Spontaneous development of plasmacytoid tumors in mice with defective Fas-Fas ligand interactions. - Wikidata - wikimedia - TriplyDB

Spontaneous development of plasmacytoid tumors in mice with defective Fas-Fas ligand interactions.

Spontaneous development of plasmacytoid tumors in mice with defective Fas-Fas ligand interactions.

http://www.wikidata.org/entity/Q36400965

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Natural selection of tumor variants in the generation of "tumor escape" phenotypes Transgenic models of lymphoid neoplasia and development of a pan-hematopoietic vector Aging, immunity, and cancer.Mouse models of human familial cancer syndromes.Fas and FasL gene polymorphisms are not associated with cervical cancer but differ among Black and Mixed-ancestry South Africans The spectrum of apoptotic defects and clinical manifestations, including systemic lupus erythematosus, in humans with CD95 (Fas/APO-1) mutations.Nfatc2 and Tob1 have non-overlapping function in T cell negative regulation and tumorigenesis Structural pathways of cytokines may illuminate their roles in regulation of cancer development and immunotherapy.Aberrant expression and reverse signalling of CD70 on malignant B cells.Inducible resistance to Fas-mediated apoptosis in B cells.The many roles of FAS receptor signaling in the immune system Role of TNF family ligands in antitumor activity of natural killer cells.Immune surveillance of tumors.Cytolytic pathways in haematopoietic stem-cell transplantation.Anaplastic plasmacytoma of mouse--establishing parallels between subtypes of mouse and human plasma cell neoplasia.Tracking death dealing by Fas and TRAIL in lymphatic neoplastic disorders: pathways, targets, and therapeutic tools.Functional polymorphisms in cell death pathway genes FAS and FASL contribute to risk of lung cancer.Alterations of Fas (Apo-1/CD95) gene in cutaneous malignant melanoma.The combined risks of reduced or increased function variants in cell death pathway genes differentially influence cervical cancer risk and herpes simplex virus type 2 infection among black Africans and the Mixed Ancestry population of South Africa Soluble Fas ligand inhibits angiogenesis in rheumatoid arthritis Cytokines in cancer immunity and immunotherapy.Oral Squamous Carcinoma Cells Express B7-H1 and B7-DC Receptors in Vivo.Strain differences in the proteome of dioxin-sensitive and dioxin-resistant mice treated with 2,3,7,8-tetrabromodibenzo-p-dioxin.Anaplastic plasmacytomas: relationships to normal memory B cells and plasma cell neoplasms of immunodeficient and autoimmune mice.Differentiation of rodent immune and hematopoietic system reactive lesions from neoplasias Targeting the Fas/Fas ligand pathway in cancer.Deficiencies of GM-CSF and interferon gamma link inflammation and cancer Ionizing radiation and chemotherapeutic drugs induce apoptosis in lymphocytes in the absence of Fas or FADD/MORT1 signaling. Implications for cancer therapy.Tumor necrosis factor sustains the generalized lymphoproliferative disorder (gld) phenotype.c-FLIP mediates resistance of Hodgkin/Reed-Sternberg cells to death receptor-induced apoptosis.Evidence for selective transformation of autoreactive immature plasma cells in mice deficient in Fasl FASL -844C polymorphism is associated with increased activation-induced T cell death and risk of cervical cancer.CCR2-dependent recruitment of macrophages by tumor-educated mesenchymal stromal cells promotes tumor development and is mimicked by TNFα The role of death receptor ligands in shaping tumor microenvironment.Quantitative assessment of the association between Fas/FasL gene polymorphism and susceptibility to esophageal carcinoma in a north Chinese population.CD70 as a therapeutic target in human malignancies.RAG-induced DNA lesions activate proapoptotic BIM to suppress lymphomagenesis in p53-deficient mice.FAS promoter polymorphism: outcome of childhood acute myeloid leukemia. A children's oncology group report.Transgenic mouse model of IgM+ lymphoproliferative disease mimicking Waldenström macroglobulinemia.The Roles of Radiotherapy and Immunotherapy for the Treatment of Lymphoma

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P2860

Spontaneous development of plasmacytoid tumors in mice with defective Fas-Fas ligand interactions.

http://www.wikidata.org/entity/Q36400965

description

1998 nî lūn-bûn

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1998年の論文

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年學術文章

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1998年學術文章

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1998年學術文章

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Journal of Experimental Medicine

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T Giese

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T N Fredrickson

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P2860

Helicobacter pylori infection and gastric lymphoma

Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand

The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

Mutations in Fas associated with human lymphoproliferative syndrome and autoimmunity.

Fas gene mutations in the Canale-Smith syndrome, an inherited lymphoproliferative disorder associated with autoimmunity.

Pvt-1 transcripts are found in normal tissues and are altered by reciprocal(6;15) translocations in mouse plasmacytomas.

Lpr and gld: single gene models of systemic autoimmunity and lymphoproliferative disease.

A cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen co-downregulated with the receptor of tumor necrosis factor.

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