Role of advanced glycation end products (AGEs) in thrombogenic abnormalities in diabetes.
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Contribution of the toxic advanced glycation end-products-receptor axis in nonalcoholic steatohepatitis-related hepatocellular carcinomaRole of receptor for advanced glycation end products (RAGE) in liver diseaseDiabetes Mellitus and Blood-Brain Barrier Dysfunction: An OverviewInvolvement of the TAGE-RAGE system in non-alcoholic steatohepatitis: Novel treatment strategiesThe receptor for advanced glycation end products and risk of peripheral arterial disease, amputation or death in type 2 diabetes: a population-based cohort study.DKK1 rescues osteogenic differentiation of mesenchymal stem cells isolated from periodontal ligaments of patients with diabetes mellitus induced periodontitisStatins and Fibrates for Diabetic Retinopathy: Protocol for a Systematic ReviewN-butanol extracts of Morinda citrifolia suppress advanced glycation end products (AGE)-induced inflammatory reactions in endothelial cells through its anti-oxidative properties.Association of Diabetic Macular Edema and Proliferative Diabetic Retinopathy With Cardiovascular Disease: A Systematic Review and Meta-analysis.Inhibition of NA(+)/H(+) Exchanger 1 Attenuates Renal Dysfunction Induced by Advanced Glycation End Products in Rats.Dietary consumption of meat, fat, animal products and advanced glycation end-products and the risk of Barrett's oesophagus.Crosstalk between advanced glycation end products (AGEs)-receptor RAGE axis and dipeptidyl peptidase-4-incretin system in diabetic vascular complications.Protective role of sulphoraphane against vascular complications in diabetes.Advanced glycation end products potentiate citrated plasma-evoked oxidative and inflammatory reactions in endothelial cells by up-regulating protease-activated receptor-1 expression.PEDF in diabetic retinopathy: a protective effect of oxidative stress.Involvement of TAGE-RAGE System in the Pathogenesis of Diabetic Retinopathy.Advanced Glycation End Products: A Molecular Target for Vascular Complications in Diabetes.Advanced glycation end products evoke endothelial cell damage by stimulating soluble dipeptidyl peptidase-4 production and its interaction with mannose 6-phosphate/insulin-like growth factor II receptor.The nucleotide hydrolysis is altered in blood serum of streptozotocin-induced diabetic rats.Glyceraldehyde-derived pyridinium (GLAP) evokes oxidative stress and inflammatory and thrombogenic reactions in endothelial cells via the interaction with RAGE.Inhibitory Effects of Siegesbeckia orientalis Extracts on Advanced Glycation End Product Formation and Key Enzymes Related to Metabolic Syndrome.Chemical Constituents of Smilax china L. Stems and Their Inhibitory Activities against Glycation, Aldose Reductase, α-Glucosidase, and Lipase.The Role of Advanced Glycation End Products in Diabetic Vascular Complications.Role of Hyperglycemia-Induced Advanced Glycation End Product (AGE) Accumulation in Atherosclerosis
P2860
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P2860
Role of advanced glycation end products (AGEs) in thrombogenic abnormalities in diabetes.
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年學術文章
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2006年學術文章
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name
Role of advanced glycation end products (AGEs) in thrombogenic abnormalities in diabetes.
@ast
Role of advanced glycation end products (AGEs) in thrombogenic abnormalities in diabetes.
@en
type
label
Role of advanced glycation end products (AGEs) in thrombogenic abnormalities in diabetes.
@ast
Role of advanced glycation end products (AGEs) in thrombogenic abnormalities in diabetes.
@en
prefLabel
Role of advanced glycation end products (AGEs) in thrombogenic abnormalities in diabetes.
@ast
Role of advanced glycation end products (AGEs) in thrombogenic abnormalities in diabetes.
@en
P2093
P1476
Role of advanced glycation end products (AGEs) in thrombogenic abnormalities in diabetes.
@en
P2093
Katsuhiko Takenaka
Kazuo Nakamura
Sho-ichi Yamagishi
Takanori Matsui
Tsutomu Imaizumi
P356
10.2174/156720206775541804
P577
2006-02-01T00:00:00Z