmonary disease and idiopathic pulmonary fibrosis

Stats: transcriptional control and biological impact

Alveolar epithelial cell mesenchymal transition develops in vivo during pulmonary fibrosis and is regulated by the extracellular matrix

Incidence, prevalence, and clinical course of idiopathic pulmonary fibrosis: a population-based study.

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129-140

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scholarly article

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10.1096/FJ.15-273953

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1

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30

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Michael R. Blackburn

Harry Karmouty-Quintana

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2015-08-31T00:00:00Z

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281553715

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26324850

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pulmonary fibrosis

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4684532

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