Aimless mutants of Cryptococcus neoformans: failure to disseminate.
about
A Zebrafish Model of Cryptococcal Infection Reveals Roles for Macrophages, Endothelial Cells, and Neutrophils in the Establishment and Control of Sustained Fungemia.Defects in phosphate acquisition and storage influence virulence of Cryptococcus neoformans.A defect in ATP-citrate lyase links acetyl-CoA production, virulence factor elaboration and virulence in Cryptococcus neoformans.It's not all about us: evolution and maintenance of Cryptococcus virulence requires selection outside the human host.The calcium transporter Pmc1 provides Ca2+ tolerance and influences the progression of murine cryptococcal infection.Cryptococcal dissemination to the central nervous system requires the vacuolar calcium transporter Pmc1.Cryptococcal pathogenic mechanisms: a dangerous trip from the environment to the brain.
P2860
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P2860
Aimless mutants of Cryptococcus neoformans: failure to disseminate.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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name
Aimless mutants of Cryptococcus neoformans: failure to disseminate.
@ast
Aimless mutants of Cryptococcus neoformans: failure to disseminate.
@en
type
label
Aimless mutants of Cryptococcus neoformans: failure to disseminate.
@ast
Aimless mutants of Cryptococcus neoformans: failure to disseminate.
@en
prefLabel
Aimless mutants of Cryptococcus neoformans: failure to disseminate.
@ast
Aimless mutants of Cryptococcus neoformans: failure to disseminate.
@en
P2093
P2860
P1476
Aimless mutants of Cryptococcus neoformans: failure to disseminate.
@en
P2093
E J Griffiths
J W Kronstad
M Kretschmer
P2860
P356
10.1016/J.FBR.2012.02.004
P577
2012-03-30T00:00:00Z