Prostaglandin E2 stimulates normal bronchial epithelial cell growth through induction of c-Jun and PDK1, a kinase implicated in oncogenesis.
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Targeting EP4 downstream c-Jun through ERK1/2-mediated reduction of DNMT1 reveals novel mechanism of solamargine-inhibited growth of lung cancer cellsActivation of PGE2/EP2 and PGE2/EP4 signaling pathways positively regulate the level of PD-1 in infiltrating CD8+ T cells in patients with lung cancer.
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Prostaglandin E2 stimulates normal bronchial epithelial cell growth through induction of c-Jun and PDK1, a kinase implicated in oncogenesis.
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2015 nî lūn-bûn
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name
Prostaglandin E2 stimulates no ...... ase implicated in oncogenesis.
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Prostaglandin E2 stimulates no ...... ase implicated in oncogenesis.
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type
label
Prostaglandin E2 stimulates no ...... ase implicated in oncogenesis.
@ast
Prostaglandin E2 stimulates no ...... ase implicated in oncogenesis.
@en
prefLabel
Prostaglandin E2 stimulates no ...... ase implicated in oncogenesis.
@ast
Prostaglandin E2 stimulates no ...... ase implicated in oncogenesis.
@en
P2093
P2860
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Prostaglandin E2 stimulates no ...... ase implicated in oncogenesis.
@en
P2093
P2860
P2888
P356
10.1186/S12931-015-0309-0
P577
2015-12-18T00:00:00Z
P5875
P6179
1045254197