G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins - Wikidata - wikimedia - TriplyDB

G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins

G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins

http://www.wikidata.org/entity/Q36430707

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Hunger and Satiety Gauge Reward Sensitivity Gsα deficiency in the dorsomedial hypothalamus underlies obesity associated with Gsα mutations.The Gq signalling pathway inhibits brown and beige adipose tissue.Effects of Melanocortin 3 and 4 Receptor Deficiency on Energy Homeostasis in Rats.A Life without Hunger: The Ups (and Downs) to Modulating Melanocortin-3 Receptor Signaling.Novel approaches to the design of bioavailable melanotropins.Mice maintain predominantly maternal Gαs expression throughout life in brown fat tissue (BAT), but not other tissues.The Role of the Melanocortin System in Metabolic Disease: New Developments and Advances.Nonclassic features of pseudohypoparathyroidism type 1A.Biased signaling at neural melanocortin receptors in regulation of energy homeostasis.Exchange factors directly activated by cAMP mediate melanocortin 4 receptor-induced gene expression.Evaluation of a melanocortin-4 receptor (MC4R) agonist (Setmelanotide) in MC4R deficiency.Lipid stress inhibits endocytosis of melanocortin-4 receptor from modified clathrin-enriched sites and impairs receptor desensitization.Early-Onset Obesity: Unrecognized First Evidence for GNAS Mutations and Methylation Changes.

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P2860

G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins

http://www.wikidata.org/entity/Q36430707

description

2015 nî lūn-bûn

@nan

2015年の論文

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2015年学术文章

@wuu

2015年学术文章

@zh-cn

2015年学术文章

@zh-hans

2015年学术文章

@zh-my

2015年学术文章

@zh-sg

2015年學術文章

@yue

2015年學術文章

@zh

2015年學術文章

@zh-hant

name

G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins

@ast

G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins

@en

type

label

G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins

@ast

G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins

@en

prefLabel

G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins

@ast

G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins

@en

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P1433

Journal of Clinical Investigation

P1476

G(q/11)α and G(s)α mediate distinct physiological responses to central melanocortins

@en

P2093

Ahmed Kablan

http://www.w3.org/2001/XMLSchema#string

Min Chen

http://www.w3.org/2001/XMLSchema#string

Mritunjay Pandey

http://www.w3.org/2001/XMLSchema#string

Oksana Gavrilova

http://www.w3.org/2001/XMLSchema#string

Yogendra Shrestha

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Yong-Qi Li

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P2860

International Union of Pharmacology. LXVIII. Mammalian bombesin receptors: nomenclature, distribution, pharmacology, signaling, and functions in normal and disease states

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

Clinical spectrum of obesity and mutations in the melanocortin 4 receptor gene

Targeted disruption of the melanocortin-4 receptor results in obesity in mice

Impaired motor coordination and persistent multiple climbing fiber innervation of cerebellar Purkinje cells in mice lacking Galphaq

Embryonic cardiomyocyte hypoplasia and craniofacial defects in G alpha q/G alpha 11-mutant mice.

An essential role for the K+-dependent Na+/Ca2+-exchanger, NCKX4, in melanocortin-4-receptor-dependent satiety.

Control of food intake and energy expenditure by Nos1 neurons of the paraventricular hypothalamus.

Profound obesity associated with a balanced translocation that disrupts the SIM1 gene.

The role of GNAS and other imprinted genes in the development of obesity.

P304

40-49

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scholarly article

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10.1172/JCI76348

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1

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126

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Lee S. Weinstein

Stefan Offermanns

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2015-11-23T00:00:00Z

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284433689

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26595811

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4701544

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